Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice - Wikidata - awgldk - TriplyDB

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

http://www.wikidata.org/entity/Q24644715

about

Postnatal Sim1 deficiency causes hyperphagic obesity and reduced Mc4r and oxytocin expression Oxytocin: the great facilitator of life.From Autism to Eating Disorders and More: The Role of Oxytocin in Neuropsychiatric Disorders Chronic Oxytocin Administration as a Treatment Against Impaired Leptin Signaling or Leptin Resistance in Obesity Embryonic development of the hypothalamic feeding circuitry: Transcriptional, nutritional, and hormonal influences The Role of PVH Circuits in Leptin Action and Energy Balance Small 6q16.1 Deletions Encompassing POU3F2 Cause Susceptibility to Obesity and Variable Developmental Delay with Intellectual Disability Developmental specification of metabolic circuitry Control of food intake and energy expenditure by Nos1 neurons of the paraventricular hypothalamus.Role of developmental factors in hypothalamic function Novel leptin-regulated genes revealed by transcriptional profiling of the hypothalamic paraventricular nucleus.Oxytocin secretion is related to measures of energy homeostasis in young amenorrheic athletes.Neural circuitry underlying the central hypertensive action of nesfatin-1: melanocortins, corticotropin-releasing hormone, and oxytocin Inducible neuronal inactivation of Sim1 in adult mice causes hyperphagic obesity.Neurobiology of food intake in health and disease.Oxytocin administration suppresses hypothalamic activation in response to visual food cues.Cyto- and chemoarchitecture of the hypothalamic paraventricular nucleus in the C57BL/6J male mouse: a study of immunostaining and multiple fluorescent tract tracing The fat-induced satiety factor oleoylethanolamide suppresses feeding through central release of oxytocin.The role of GNAS and other imprinted genes in the development of obesity.Oxytocin as feeding inhibitor: maintaining homeostasis in consummatory behavior.Oxytocin in the ventromedial hypothalamic nucleus reduces feeding and acutely increases energy expenditure.Ablation of Sim1 neurons causes obesity through hyperphagia and reduced energy expenditure.Oxytocin and vasopressin agonists and antagonists as research tools and potential therapeutics.Deconstruction of a neural circuit for hunger.Oxytocin and bone Role of oxytocin signaling in the regulation of body weight.An obligate role of oxytocin neurons in diet induced energy expenditure MC4R-expressing glutamatergic neurons in the paraventricular hypothalamus regulate feeding and are synaptically connected to the parabrachial nucleus Lack of cAMP-response element-binding protein 1 in the hypothalamus causes obesity.Leptin activates oxytocin neurons of the hypothalamic paraventricular nucleus in both control and diet-induced obese rodents.Treatment of obesity and diabetes using oxytocin or analogs in patients and mouse models.Effects of deficiency of the G protein Gsα on energy and glucose homeostasis Neuronostatin acts in brain to biphasically increase mean arterial pressure through sympatho-activation followed by vasopressin secretion: the role of melanocortin receptors.Hypothalamic survival circuits: blueprints for purposive behaviors Genetic approaches to understanding human obesity.Paraventricular nucleus Sim1 neuron ablation mediated obesity is resistant to high fat diet.Melanocortin control of energy balance: evidence from rodent models.GABAergic projections from lateral hypothalamus to paraventricular hypothalamic nucleus promote feeding.Ontogenesis of oxytocin pathways in the mammalian brain: late maturation and psychosocial disorders Chronic oxytocin administration inhibits food intake, increases energy expenditure, and produces weight loss in fructose-fed obese rhesus monkeys

