Cardiac adenoviral S100A1 gene delivery rescues failing myocardium - Wikidata - awgldk - TriplyDB

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

http://www.wikidata.org/entity/Q28296403

about

Multifaceted role of β-arrestins in inflammation and disease Gene therapy for cardiovascular disease: advances in vector development, targeting, and delivery for clinical translation Model-specific selection of molecular targets for heart failure gene therapy Mechanisms of altered Ca²⁺ handling in heart failure Cardiovascular gene therapy for myocardial infarction Calcium mishandling in diastolic dysfunction: mechanisms and potential therapies S100A1 and Calmodulin Compete for the Same Binding Site on Ryanodine Receptor S100A1 is released from ischemic cardiomyocytes and signals myocardial damage via Toll-like receptor 4 S100A1 in human heart failure: lack of recovery following left ventricular assist device support The beta-catenin/T-cell factor/lymphocyte enhancer factor signaling pathway is required for normal and stress-induced cardiac hypertrophy Cardiac AAV9-S100A1 gene therapy rescues post-ischemic heart failure in a preclinical large animal model S100A1 and calmodulin regulation of ryanodine receptor in striated muscle S100A1 genetically targeted therapy reverses dysfunction of human failing cardiomyocytes.S100A1 in cardiovascular health and disease: closing the gap between basic science and clinical therapy S100A1 binds to the calmodulin-binding site of ryanodine receptor and modulates skeletal muscle excitation-contraction coupling S100A1: Structure, Function, and Therapeutic Potential S100A1: a multifaceted therapeutic target in cardiovascular disease AAV-mediated gene therapy for heart failure: enhancing contractility and calcium handling Gene therapy delivery systems for enhancing viral and nonviral vectors for cardiac diseases: current concepts and future applications.The inotropic peptide βARKct improves βAR responsiveness in normal and failing cardiomyocytes through G(βγ)-mediated L-type calcium current disinhibition.Differential effects of S100 proteins A2 and A6 on cardiac Ca(2+) cycling and contractile performance.Myoscape controls cardiac calcium cycling and contractility via regulation of L-type calcium channel surface expression.S100A1: a regulator of striated muscle sarcoplasmic reticulum Ca2+ handling, sarcomeric, and mitochondrial function Orai1 and Stim1 regulate normal and hypertrophic growth in cardiomyocytes.Dot1a contains three nuclear localization signals and regulates the epithelial Na+ channel (ENaC) at multiple levels.Cardiac gene therapy Simultaneous administration of insulin-like growth factor-1 and darbepoetin alfa protects the rat myocardium against myocardial infarction and enhances angiogenesis Rescuing the failing heart by targeted gene transfer Parvalbumin isoforms for enhancing cardiac diastolic function.Altered myocardial calcium cycling and energetics in heart failure--a rational approach for disease treatment.Domestication of the cardiac mitochondrion for energy conversion.Oxidative stress in myocardial ischaemia reperfusion injury: a renewed focus on a long-standing area of heart research.Molecular aspects of ischemic heart disease: ischemia/reperfusion-induced genetic changes and potential applications of gene and RNA interference therapy.Potential of gene therapy as a treatment for heart failure S100A1 DNA-based Inotropic Therapy Protects Against Proarrhythmogenic Ryanodine Receptor 2 Dysfunction.Gene therapy in the treatment of heart failure.S100A1: a novel inotropic regulator of cardiac performance. Transition from molecular physiology to pathophysiological relevance.Altered sarcoplasmic reticulum calcium cycling--targets for heart failure therapy.Gene therapy in heart failure.Functions of S100 proteins.

P2860

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P2860

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

http://www.wikidata.org/entity/Q28296403

description

2004 nî lūn-bûn

@nan

2004 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2004 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2004年の論文

@ja

2004年論文

@yue

2004年論文

@zh-hant

2004年論文

@zh-hk

2004年論文

@zh-mo

2004年論文

@zh-tw

2004年论文

@wuu

name

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

@ast

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

@en

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

@nl

type

label

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

@ast

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

@en

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

@nl

prefLabel

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

@ast

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

@en

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

@nl

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P1433

Journal of Clinical Investigation

P1476

Cardiac adenoviral S100A1 gene delivery rescues failing myocardium

@en

P2093

Andrea D Eckhart

http://www.w3.org/2001/XMLSchema#string

Andrew Remppis

http://www.w3.org/2001/XMLSchema#string

Beatrix Heidt

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Dieter Weichenhan

http://www.w3.org/2001/XMLSchema#string

Eva Löffler

http://www.w3.org/2001/XMLSchema#string

Hugo A Katus

http://www.w3.org/2001/XMLSchema#string

Jeffrey Martini

http://www.w3.org/2001/XMLSchema#string

Matthew L Williams

http://www.w3.org/2001/XMLSchema#string

Mirko Völkers

http://www.w3.org/2001/XMLSchema#string

Patrick Most

http://www.w3.org/2001/XMLSchema#string

P2860

Restoration of contractile function in isolated cardiomyocytes from failing human hearts by gene transfer of SERCA2a.

Ca2+ -dependent interaction of S100A1 with the sarcoplasmic reticulum Ca2+ -ATPase2a and phospholamban in the human heart

S100A1: a regulator of myocardial contractility

A simplified system for generating recombinant adenoviruses

The myocardial protein S100A1 plays a role in the maintenance of normal gene expression in the adult heart

Intracellular and extracellular roles of S100 proteins

Extracellular S100A1 protein inhibits apoptosis in ventricular cardiomyocytes via activation of the extracellular signal-regulated protein kinase 1/2 (ERK1/2)

Transgenic overexpression of the Ca2+-binding protein S100A1 in the heart leads to increased in vivo myocardial contractile performance

Impaired cardiac contractility response to hemodynamic stress in S100A1-deficient mice

Cardiac excitation-contraction coupling

P304

1550-63

http://www.w3.org/2001/XMLSchema#string

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scholarly article

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559266

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10.1172/JCI21454

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11

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114

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P50

Melanie Boerries

P577

2004-12-01T00:00:00Z

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15578088

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P932

529280

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