Lung epithelial GM-CSF improves host defense function and epithelial repair in influenza virus pneumonia-a new therapeutic strategy?IFNs Modify the Proteome of Legionella-Containing Vacuoles and Restrict Infection Via IRG1-Derived Itaconic AcidMacrophage-expressed IFN-β contributes to apoptotic alveolar epithelial cell injury in severe influenza virus pneumoniaInfluenza virus-induced lung injury: pathogenesis and implications for treatmentInfluenza, a One Health paradigm--novel therapeutic strategies to fight a zoonotic pathogen with pandemic potential.A Highly Immunogenic and Protective Middle East Respiratory Syndrome Coronavirus Vaccine Based on a Recombinant Measles Virus Vaccine Platform.pH Optimum of Hemagglutinin-Mediated Membrane Fusion Determines Sensitivity of Influenza A Viruses to the Interferon-Induced Antiviral State and IFITMs.Fgf10-positive cells represent a progenitor cell population during lung development and postnatallyInflammatory Responses Regulating Alveolar Ion Transport during Pulmonary Infections.The NS segment of an H5N1 highly pathogenic avian influenza virus (HPAIV) is sufficient to alter replication efficiency, cell tropism, and host range of an H7N1 HPAIVTransglutaminase 2: a new player in bronchopulmonary dysplasia?Lysyl oxidases play a causal role in vascular remodeling in clinical and experimental pulmonary arterial hypertension.Attenuating endogenous Fgfr2b ligands during bleomycin-induced lung fibrosis does not compromise murine lung repairRegulation of immunoproteasome function in the lung.Generation of a variety of stable Influenza A reporter viruses by genetic engineering of the NS gene segment.The H2S-generating enzymes cystathionine β-synthase and cystathionine γ-lyase play a role in vascular development during normal lung alveolarization.Overdiagnosis of a typical carcinoid tumor as an adenocarcinoma of the lung: a case report and review of the literature.The Human Antimicrobial Protein Bactericidal/Permeability-Increasing Protein (BPI) Inhibits the Infectivity of Influenza A VirusInfluenza Virus Infects Epithelial Stem/Progenitor Cells of the Distal Lung: Impact on Fgfr2b-Driven Epithelial Repair.Macrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection.Lung epithelial apoptosis in influenza virus pneumonia: the role of macrophage-expressed TNF-related apoptosis-inducing ligand.TGF-β directs trafficking of the epithelial sodium channel ENaC which has implications for ion and fluid transport in acute lung injury.Glucocorticoids recruit Tgfbr3 and Smad1 to shift transforming growth factor-β signaling from the Tgfbr1/Smad2/3 axis to the Acvrl1/Smad1 axis in lung fibroblasts.Deregulation of the lysyl hydroxylase matrix cross-linking system in experimental and clinical bronchopulmonary dysplasia.Standardisation of oxygen exposure in the development of mouse models for bronchopulmonary dysplasia.Localized retroperitoneal Castleman's disease: a case report and review of the literature.Apoptosis signaling in influenza virus propagation, innate host defense, and lung injury.Acute lung injury: how macrophages orchestrate resolution of inflammation and tissue repairNovel concepts of acute lung injury and alveolar-capillary barrier dysfunction.Surface expression of CD74 by type II alveolar epithelial cells: a potential mechanism for macrophage migration inhibitory factor-induced epithelial repair.Characteristics and outcomes of a cohort hospitalized for pandemic and seasonal influenza in Germany based on nationwide inpatient data.Hypercapnia Impairs ENaC Cell Surface Stability by Promoting Phosphorylation, Polyubiquitination and Endocytosis of β-ENaC in a Human Alveolar Epithelial Cell Line.Influenza A Virus Virulence Depends on Two Amino Acids in the N-Terminal Domain of Its NS1 Protein To Facilitate Inhibition of the RNA-Dependent Protein Kinase PKR.Human CD8(+) T Cells Damage Non-infected Epithelial Cells During Influenza Virus Infection In Vitro.Influenza virus damages the alveolar barrier by disrupting epithelial cell tight junctions.FXYD1 negatively regulates Na(+)/K(+)-ATPase activity in lung alveolar epithelial cells.Macrophage and cancer cell cross-talk via CCR2 and CX3CR1 is a fundamental mechanism driving lung cancer.Efficient gene delivery to primary alveolar epithelial cells by nucleofection.The "Choosing Wisely" initiative in infectious diseases.Two-Way Conversion between Lipogenic and Myogenic Fibroblastic Phenotypes Marks the Progression and Resolution of Lung Fibrosis.
P50
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P50
description
German infectiologist and university teacher
@en
deutsche Infektiologin und Hochschullehrerin
@de
hulumtuese
@sq
onderzoeker
@nl
հետազոտող
@hy
name
Susanne Herold
@ast
Susanne Herold
@de
Susanne Herold
@en
Susanne Herold
@es
Susanne Herold
@nl
Susanne Herold
@sl
type
label
Susanne Herold
@ast
Susanne Herold
@de
Susanne Herold
@en
Susanne Herold
@es
Susanne Herold
@nl
Susanne Herold
@sl
prefLabel
Susanne Herold
@ast
Susanne Herold
@de
Susanne Herold
@en
Susanne Herold
@es
Susanne Herold
@nl
Susanne Herold
@sl
P214
P227
P19
P21
P214
P227
P31
P496
0000-0001-6343-0911
P569
1975-11-10T00:00:00Z
P734
P735
P7859
viaf-168782817