P2860

Q24630590-5864E683-AA81-4EA5-AA29-05DEA1A18657 Q24651638-155E4FFB-3202-4D61-ACB9-2F2F7CA28EBE Q26770469-EB8BB88C-4BA3-4E1F-A1D7-79E5279B69D1 Q26795486-EE660CF2-A333-4CFF-B280-DF4300D0DB4D Q27004256-296EB107-CD8D-48F0-8A84-7CE64E4F5742 Q28067395-7AAE138A-0F88-4DFF-B63B-001C0C79ABF8 Q28272815-4E43A58A-EDA3-41CA-AC8B-72EF3636E10C Q30373177-AAF853ED-5E8E-4265-A291-485AB09A964E Q30405244-3DF84BEB-4672-425A-A926-44FF14B04C3D Q30412468-88B4A2BA-79DE-434F-B4E1-6B92E1435DF8 Q33385599-C2C3B996-85D4-4AA1-BE1C-B1EE59770ACB Q33568999-92E2F965-3A75-48F8-B9FD-69E32099CE99 Q33628107-E2009510-02E5-43A5-86B2-99DC8A289166 Q33765508-C65886D4-B721-4BFB-A1A6-559485106747 Q33825213-29CB5D31-CF5E-4991-87EA-B96F3E1847C6 Q33842858-730C07D8-1FB7-4BD9-AC14-C5ABE4D9AE8A Q33925499-ECC2970F-9713-4625-A59C-B8A9D81269D5 Q33975720-5053A929-270E-4E77-A948-61201C56F6E5 Q34099397-9C7A82BC-357F-44CA-85E5-24F11112C3CD Q34188589-D529C729-2614-4996-93EE-45141FDAA0D1 Q34201138-F5EAD0DB-7154-40E6-84CA-41A1F4A33E77 Q34257527-C8F0DA5D-B075-40F3-9918-CEFB219EB3AD Q34257617-B8F00F87-120C-4109-B8E0-A735B79815AA Q34288463-D4BFE715-A482-4BF3-BA51-6CAA23F93D1E Q34356431-5A05186F-9F0B-45C9-B847-FE1A9927EA24 Q34425122-4B4EA479-3B2F-4362-938D-B367F64AE99A Q34428252-0B65D590-9107-4531-9DE4-23478A749884 Q34583537-EC7EC658-E974-461E-A0BF-A4043C5AD064 Q34624262-0E0287E2-D7AB-4222-8218-F4E13BCC0D60 Q34635349-0362568A-2671-40D5-AF9C-A7FE97F6D2D2 Q34734936-62F5CBDF-9085-4744-8668-1125A454E77D Q34883776-8F267B14-4746-4A06-B749-EA5BAAC28F8D Q34979573-A69636EA-F3C6-405A-9230-DC599AEAD302 Q35012837-D2F66BF2-7AB9-454D-A04F-23F1A1B47BB6 Q35015765-59189248-E7C5-4DA3-8D65-61B83BF961E9 Q35049439-C3287147-5F2E-4C28-9A2B-A532D7CFADFC Q35103464-D6D48890-7CD8-4E8D-9B1F-1CFBD4689212 Q35117558-AB417019-3085-4143-B0CA-FBA703E5EC1B Q35123962-774AC633-D622-4307-9037-5D17DEF50FC9 Q35138545-202C13CD-C04F-4713-AAE5-39BE5F40F5A1

