Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias - Wikidata - awgldk - TriplyDB

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

http://www.wikidata.org/entity/Q24654907

about

Panhandle and reverse-panhandle PCR enable cloning of der(11) and der(other) genomic breakpoint junctions of MLL translocations and identify complex translocation of MLL, AF-4, and CDK6.t(11;22)(q23;q11.2) In acute myeloid leukemia of infant twins fuses MLL with hCDCrel, a cell division cycle gene in the genomic region of deletion in DiGeorge and velocardiofacial syndromes Detection of leukemia-associated MLL-GAS7 translocation early during chemotherapy with DNA topoisomerase II inhibitors Human LPP gene is fused to MLL in a secondary acute leukemia with a t(3;11) (q28;q23)Roles of retrotransposons in benign and malignant hematologic disease.Diagnostic tool for the identification of MLL rearrangements including unknown partner genes Near-precise interchromosomal recombination and functional DNA topoisomerase II cleavage sites at MLL and AF-4 genomic breakpoints in treatment-related acute lymphoblastic leukemia with t(4;11) translocation Genomic inverse PCR for exploration of ligated breakpoints (GIPFEL), a new method to detect translocations in leukemia Leukemias related to treatment with DNA topoisomerase II inhibitors.Therapy related leukemias: susceptibility, prevention and treatment.Therapy-related acute lymphoblastic leukaemia with MLL rearrangements following DNA topoisomerase II inhibitors, an increasing problem: report on two new cases and review of the literature since 1992.Translocation t(X;11)(q13;q23) in B-cell chronic lymphocytic leukemia disrupts two novel genes.Proteomic identification of oncogenic chromosomal translocation partners encoding chimeric anaplastic lymphoma kinase fusion proteins.The role of Alu repeat clusters as mediators of recurrent chromosomal aberrations in tumors.Panhandle PCR for cDNA: a rapid method for isolation of MLL fusion transcripts involving unknown partner genes.Split-signal FISH for detection of chromosome aberrations in acute lymphoblastic leukemia.The MLL partial tandem duplication: evidence for recessive gain-of-function in acute myeloid leukemia identifies a novel patient subgroup for molecular-targeted therapy Split-signal FISH for detection of chromosome aberrations.Prospective tracing of MLL-FRYL clone with low MEIS1 expression from emergence during neuroblastoma treatment to diagnosis of myelodysplastic syndrome Mass spectrometry-based proteomic studies of human anaplastic large cell lymphoma.Association of CYP3A4 genotype with treatment-related leukemia.Antileukemic Scalarane Sesterterpenoids and Meroditerpenoid from Carteriospongia (Phyllospongia) sp., Induce Apoptosis via Dual Inhibitory Effects on Topoisomerase II and Hsp90.Topoisomerase II and leukemia.Cryptic insertion of PML-RARA into the 3p25 locus in an acute promyelocytic leukemia with t(3;17)(p25;q21)Reciprocal DNA topoisomerase II cleavage events at 5'-TATTA-3' sequences in MLL and AF-9 create homologous single-stranded overhangs that anneal to form der(11) and der(9) genomic breakpoint junctions in treatment-related AML without further process

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Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

http://www.wikidata.org/entity/Q24654907

description

1997 nî lūn-bûn

@nan

1997 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1997 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

1997年の論文

@ja

1997年論文

@yue

1997年論文

@zh-hant

1997年論文

@zh-hk

1997年論文

@zh-mo

1997年論文

@zh-tw

1997年论文

@wuu

name

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

@ast

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

@en

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

@nl

type

label

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

@ast

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

@en

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

@nl

prefLabel

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

@ast

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

@en

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

@nl

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PNAS.94.21.11583

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Panhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemias

@en

P2093

B J Lange

http://www.w3.org/2001/XMLSchema#string

C A Felix

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C S Kim

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D H Jones

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E F Rappaport

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M D Megonigal

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P C Nowell

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P2860

Genes on chromosomes 4, 9, and 19 involved in 11q23 abnormalities in acute leukemia share sequence homology and/or common motifs

Structure and mechanism of DNA topoisomerase II

The t(4;11) chromosome translocation of human acute leukemias fuses the ALL-1 gene, related to Drosophila trithorax, to the AF-4 gene

ALL-1 partial duplication in acute leukemia.

The partial tandem duplication of ALL1 in acute myeloid leukemia with normal cytogenetics or trisomy 11 is restricted to one chromosome.

Radiotherapy- and chemotherapy-induced myelodysplasia and acute myeloid leukemia. A review.

Complete exon structure of the ALL1 gene.

When good enzymes go bad: conversion of topoisomerase II to a cellular toxin by antineoplastic drugs.

Alu: structure, origin, evolution, significance and function of one-tenth of human DNA.

An STS-based radiation hybrid map of the human genome.

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11583-8

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94

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