Altered expression and activation of signal transducers and activators of transcription (STATs) in hepatitis C virus infection: in vivo and in vitro studies - Wikidata - awgldk - TriplyDB

Altered expression and activation of signal transducers and activators of transcription (STATs) in hepatitis C virus infection: in vivo and in vitro studies

Altered expression and activation of signal transducers and activators of transcription (STATs) in hepatitis C virus infection: in vivo and in vitro studies

http://www.wikidata.org/entity/Q27478325

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Recent molecular insights into rickettsial pathogenesis and immunity Hepatitis C Virus Infection Induces the Beta Interferon Signaling Pathway in Immortalized Human Hepatocytes Heme oxygenase-1 suppresses hepatitis C virus replication and increases resistance of hepatocytes to oxidant injury Oncostatin M Enhances the Antiviral Effects of Type I Interferon and Activates Immunostimulatory Functions in Liver Epithelial Cells Signal transduction pathways in liver and the influence of hepatitis C virus infection on their activities A long-range RNA-RNA interaction between the 5' and 3' ends of the HCV genome Combination antiretroviral therapy (cART) restores HIV-1 infection-mediated impairment of JAK-STAT signaling pathway.Improved dendritic cell-based immunization against hepatitis C virus using peptide inhibitors of interleukin 10.Activated STAT3 is a mediator and biomarker of VEGF endothelial activation Impairment of type I but not type III IFN signaling by hepatitis C virus infection influences antiviral responses in primary human hepatocytes Upregulation of indoleamine 2,3-dioxygenase in hepatitis C virus infection A Versatile Vector for In Vivo Monitoring of Type I Interferon Induction and Signaling.Liver regeneration is suppressed in alcoholic cirrhosis: correlation with decreased STAT3 activation Cardiotrophin-1 defends the liver against ischemia-reperfusion injury and mediates the protective effect of ischemic preconditioning.Anaphylatoxin C5a creates a favorable microenvironment for lung cancer progression.dsRNA-dependent protein kinase PKR and its role in stress, signaling and HCV infection.Cell type-dependent pro- and anti-inflammatory role of signal transducer and activator of transcription 3 in alcoholic liver injury.The interferon inducing pathways and the hepatitis C virus The role of hepatic expression of STAT1, SOCS3 and PIAS1 in the response of chronic hepatitis C patients to therapy Molecular mechanisms of alcoholic fatty liver Anti-HCV RNA Aptamers Targeting the Genomic cis-Acting Replication Element.Between Scylla and Charybdis: the role of the human immune system in the pathogenesis of hepatitis C.Hepatitis C virus-host interactions: Etiopathogenesis and therapeutic strategies.RNA Aptamers as Molecular Tools to Study the Functionality of the Hepatitis C Virus CRE Region.Role of signal transducer and activator of transcription 3 in liver fibrosis progression in chronic hepatitis C-infected patients.RNA aptamer-mediated interference of HCV replication by targeting the CRE-5BSL3.2 domain.HCV innate immune responses.Development of Optimized Inhibitor RNAs Allowing Multisite-Targeting of the HCV Genome.The cis-acting replication element of the Hepatitis C virus genome recruits host factors that influence viral replication and translation Enhanced therapeutic effect using sequential administration of antigenically distinct oncolytic viruses expressing oncostatin M in a Syrian hamster orthotopic pancreatic cancer model.Pegylated interferon-α induced hypoferremia is associated with the immediate response to treatment in hepatitis C.Gene expression profile after knockdown of USP18 in Hepg2.2.15 cells.Differential association of STAT3 and HK-II expression in hepatitis B virus- and hepatitis C virus-related hepatocellular carcinoma.Dysregulation of interferon regulatory factors impairs the expression of immunostimulatory molecules in hepatitis C virus genotype 1-infected hepatocytes.Hepatitis C virus targets the interferon-α JAK/STAT pathway by promoting proteasomal degradation in immune cells and hepatocytes.

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P2860

Altered expression and activation of signal transducers and activators of transcription (STATs) in hepatitis C virus infection: in vivo and in vitro studies

http://www.wikidata.org/entity/Q27478325

description

2006 nî lūn-bûn

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2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2006 թվականի օգոստոսին հրատարակված գիտական հոդված

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2006年の論文

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P2860

Distinct roles of the NH2- and COOH-terminal domains of the protein inhibitor of activated signal transducer and activator of transcription (STAT) 1 (PIAS1) in cytokine-induced PIAS1-Stat1 interaction

Hepatitis C virus expression suppresses interferon signaling by degrading STAT1

STAT3beta, a splice variant of transcription factor STAT3, is a dominant negative regulator of transcription

Interleukin-6-type cytokine signalling through the gp130/Jak/STAT pathway

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Expression of hepatitis C virus proteins inhibits signal transduction through the Jak-STAT pathway

Persistent and transient replication of full-length hepatitis C virus genomes in cell culture

How cells respond to interferons

Signal transduction by type I interferons

The IFN gamma receptor: a paradigm for cytokine receptor signaling

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