T cell cytokine pattern in the joints of patients with Lyme arthritis and its regulation by cytokines and anticytokines
about
Decreased up-regulation of the interleukin-12Rbeta2-chain and interferon-gamma secretion and increased number of forkhead box P3-expressing cells in patients with a history of chronic Lyme borreliosis compared with asymptomatic Borrelia-exposed indiLyme disease and current aspects of immunization.Pulmonary and systemic immune response to inhaled multiwalled carbon nanotubesBorrelia burgdorferi stimulates the production of interleukin-10 in peripheral blood mononuclear cells from uninfected humans and rhesus monkeys.Borrelia burgdorferi spirochetes induce mast cell activation and cytokine release.Decreased Th1-type inflammatory cytokine expression in the skin is associated with persisting symptoms after treatment of erythema migrans.Interleukin-10 modulates proinflammatory cytokines in the human monocytic cell line THP-1 stimulated with Borrelia burgdorferi lipoproteins.Modulation of cytokine release in ex vivo-stimulated blood from borreliosis patients.Activation of endothelium by Borrelia burgdorferi in vitro enhances transmigration of specific subsets of T lymphocytesCoinfection with Borrelia burgdorferi and the agent of human granulocytic ehrlichiosis alters murine immune responses, pathogen burden, and severity of Lyme arthritis.Gamma interferon is not required for arthritis resistance in the murine Lyme disease modelAutocrine and exocrine regulation of interleukin-10 production in THP-1 cells stimulated with Borrelia burgdorferi lipoproteins.Murine Lyme arthritis development mediated by p38 mitogen-activated protein kinase activity.Borrelia burgdorferi-induced inflammation facilitates spirochete adaptation and variable major protein-like sequence locus recombinationIndications of Th1 and Th17 responses in cerebrospinal fluid from patients with Lyme neuroborreliosis: a large retrospective study.Interleukin-10 anti-inflammatory response to Borrelia burgdorferi, the agent of Lyme disease: a possible role for suppressors of cytokine signaling 1 and 3.T-helper-cell cytokines in the early evolution of murine Lyme arthritisInterleukin-10 alters effector functions of multiple genes induced by Borrelia burgdorferi in macrophages to regulate Lyme disease inflammationPopulations of human T lymphocytes that traverse the vascular endothelium stimulated by Borrelia burgdorferi are enriched with cells that secrete gamma interferon.Adenoviral delivery of interleukin-10 fails to attenuate experimental Lyme diseaseEmergence of chronic Lyme arthritis: putting the breaks on CD28 costimulation.Persistent arthritis in Borrelia burgdorferi-infected HLA-DR4-positive CD28-negative mice post-antibiotic treatment.Can ELISPOT Be Applied to A Clinical Setting as A Diagnostic Utility for Neuroborreliosis?Borrelia burgdorferi and interleukin-1 promote the transendothelial migration of monocytes in vitro by different mechanisms.Lipopeptides of Borrelia burgdorferi outer surface proteins induce Th1 phenotype development in alphabeta T-cell receptor transgenic mice.Arthritis is inhibited in Borrelia-primed and infected interleukin-17A-deficient mice after administration of anti-gamma-interferon, anti-tumor necrosis factor alpha and anti-interleukin-6 antibodies.Borrelia-primed and -infected mice deficient of interleukin-17 develop arthritis after neutralization of gamma-interferon.CD4+ cell-derived interleukin-17 in a model of dysregulated, Borrelia-induced arthritis.Arthritis is developed in Borrelia-primed and -infected mice deficient of interleukin-17.CD83 Modulates B Cell Activation and Germinal Center Responses.Interleukin-10 (IL-10) inhibits Borrelia burgdorferi-induced IL-17 production and attenuates IL-17-mediated Lyme arthritis.The Th1-Th2 model--what relevance to inflammatory arthritis?Downregulation of class II molecules on epidermal Langerhans cells in Lyme borreliosis.Mercury exposure as a model for deviation of cytokine responses in experimental Lyme arthritis: HgCl2 treatment decreases T helper cell type 1-like responses and arthritis severity but delays eradication of Borrelia burgdorferi in C3H/HeN mice.Reduced number of interleukin-12 secreting cells in patients with Lyme borreliosis previously exposed to Anaplasma phagocytophilum.Innate immune responses in Lyme borreliosis: enhanced tumour necrosis factor-alpha and interleukin-12 in asymptomatic individuals in response to live spirochetes.
P2860
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P2860
T cell cytokine pattern in the joints of patients with Lyme arthritis and its regulation by cytokines and anticytokines
description
1997 nî lūn-bûn
@nan
1997 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
name
T cell cytokine pattern in the ...... by cytokines and anticytokines
@ast
T cell cytokine pattern in the ...... by cytokines and anticytokines
@en
T cell cytokine pattern in the ...... by cytokines and anticytokines
@nl
type
label
T cell cytokine pattern in the ...... by cytokines and anticytokines
@ast
T cell cytokine pattern in the ...... by cytokines and anticytokines
@en
T cell cytokine pattern in the ...... by cytokines and anticytokines
@nl
prefLabel
T cell cytokine pattern in the ...... by cytokines and anticytokines
@ast
T cell cytokine pattern in the ...... by cytokines and anticytokines
@en
T cell cytokine pattern in the ...... by cytokines and anticytokines
@nl
P2093
P356
P1476
T cell cytokine pattern in the ...... by cytokines and anticytokines
@en
P2093
P356
10.1002/ART.1780400111
P407
P577
1997-01-01T00:00:00Z