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Emerging Role of Spinal Cord TRPV1 in Pain ExacerbationBiological Roles of Resolvins and Related Substances in the Resolution of PainSensory TRP channel interactions with endogenous lipids and their biological outcomesModulation of the Activities of Neuronal Ion Channels by Fatty Acid-Derived Pro-ResolventsAtypical sensors for direct and rapid neuronal detection of bacterial pathogensDirect activation of capsaicin receptors by products of lipoxygenases: endogenous capsaicin-like substancesANKTM1, a TRP-like channel expressed in nociceptive neurons, is activated by cold temperaturesNoxious cold ion channel TRPA1 is activated by pungent compounds and bradykininHigh-throughput random mutagenesis screen reveals TRPM8 residues specifically required for activation by menthol.tmc-1 encodes a sodium-sensitive channel required for salt chemosensation in C. elegans.Bradykinin-12-lipoxygenase-VR1 signaling pathway for inflammatory hyperalgesia.Specific roles for DEG/ENaC and TRP channels in touch and thermosensation in C. elegans nociceptors.Resolvin D1 attenuates activation of sensory transient receptor potential channels leading to multiple anti-nociception.Are sensory TRP channels biological alarms for lipid peroxidation?Impaired thermosensation in mice lacking TRPV3, a heat and camphor sensor in the skin.The pungency of garlic: activation of TRPA1 and TRPV1 in response to allicin.Hot channels in airways: pharmacology of the vanilloid receptor.An ion channel essential for sensing chemical damage.17(R)-resolvin D1 specifically inhibits transient receptor potential ion channel vanilloid 3 leading to peripheral antinociception.Current concepts of nociception: nociceptive molecular sensors in sensory neurons.Voluntary movements as a possible non-reflexive pain assay.EDITORIAL: Advances in Research on Pharmacological Targets for Pain Relief.Resolvins: Endogenously-Generated Potent Painkilling Substances and their Therapeutic Perspectives.Endogenous lipid-derived ligands for sensory TRP ion channels and their pain modulation.Emerging roles of TRPA1 in sensation of oxidative stress and its implications in defense and danger.Comparison of growth factor and cytokine expression in patients with degenerated disc disease and herniated nucleus pulposus.Agonist recognition sites in the cytosolic tails of vanilloid receptor 1.A role of TRPA1 in mechanical hyperalgesia is revealed by pharmacological inhibition.Heterogeneity in the Drosophila gustatory receptor complexes that detect aversive compounds.Opposite thermosensor in fruitfly and mouse.Lipoxygenase inhibitors suppressed carrageenan-induced Fos-expression and inflammatory pain responses in the rat.TRPV1 recapitulates native capsaicin receptor in sensory neurons in association with Fas-associated factor 1.Endogenous TRPV4 Expression of a Hybrid Neuronal Cell Line N18D3 and Its Utilization to Find a Novel Synthetic Ligand.Peripheral bee venom's anti-inflammatory effect involves activation of the coeruleospinal pathway and sympathetic preganglionic neurons.Nociceptive Roles of TRPM2 Ion Channel in Pathologic Pain.P2X1 Receptor-Mediated Ca2+ Influx Triggered by DA-9801 Potentiates Nerve Growth Factor-Induced Neurite Outgrowth.Romo1 is a mitochondrial nonselective cation channel with viroporin-like characteristics.Peripheral serotonin receptor 2B and transient receptor potential channel 4 mediate pruritus to serotonergic antidepressants in miceA capsaicin-receptor antagonist, capsazepine, reduces inflammation-induced hyperalgesic responses in the rat: evidence for an endogenous capsaicin-like substanceNanoparticle formulation for controlled release of capsaicin
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description
researcher
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wetenschapper
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հետազոտող
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name
Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
@nl
Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
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Sun Wook Hwang
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P108
P1153
7404625746
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P496
0000-0001-8460-7480