Functional consequences of the arrhythmogenic G306R KvLQT1 K+ channel mutant probed by viral gene transfer in cardiomyocytes
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SUMOylation determines the voltage required to activate cardiac IKs channels.Proarrhythmic potential of halofantrine, terfenadine and clofilium in a modified in vivo model of torsade de pointes.Phospholamban as a crucial determinant of the inotropic response of human pluripotent stem cell-derived ventricular cardiomyocytes and engineered 3-dimensional tissue constructs.
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Functional consequences of the arrhythmogenic G306R KvLQT1 K+ channel mutant probed by viral gene transfer in cardiomyocytes
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2001 nî lūn-bûn
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2001 թուականի Մայիսին հրատարակուած գիտական յօդուած
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2001 թվականի մայիսին հրատարակված գիտական հոդված
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2001年の論文
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2001年論文
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2001年論文
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2001年論文
@zh-hk
2001年論文
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2001年論文
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2001年论文
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Functional consequences of the ...... ene transfer in cardiomyocytes
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Functional consequences of the ...... ene transfer in cardiomyocytes
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Functional consequences of the ...... ene transfer in cardiomyocytes
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Functional consequences of the ...... ene transfer in cardiomyocytes
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Functional consequences of the ...... ene transfer in cardiomyocytes
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Functional consequences of the ...... ene transfer in cardiomyocytes
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Functional consequences of the ...... ene transfer in cardiomyocytes
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Functional consequences of the ...... ene transfer in cardiomyocytes
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Functional consequences of the ...... ene transfer in cardiomyocytes
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Functional consequences of the ...... ene transfer in cardiomyocytes
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10.1111/J.1469-7793.2001.0127B.X
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2001-05-15T00:00:00Z