about
A mouse model uncovers LKB1 as an UVB-induced DNA damage sensor mediating CDKN1A (p21WAF1/CIP1) degradationEpithelial-mesenchymal transition markers and HER3 expression are predictors of elisidepsin treatment response in breast and pancreatic cancer cell linesMethylthioadenosine (MTA) inhibits melanoma cell proliferation and in vivo tumor growthStem cell-like transcriptional reprogramming mediates metastatic resistance to mTOR inhibition.Fluorescence in situ hybridization and immunohistochemistry as diagnostic methods for ALK positive non-small cell lung cancer patientsIdentification of the novel mutation m.5658T>C in the mitochondrial tRNA(Asn) gene in a patient with myopathy, bilateral ptosis and ophthalmoparesis.RHOA inactivation enhances Wnt signalling and promotes colorectal cancer.Lkb1 loss promotes tumor progression of BRAF(V600E)-induced lung adenomasmiR-125b acts as a tumor suppressor in breast tumorigenesis via its novel direct targets ENPEP, CK2-α, CCNJ, and MEGF9.Lymphangioleiomyomatosis Biomarkers Linked to Lung Metastatic Potential and Cell StemnessBrush border myosin Ia has tumor suppressor activity in the intestine.Myo5b knockout mice as a model of microvillus inclusion disease.Increased voltage-dependent K(+) channel Kv1.3 and Kv1.5 expression correlates with leiomyosarcoma aggressivenessEffect of low doses of actinomycin D on neuroblastoma cell linespeIF4E as an independent prognostic factor and a potential therapeutic target in diffuse infiltrating astrocytomas.Loss of the EPH receptor B6 contributes to colorectal cancer metastasisTargeting the voltage-dependent K(+) channels Kv1.3 and Kv1.5 as tumor biomarkers for cancer detection and prevention.The voltage-dependent K(+) channels Kv1.3 and Kv1.5 in human cancer.Aprataxin tumor levels predict response of colorectal cancer patients to irinotecan-based treatment.E1a is an exogenous in vivo tumour suppressor.ALDH1A3 is epigenetically regulated during melanocyte transformation and is a target for melanoma treatment.Emerging role for the voltage-dependent K+ channel Kv1.5 in B-lymphocyte physiology: expression associated with human lymphoma malignancy.The effect of p-4E-BP1 and p-eIF4E on cell proliferation in a breast cancer model.The dual PI3K/mTOR inhibitor PI-103 promotes immunosuppression, in vivo tumor growth and increases survival of sorafenib-treated melanoma cells.The receptor tyrosine kinase EPHB4 has tumor suppressor activities in intestinal tumorigenesis.Regulation of replicative and stress-induced senescence by RSK4, which is down-regulated in human tumors.c-Abl activates p38 MAPK independently of its tyrosine kinase activity: Implications in cisplatin-based therapy.The role of oncogenic HPV determination for diagnosis of high-grade anal intraepithelial neoplasia in HIV-infected MSM.Risk factors of high-grade anal intraepithelial neoplasia recurrence in HIV-infected MSM.Regulation of the transcriptional activity of c-Fos by ERK. A novel role for the prolyl isomerase PIN1.Modulation of the p38 MAPK (mitogen-activated protein kinase) pathway through Bcr/Abl: implications in the cellular response to Ara-C.Focal epithelial hyperplasia by human papillomavirus (HPV)-32 misdiagnosed as HPV-16 and treated with combination of retinoids, imiquimod and quadrivalent HPV vaccine.Genetic profile of GNAQ-mutated blue melanocytic neoplasms reveals mutations in genes linked to genomic instability and the PI3K pathway.Modulation of PI3K/Akt pathway by E1a mediates sensitivity to cisplatin.S-adenosylhomocysteine hydrolase downregulation contributes to tumorigenesis.Atrophy of mesenteric sympathetic innervation may contribute to splanchnic vasodilation in rat portal hypertension.mTOR signaling pathway in penile squamous cell carcinoma: pmTOR and peIF4E over expression correlate with aggressive tumor behavior.Down-regulation of spinophilin in lung tumours contributes to tumourigenesis.JC virus and human colon carcinoma: an intriguing and inconclusive association.Clinical response to a lapatinib-based therapy for a Li-Fraumeni syndrome patient with a novel HER2V659E mutation.
P50
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P50
description
researcher, ORCID id # 0000-0003-1526-3201
@en
wetenschapper
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name
Javier Hernandez-Losa
@ast
Javier Hernandez-Losa
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Javier Hernandez-Losa
@es
Javier Hernandez-Losa
@nl
type
label
Javier Hernandez-Losa
@ast
Javier Hernandez-Losa
@en
Javier Hernandez-Losa
@es
Javier Hernandez-Losa
@nl
prefLabel
Javier Hernandez-Losa
@ast
Javier Hernandez-Losa
@en
Javier Hernandez-Losa
@es
Javier Hernandez-Losa
@nl
P106
P1153
23034454900
6602976648
P21
P31
P496
0000-0003-1526-3201