beta-Catenin status predicts a favorable outcome in childhood medulloblastoma: the United Kingdom Children's Cancer Study Group Brain Tumour Committee
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Chemotherapy for children with medulloblastomaChemotherapy for children with medulloblastomaThe SFRP family of WNT inhibitors function as novel tumor suppressor genes epigenetically silenced in medulloblastomaCombined MYC and P53 defects emerge at medulloblastoma relapse and define rapidly progressive, therapeutically targetable diseaseMolecular subgroups of medulloblastoma: the current consensusBiological background of pediatric medulloblastoma and ependymoma: a review from a translational research perspectiveAdvances in managing medulloblastoma and intracranial primitive neuro-ectodermal tumorsEcto-5'-Nucleotidase Overexpression Reduces Tumor Growth in a Xenograph Medulloblastoma ModelDNA methylation profiling of medulloblastoma allows robust subclassification and improved outcome prediction using formalin-fixed biopsiesIntegrative genomic analysis of medulloblastoma identifies a molecular subgroup that drives poor clinical outcomeSubtypes of medulloblastoma have distinct developmental originsDevelopment and cancer of the cerebellumChronic viral infection and primary central nervous system malignancy.Cancer diagnosis marker extraction for soft tissue sarcomas based on gene expression profiling data by using projective adaptive resonance theory (PART) filtering method.Integrated genomics identifies five medulloblastoma subtypes with distinct genetic profiles, pathway signatures and clinicopathological features.Amplification and overexpression of Hsa-miR-30b, Hsa-miR-30d and KHDRBS3 at 8q24.22-q24.23 in medulloblastoma.The potential involvement of E-cadherin and beta-catenins in meningioma.Prognostic significance of clinical, histopathological, and molecular characteristics of medulloblastomas in the prospective HIT2000 multicenter clinical trial cohort.WNT/β-catenin signaling regulates mitochondrial activity to alter the oncogenic potential of melanoma in a PTEN-dependent manner.Molecular subgroups of medulloblastoma: an international meta-analysis of transcriptome, genetic aberrations, and clinical data of WNT, SHH, Group 3, and Group 4 medulloblastomas.Genetic alterations in microRNAs in medulloblastomas.Children's Oncology Group's 2013 blueprint for research: central nervous system tumors.Characterization of ectonucleotidases in human medulloblastoma cell lines: ecto-5'NT/CD73 in metastasis as potential prognostic factor.Rapid diagnosis of medulloblastoma molecular subgroups.Downregulation of Wnt2 and beta-catenin by siRNA suppresses malignant glioma cell growth.WNT activation by lithium abrogates TP53 mutation associated radiation resistance in medulloblastomaMedulloblastoma: advances and challenges.WNT3 inhibits cerebellar granule neuron progenitor proliferation and medulloblastoma formation via MAPK activation.Molecular subgroups of medulloblastoma.Prognostic classification of pediatric medulloblastoma based on chromosome 17p loss, expression of MYCC and MYCN, and Wnt pathway activation.Pediatric Brain Tumors: Innovative Genomic Information Is Transforming the Diagnostic and Clinical Landscape.Urokinase-type plasminogen activator receptor (uPAR)-mediated regulation of WNT/β-catenin signaling is enhanced in irradiated medulloblastoma cells.Genetic and molecular alterations across medulloblastoma subgroupsAn interferon response gene expression signature is activated in a subset of medulloblastomasMedulloblastoma: clinicopathological correlates of SHH, WNT, and non-SHH/WNT molecular subgroups.Definition of disease-risk stratification groups in childhood medulloblastoma using combined clinical, pathologic, and molecular variablesAdvances in paediatric cancer treatmentMolecular diagnostics in embryonal brain tumors.Biomarker-driven stratification of disease-risk in non-metastatic medulloblastoma: Results from the multi-center HIT-SIOP-PNET4 clinical trialAberrant patterns of H3K4 and H3K27 histone lysine methylation occur across subgroups in medulloblastoma.
P2860
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P2860
beta-Catenin status predicts a favorable outcome in childhood medulloblastoma: the United Kingdom Children's Cancer Study Group Brain Tumour Committee
description
2005 nî lūn-bûn
@nan
2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
beta-Catenin status predicts a ...... y Group Brain Tumour Committee
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beta-Catenin status predicts a ...... y Group Brain Tumour Committee
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beta-Catenin status predicts a ...... y Group Brain Tumour Committee
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label
beta-Catenin status predicts a ...... y Group Brain Tumour Committee
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beta-Catenin status predicts a ...... y Group Brain Tumour Committee
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beta-Catenin status predicts a ...... y Group Brain Tumour Committee
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beta-Catenin status predicts a ...... y Group Brain Tumour Committee
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beta-Catenin status predicts a ...... y Group Brain Tumour Committee
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beta-Catenin status predicts a ...... y Group Brain Tumour Committee
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P2093
P3181
P356
P1476
beta-Catenin status predicts a ...... y Group Brain Tumour Committee
@en
P2093
Andrew D Pearson
Claire L Weston
David W Ellison
Meryl E Lusher
Olabisi E Onilude
Roger E Taylor
Steven C Clifford
P304
P3181
P356
10.1200/JCO.2005.01.5479
P407
P577
2005-11-01T00:00:00Z