about
NeuroAIDS, drug abuse, and inflammation: building collaborative research activitiesDiesel exhaust particles induce oxidative stress, proinflammatory signaling, and P-glycoprotein up-regulation at the blood-brain barrierGene-environment interactions: key to unraveling the mystery of Parkinson's diseaseMicroglial regulation of immunological and neuroprotective functions of astrogliaRole of microglia in inflammation-mediated degeneration of dopaminergic neurons: neuroprotective effect of interleukin 10Changes of hippocampal Cu/Zn-superoxide dismutase after kainate treatment in the ratMicroglia-mediated neurotoxicity: uncovering the molecular mechanismsMicroglia and inflammation-mediated neurodegeneration: multiple triggers with a common mechanismBrain injury in a dish: a model for reactive gliosisRole of oxidative stress in epileptic seizures.Endogenous dynorphin protects against neurotoxin-elicited nigrostriatal dopaminergic neuron damage and motor deficits in miceSubstance P enhances microglial density in the substantia nigra through neurokinin-1 receptor/NADPH oxidase-mediated chemotaxis in miceReactive microgliosis: extracellular micro-calpain and microglia-mediated dopaminergic neurotoxicity.Naloxone protects rat dopaminergic neurons against inflammatory damage through inhibition of microglia activation and superoxide generation.Inducible nitric oxide synthase is key to peroxynitrite-mediated, LPS-induced protein radical formation in murine microglial BV2 cells.Resveratrol protects dopamine neurons against lipopolysaccharide-induced neurotoxicity through its anti-inflammatory actions.Substance P exacerbates dopaminergic neurodegeneration through neurokinin-1 receptor-independent activation of microglial NADPH oxidaseCritical role for microglial NADPH oxidase in rotenone-induced degeneration of dopaminergic neurons.3-hydroxymorphinan is neurotrophic to dopaminergic neurons and is also neuroprotective against LPS-induced neurotoxicity.Subpicomolar diphenyleneiodonium inhibits microglial NADPH oxidase with high specificity and shows great potential as a therapeutic agent for neurodegenerative diseases.Verapamil protects dopaminergic neuron damage through a novel anti-inflammatory mechanism by inhibition of microglial activation.3-Hydroxymorphinan, a metabolite of dextromethorphan, protects nigrostriatal pathway against MPTP-elicited damage both in vivo and in vitro.Modulation of Endogenous Opioid Systems by Electroconvulsive Shock.HMGB1 acts on microglia Mac1 to mediate chronic neuroinflammation that drives progressive neurodegeneration.Role of nitric oxide in inflammation-mediated neurodegeneration.Post-insult valproate treatment potentially improved functional recovery in patients with acute middle cerebral artery infarction.Role of microglia in inflammation-mediated neurodegenerative diseases: mechanisms and strategies for therapeutic intervention.The BDNF Val66Met polymorphism and plasma brain-derived neurotrophic factor levels in Han Chinese heroin-dependent patients.Neuroinflammation and α-synuclein dysfunction potentiate each other, driving chronic progression of neurodegeneration in a mouse model of Parkinson's disease.Low dose dextromethorphan attenuates moderate experimental autoimmune encephalomyelitis by inhibiting NOX2 and reducing peripheral immune cells infiltration in the spinal cordPGE2 Inhibits IL-10 Production via EP2-Mediated β-Arrestin Signaling in Neuroinflammatory ConditionNovel anti-inflammatory therapy for Parkinson's disease.α-Synuclein, a chemoattractant, directs microglial migration via H2O2-dependent Lyn phosphorylation.Pro-inflammatory cytokines and lipopolysaccharide induce changes in cell morphology, and upregulation of ERK1/2, iNOS and sPLA₂-IIA expression in astrocytes and microglia.A novel role of microglial NADPH oxidase in mediating extra-synaptic function of norepinephrine in regulating brain immune homeostasis.Post-treatment with an ultra-low dose of NADPH oxidase inhibitor diphenyleneiodonium attenuates disease progression in multiple Parkinson's disease modelsLow-dose add-on memantine treatment may improve cognitive performance and self-reported health conditions in opioid-dependent patients undergoing methadone-maintenance-therapy.Rotenone activates phagocyte NADPH oxidase by binding to its membrane subunit gp91phox.Suberoylanilide hydroxamic acid, a histone deacetylase inhibitor, protects dopaminergic neurons from neurotoxin-induced damageNaloxone inhibits immune cell function by suppressing superoxide production through a direct interaction with gp91phox subunit of NADPH oxidase
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Jau-Shyong Hong
@ast
Jau-Shyong Hong
@en
Jau-Shyong Hong
@es
Jau-Shyong Hong
@nl
type
label
Jau-Shyong Hong
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Jau-Shyong Hong
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Jau-Shyong Hong
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Jau-Shyong Hong
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prefLabel
Jau-Shyong Hong
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Jau-Shyong Hong
@en
Jau-Shyong Hong
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Jau-Shyong Hong
@nl
P106
P31
P496
0000-0002-3056-8401