Phosphatase and tensin homolog, deleted on chromosome 10 deficiency in brain causes defects in synaptic structure, transmission and plasticity, and myelination abnormalities
about
Mitochondrial dysfunction in Pten haplo-insufficient mice with social deficits and repetitive behavior: interplay between Pten and p53Phosphorylation of the actin binding protein Drebrin at S647 is regulated by neuronal activity and PTENA comprehensive functional analysis of PTEN mutations: implications in tumor- and autism-related syndromesCharacterizing autism spectrum disorders by key biochemical pathways.Balancing Proliferation and Connectivity in PTEN-associated Autism Spectrum DisorderPTEN: A master regulator of neuronal structure, function, and plasticitySubcellular targeting and dynamic regulation of PTEN: implications for neuronal cells and neurological disordersCell-type specific roles for PTEN in establishing a functional retinal architectureIn vivo AAV1 transduction with hRheb(S16H) protects hippocampal neurons by BDNF productionThe parvalbumin/somatostatin ratio is increased in Pten mutant mice and by human PTEN ASD alleles.MTM-6, a phosphoinositide phosphatase, is required to promote synapse formation in Caenorhabditis elegansSelective neuronal PTEN deletion: can we take the brakes off of growth without losing control?A Subset of Autism-Associated Genes Regulate the Structural Stability of Neurons.Common mechanisms of excitatory and inhibitory imbalance in schizophrenia and autism spectrum disordersDiscovery of Rare Mutations in Autism: Elucidating Neurodevelopmental MechanismsNO signaling and S-nitrosylation regulate PTEN inhibition in neurodegenerationPTEN is recruited to the postsynaptic terminal for NMDA receptor-dependent long-term depressionActivation of PPAR-γ and PTEN cascade participates in lovastatin-mediated accelerated differentiation of oligodendrocyte progenitor cells.Dendritic spine pathology in neuropsychiatric disordersPTEN knockdown alters dendritic spine/protrusion morphology, not densityPTEN regulation of local and long-range connections in mouse auditory cortex.A critical step for postsynaptic F-actin organization: regulation of Baz/Par-3 localization by aPKC and PTENExcessive activation of mTOR in postnatally generated granule cells is sufficient to cause epilepsy.Cell type specificity of PI3K signaling in Pdk1- and Pten-deficient brainsLaminar and compartmental regulation of dendritic growth in mature cortex.Pten loss in Olig2 expressing neural progenitor cells and oligodendrocytes leads to interneuron dysplasia and leukodystrophy.Plasma corticosterone activates SGK1 and induces morphological changes in oligodendrocytes in corpus callosum.SHP-2 promotes the maturation of oligodendrocyte precursor cells through Akt and ERK1/2 signaling in vitro.Hippocampal granule cell pathology in epilepsy - a possible structural basis for comorbidities of epilepsy?Using the neurofibromatosis tumor predisposition syndromes to understand normal nervous system development.Functional analysis of the protein phosphatase activity of PTENTranscriptomic analysis of genetically defined autism candidate genes reveals common mechanisms of actionTranscription factor TFAP2C regulates major programs required for murine fetal germ cell maintenance and haploinsufficiency predisposes to teratomas in male miceHyperactivity of newborn Pten knock-out neurons results from increased excitatory synaptic driveViral vector-based improvement of optic nerve regeneration: characterization of individual axons' growth patterns and synaptogenesis in a visual target.Multiple roles for mammalian target of rapamycin signaling in both glutamatergic and GABAergic synaptic transmissionCytoplasm-predominant Pten associates with increased region-specific brain tyrosine hydroxylase and dopamine D2 receptors in mouse model with autistic traits.Hypomyelination following deletion of Tsc2 in oligodendrocyte precursors.The tumor suppressor PTEN and the PDK1 kinase regulate formation of the columnar neural epitheliumMouse models of human PIK3CA-related brain overgrowth have acutely treatable epilepsy
P2860
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P2860
Phosphatase and tensin homolog, deleted on chromosome 10 deficiency in brain causes defects in synaptic structure, transmission and plasticity, and myelination abnormalities
description
2008 nî lūn-bûn
@nan
2008 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Phosphatase and tensin homolog ...... and myelination abnormalities
@ast
Phosphatase and tensin homolog ...... and myelination abnormalities
@en
Phosphatase and tensin homolog ...... and myelination abnormalities
@en-gb
Phosphatase and tensin homolog ...... and myelination abnormalities
@nl
type
label
Phosphatase and tensin homolog ...... and myelination abnormalities
@ast
Phosphatase and tensin homolog ...... and myelination abnormalities
@en
Phosphatase and tensin homolog ...... and myelination abnormalities
@en-gb
Phosphatase and tensin homolog ...... and myelination abnormalities
@nl
prefLabel
Phosphatase and tensin homolog ...... and myelination abnormalities
@ast
Phosphatase and tensin homolog ...... and myelination abnormalities
@en
Phosphatase and tensin homolog ...... and myelination abnormalities
@en-gb
Phosphatase and tensin homolog ...... and myelination abnormalities
@nl
P2093
P2860
P921
P3181
P1433
P1476
Phosphatase and tensin homolog ...... and myelination abnormalities
@en
P2093
I T Bayazitov
M M Fraser
P2860
P304
P3181
P356
10.1016/J.NEUROSCIENCE.2007.10.048
P407
P577
2007-11-17T00:00:00Z