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The Role of Leucine-Rich Repeat Containing Protein 10 (LRRC10) in Dilated CardiomyopathyNovel Perspectives in Redox Biology and Pathophysiology of Failing Myocytes: Modulation of the Intramyocardial Redox Milieu for Therapeutic Interventions-A Review Article from the Working Group of Cardiac Cell Biology, Italian Society of CardiologyCounteracting Protein Kinase Activity in the Heart: The Multiple Roles of Protein PhosphatasesCaMKII regulation of cardiac K channelsRationally engineered Troponin C modulates in vivo cardiac function and performance in health and diseaseMonoamine Oxidases as Potential Contributors to Oxidative Stress in Diabetes: Time for a Study in Patients Undergoing Heart SurgeryExcitotoxic insult results in a long-lasting activation of CaMKIIα and mitochondrial damage in living hippocampal neuronsCitrullination of myofilament proteins in heart failure.Silent disease progression in clinically stable heart failure.CaMKII regulation of cardiac ryanodine receptors and inositol triphosphate receptors.Role of RyR2 phosphorylation in heart failure and arrhythmias: protein kinase A-mediated hyperphosphorylation of the ryanodine receptor at serine 2808 does not alter cardiac contractility or cause heart failure and arrhythmiasAstragaloside IV alleviates heart failure via activating PPARα to switch glycolysis to fatty acid β-oxidation.Compensatory and decompensatory alterations in cardiomyocyte Ca2+ dynamics in hearts with diastolic dysfunction following aortic banding.Loss of MD1 exacerbates pressure overload-induced left ventricular structural and electrical remodelling.Cardiac-specific suppression of NF-κB signaling prevents diabetic cardiomyopathy via inhibition of the renin-angiotensin system.Defects in T-tubular electrical activity underlie local alterations of calcium release in heart failureAnkyrin-G coordinates intercalated disc signaling platform to regulate cardiac excitability in vivoDysregulation of intracellular calcium transporters in animal models of sepsis-induced cardiomyopathyMitochondrial Ca(2+) uptake by the voltage-dependent anion channel 2 regulates cardiac rhythmicity.FGF-23 dysregulates calcium homeostasis and electrophysiological properties in HL-1 atrial cells.Abnormalities of capillary microarchitecture in a rat model of coronary ischemic congestive heart failure.Downregulation of connexin43 by microRNA-130a in cardiomyocytes results in cardiac arrhythmias.Chasing cardiac physiology and pathology down the CaMKII cascadePolycystin-1 Is a Cardiomyocyte Mechanosensor That Governs L-Type Ca2+ Channel Protein StabilityCardiac Dysfunction in a Porcine Model of Pediatric Malnutrition.Sarcoendoplasmic reticulum Ca(2+) ATPase. A critical target in chlorine inhalation-induced cardiotoxicity.Contractile Function During Angiotensin-II Activation: Increased Nox2 Activity Modulates Cardiac Calcium Handling via Phospholamban Phosphorylation.Calcium Homeostasis and Organelle Function in the Pathogenesis of Obesity and Diabetes.Recombinant Atrial Natriuretic Peptide Prevents Aberrant Ca2+ Leakage through the Ryanodine Receptor by Suppressing Mitochondrial Reactive Oxygen Species Production Induced by Isoproterenol in Failing Cardiomyocytes.Orphan nuclear receptor Nur77 affects cardiomyocyte calcium homeostasis and adverse cardiac remodellingPhysiological and pathological roles of the mitochondrial permeability transition pore in the heart.Emerin plays a crucial role in nuclear invagination and in the nuclear calcium transientA mathematical model for active contraction in healthy and failing myocytes and left ventricles.Human G109E-inhibitor-1 impairs cardiac function and promotes arrhythmias.Ablation of Matrix Metalloproteinase-9 Prevents Cardiomyocytes Contractile Dysfunction in DiabeticsNanotechnology inspired tools for mitochondrial dysfunction related diseases.Molecular mechanism of activation-triggered subunit exchange in Ca(2+)/calmodulin-dependent protein kinase IIR-CEPIA1er as a new tool to directly measure sarcoplasmic reticulum [Ca] in ventricular myocytes.Progressive decay of Ca2+ homeostasis in the development of diabetic cardiomyopathy.Redox control of enzymatic functions: The electronics of life's circuitry.
P2860
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P2860
description
2013 nî lūn-bûn
@nan
2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Mechanisms of altered Ca²⁺ handling in heart failure
@ast
Mechanisms of altered Ca²⁺ handling in heart failure
@en
Mechanisms of altered Ca²⁺ handling in heart failure
@nl
type
label
Mechanisms of altered Ca²⁺ handling in heart failure
@ast
Mechanisms of altered Ca²⁺ handling in heart failure
@en
Mechanisms of altered Ca²⁺ handling in heart failure
@nl
prefLabel
Mechanisms of altered Ca²⁺ handling in heart failure
@ast
Mechanisms of altered Ca²⁺ handling in heart failure
@en
Mechanisms of altered Ca²⁺ handling in heart failure
@nl
P2860
P3181
P1433
P1476
Mechanisms of altered Ca²⁺ handling in heart failure
@en
P2093
Mark E. Anderson
P2860
P304
P3181
P356
10.1161/CIRCRESAHA.113.301651
P407
P577
2013-08-30T00:00:00Z