P2860

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

http://www.wikidata.org/entity/Q24644715

description

2008 nî lūn-bûn

@nan

2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2008年の論文

@ja

2008年論文

@yue

2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

name

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

@ast

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

@en

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

@nl

type

label

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

@ast

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

@en

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

@nl

prefLabel

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

@ast

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

@en

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

@nl

P1433

Q24644715-FF1902E8-C40A-4336-9213-C971A67399EB

P1476

Q24644715-312E7A0F-4710-42C7-886D-F0F48755EAFC

P2093

Q24644715-0739CB13-AD6A-4FB0-A85B-87AA51781EBC

Q24644715-2238FA94-F049-4439-96C6-26D12732D1A3

Q24644715-2F46FEC3-B5ED-4A22-872E-B1402A966B2F

P2860

Q24644715-04D3D0C7-9084-4BF0-BBE9-0EAAF540B7BC

Q24644715-0D364F02-D97D-4005-8F00-AB571CE50981

Q24644715-192A1646-ACE8-493C-BEBB-56AD35E353E0

Q24644715-19C5D6B9-7914-4C30-9573-24723D905F24

Q24644715-1A4C6302-6DA7-4059-B46A-6170D1799CEF

Q24644715-1B7B6611-8938-4AB1-90C4-C64AC4A5F08B

Q24644715-1CA1B665-7EDA-4DFE-BBD5-B9020DC585AC

Q24644715-1D715A3E-BEC2-4628-ADD0-52C45657EBED

Q24644715-281D612F-F3D3-4D61-AFDE-AF7FB5788533

Q24644715-287DD7A1-176E-4FF3-8AB4-DA59089A094F

P304

Q24644715-6B6AB619-3B79-4F36-9BFE-51CE4F19A9B0

P31

Q24644715-D5991152-28D7-49BE-AECC-B881EB272F00

P3181

Q24644715-D9EE2DE9-6C78-4EAC-BA9B-5708ACA6E4BC

P356

Q24644715-B9602857-414C-4D3A-9D86-79316D40FE09

P407

Q24644715-B5BD277B-212D-4680-8BE2-428EFF8E9E93

P433

Q24644715-580E1AAD-7430-4B0E-A96C-ED330F99DAF7

P478

Q24644715-E51E803C-5A9D-422A-BEAE-5BFF20720403

P50

Q24644715-1E2F6691-9F10-4AA2-A1E9-BFC26142B48A

Q24644715-8444B18F-AD3F-489F-9EA1-75B43106B2A7

P577

Q24644715-8C31DDAB-4B49-40D0-9B7B-A2D9B8F3C298

P5875

Q24644715-E401FC93-D53F-4F08-8E4F-8D7CB5871D48

P698

Q24644715-22E9FBF8-CFFE-4945-ACB0-0053492FC4E3

P921

Q24644715-36750886-6736-4C31-AEE2-8CCEA4ECB057

Q24644715-677E01E4-1557-4F31-9DFF-87C4C8BA81D9

Q24644715-8098426B-CA49-4F75-AC95-22A6A0635468

P932

Q24644715-C73EF4F7-E3FF-420F-8E93-1D2916514D98

P3181

P356

P1433

Molecular Endocrinology

P1476

Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice

@en

P2093

Bassil M Kublaoui

http://www.w3.org/2001/XMLSchema#string

Terry Gemelli

http://www.w3.org/2001/XMLSchema#string

Yu Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Pervasive social deficits, but normal parturition, in oxytocin receptor-deficient mice

Neither agouti-related protein nor neuropeptide Y is critically required for the regulation of energy homeostasis in mice

The human genome browser at UCSC

Clinical spectrum of obesity and mutations in the melanocortin 4 receptor gene

The oxytocin receptor system: structure, function, and regulation

Central nervous system control of food intake

The role of sex steroids in the oxytocin hormone system.

Leptin action in the forebrain regulates the hindbrain response to satiety signals.

Profound obesity associated with a balanced translocation that disrupts the SIM1 gene.

Sim1 haploinsufficiency causes hyperphagia, obesity and reduction of the paraventricular nucleus of the hypothalamus.

P304

1723-34

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P3181

3703647

http://www.w3.org/2001/XMLSchema#string

P356

10.1210/ME.2008-0067

http://www.w3.org/2001/XMLSchema#string

P407

P433

7

http://www.w3.org/2001/XMLSchema#string

P478

22

http://www.w3.org/2001/XMLSchema#string

P50

Kristen P Tolson

P577

2008-07-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

5401733

http://www.w3.org/2001/XMLSchema#string

P698

18451093

http://www.w3.org/2001/XMLSchema#string

P921

haploinsufficiency

P932

2453606

http://www.w3.org/2001/XMLSchema#string