Gamma interferon is not required for arthritis resistance in the murine Lyme disease model
about
Relative contributions of innate and acquired host responses to bacterial control and arthritis development in Lyme diseaseRole of osteopontin in murine Lyme arthritis and host defense against Borrelia burgdorferi.Different patterns of expression and of IL-10 modulation of inflammatory mediators from macrophages of Lyme disease-resistant and -susceptible mice.A critical role for type I IFN in arthritis development following Borrelia burgdorferi infection of mice.Murine Lyme arthritis development mediated by p38 mitogen-activated protein kinase activity.Control of Borrelia burgdorferi-specific CD4+-T-cell effector function by interleukin-12- and T-cell receptor-induced p38 mitogen-activated protein kinase activityAnti-p19 antibody treatment exacerbates lyme arthritis and enhances borreliacidal activity.Phagocytosis of Borrelia burgdorferi and Treponema pallidum potentiates innate immune activation and induces gamma interferon productionPhagocytosis of Borrelia burgdorferi, the Lyme disease spirochete, potentiates innate immune activation and induces apoptosis in human monocytesLyme arthritis: current concepts and a change in paradigm.Local production of IFN-gamma by invariant NKT cells modulates acute Lyme carditisAssociation of CD4+ CD25+ T cells with prevention of severe destructive arthritis in Borrelia burgdorferi-vaccinated and challenged gamma interferon-deficient mice treated with anti-interleukin-17 antibody.Borrelia species induce inflammasome activation and IL-17 production through a caspase-1-dependent mechanismInterferon-γ influences the composition of leukocytic infiltrates in murine lyme carditis.Aggravated Lyme carditis in CD11a-/- and CD11c-/- mice.Humoral immunity reflects altered T helper cell bias in Borrelia burgdorferi-infected gamma delta T-cell-deficient mice.Arthritis is inhibited in Borrelia-primed and infected interleukin-17A-deficient mice after administration of anti-gamma-interferon, anti-tumor necrosis factor alpha and anti-interleukin-6 antibodies.Localized production of IL-10 suppresses early inflammatory cell infiltration and subsequent development of IFN-γ-mediated Lyme arthritisBeta2 integrins control the severity of murine Lyme carditis.IL-10 Deficiency Reveals a Role for TLR2-Dependent Bystander Activation of T Cells in Lyme Arthritis.
P2860
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P2860
Gamma interferon is not required for arthritis resistance in the murine Lyme disease model
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Gamma interferon is not requir ...... the murine Lyme disease model
@ast
Gamma interferon is not requir ...... the murine Lyme disease model
@en
Gamma interferon is not requir ...... the murine Lyme disease model
@nl
type
label
Gamma interferon is not requir ...... the murine Lyme disease model
@ast
Gamma interferon is not requir ...... the murine Lyme disease model
@en
Gamma interferon is not requir ...... the murine Lyme disease model
@nl
prefLabel
Gamma interferon is not requir ...... the murine Lyme disease model
@ast
Gamma interferon is not requir ...... the murine Lyme disease model
@en
Gamma interferon is not requir ...... the murine Lyme disease model
@nl
P2093
P2860
P1476
Gamma interferon is not requir ...... the murine Lyme disease model
@en
P2093
L Glickstein
M Edelstein
P2860
P304
P356
10.1128/IAI.69.6.3737-3743.2001
P407
P577
2001-06-01T00:00:00Z