Inflammation and intracellular metabolism: new targets in OA - Wikidata - awgldk - TriplyDB

Inflammation and intracellular metabolism: new targets in OA

Inflammation and intracellular metabolism: new targets in OA

http://www.wikidata.org/entity/Q26777620

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Review: Metabolic Regulation of Inflammation in Osteoarthritis MicroRNAs and Autophagy: Fine Players in the Control of Chondrocyte Homeostatic Activities in Osteoarthritis.The effects of different frequency treadmill exercise on lipoxin A4 and articular cartilage degeneration in an experimental model of monosodium iodoacetate-induced osteoarthritis in rats Inflammation and epigenetic regulation in osteoarthritis.AMPK deficiency in chondrocytes accelerated the progression of instability-induced and ageing-associated osteoarthritis in adult mice.Cross-species gene modules emerge from a systems biology approach to osteoarthritis.The Role of Sirtuins in Cartilage Homeostasis and Osteoarthritis.Is osteoarthritis one disease or a collection of many?NEAT1/miR-181c Regulates Osteopontin (OPN)-Mediated Synoviocyte Proliferation in Osteoarthritis.Homocysteine causes dysfunction of chondrocytes and oxidative stress through repression of SIRT1/AMPK pathway: A possible link between hyperhomocysteinemia and osteoarthritis.Recent Insights into the Contribution of the Changing Hypertrophic Chondrocyte Phenotype in the Development and Progression of Osteoarthritis.Exosomes and Stem Cells in Degenerative Disease Diagnosis and Therapy.

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Inflammation and intracellular metabolism: new targets in OA

http://www.wikidata.org/entity/Q26777620

description

2015 nî lūn-bûn

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2015 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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Inflammation and intracellular metabolism: new targets in OA

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Inflammation and intracellular metabolism: new targets in OA

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Inflammation and intracellular metabolism: new targets in OA

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Inflammation and intracellular metabolism: new targets in OA

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Inflammation and intracellular metabolism: new targets in OA

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Inflammation and intracellular metabolism: new targets in OA

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Inflammation and intracellular metabolism: new targets in OA

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Inflammation and intracellular metabolism: new targets in OA

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Inflammation and intracellular metabolism: new targets in OA

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J.JOCA.2014.12.016

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Osteoarthritis and Cartilage

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Inflammation and intracellular metabolism: new targets in OA

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R Liu-Bryan

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Role of AMPK-mTOR-Ulk1/2 in the regulation of autophagy: cross talk, shortcuts, and feedbacks

AMP-activated protein kinase: a target for drugs both ancient and modern

The hallmarks of aging

Resveratrol inhibits interleukin 1β-mediated inducible nitric oxide synthase expression in articular chondrocytes by activating SIRT1 and thereby suppressing nuclear factor-κB activity

ATF6 upregulates XBP1S and inhibits ER stress-mediated apoptosis in osteoarthritis cartilage

Germline deletion of AMP-activated protein kinase beta subunits reduces bone mass without altering osteoclast differentiation or function.

Ionizing radiation induces cellular senescence of articular chondrocytes via negative regulation of SIRT1 by p38 kinase.

Chondrocyte autophagy is stimulated by HIF-1 dependent AMPK activation and mTOR suppression

Autophagy is a protective mechanism in normal cartilage, and its aging-related loss is linked with cell death and osteoarthritis

AMPK and SIRT1: a long-standing partnership?

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1835-1842

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10.1016/J.JOCA.2014.12.016

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biomedical engineering

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