Nox4 and redox signaling mediate TGF-β-induced endothelial cell apoptosis and phenotypic switch - Wikidata - awgldk - TriplyDB

Nox4 and redox signaling mediate TGF-β-induced endothelial cell apoptosis and phenotypic switch

Nox4 and redox signaling mediate TGF-β-induced endothelial cell apoptosis and phenotypic switch

http://www.wikidata.org/entity/Q28387033

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Suppression of NADPH Oxidase Activity May Slow the Expansion of Osteolytic Bone Metastases Regulatory RNAs controlling vascular (dys)function by affecting TGF-ß family signalling Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosis PTP1B inhibitor promotes endothelial cell motility by activating the DOCK180/Rac1 pathway.Redox-sensitive MAPK and Notch3 regulate fibroblast differentiation and activation: a dual role of ERK1/2 4-O-Methylhonokiol Protects HaCaT Cells from TGF-β1-Induced Cell Cycle Arrest by Regulating Canonical and Non-Canonical Pathways of TGF-β Signaling Polymerase δ-interacting protein 2 promotes postischemic neovascularization of the mouse hindlimb The antioxidant compound tert-butylhydroquinone activates Akt in myocardium, suppresses apoptosis and ameliorates pressure overload-induced cardiac dysfunction Kallistatin inhibits TGF-β-induced endothelial-mesenchymal transition by differential regulation of microRNA-21 and eNOS expression.Excess TGF-β mediates muscle weakness associated with bone metastases in mice.Transforming growth factor-β mediates endothelial dysfunction in rats during high salt intake.Subcutaneous administration of bovine superoxide dismutase protects lungs from radiation-induced lung injury.The Expression of NOX4 in Smooth Muscles of Small Airway Correlates with the Disease Severity of COPD Metabolic pathways regulated by TAp73 in response to oxidative stress Nitric oxide-mediated apoptosis of neutrophils through caspase-8 and caspase-3-dependent mechanism Systems Genetics Approach Identifies Gene Pathways and Adamts2 as Drivers of Isoproterenol-Induced Cardiac Hypertrophy and Cardiomyopathy in Mice Role of NADPH Oxidase-4 in Human Endothelial Progenitor Cells.Cytotoxin-induced NADPH oxides activation: roles in regulation of cell death.TGF-beta1 regulates human brain pericyte inflammatory processes involved in neurovasculature function.ROS signaling and redox biology in endothelial cells.Genome-wide association analysis identifies novel blood pressure loci and offers biological insights into cardiovascular risk.The Future of Onco-Cardiology: We Are Not Just "Side Effect Hunters".What has passed is prolog: new cellular and physiological roles of G6PD.Tert-butyl hydroperoxide (t-BHP) induced apoptosis and necroptosis in endothelial cells: Roles of NOX4 and mitochondrion.Protein Phosphotyrosine Phosphatase 1B (PTP1B) in Calpain-dependent Feedback Regulation of Vascular Endothelial Growth Factor Receptor (VEGFR2) in Endothelial Cells: IMPLICATIONS IN VEGF-DEPENDENT ANGIOGENESIS AND DIABETIC WOUND HEALING.Context-dependent function of ROS in the vascular endothelium: The role of the Notch pathway and shear stress.Precision-cut rat, mouse, and human intestinal slices as novel models for the early-onset of intestinal fibrosis Insights into the molecular mechanisms of diabetes-induced endothelial dysfunction: focus on oxidative stress and endothelial progenitor cells.Smad-independent pathway involved in transforming growth factor β1-induced Nox4 expression and proliferation of endothelial cells.MicroRNA‑126 inhibits endothelial permeability and apoptosis in apolipoprotein E‑knockout mice fed a high‑fat diet Oxidative stress caused by activation of NADPH oxidase 4 promotes contrast-induced acute kidney injury.EDIL3 depletion suppress epithelial-mesenchymal transition of lens epithelial cells via transforming growth factor β pathway.Improvement of vascular dysfunction by argirein through inhibiting endothelial cell apoptosis associated with ET-1/Nox4 signal pathway in diabetic rats

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Nox4 and redox signaling mediate TGF-β-induced endothelial cell apoptosis and phenotypic switch

http://www.wikidata.org/entity/Q28387033

description

2014 nî lūn-bûn

@nan

2014 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2014 թվականի հունվարին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Nox4 and redox signaling media ...... poptosis and phenotypic switch

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Nox4 and redox signaling media ...... poptosis and phenotypic switch

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Nox4 and redox signaling media ...... poptosis and phenotypic switch

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Nox4 and redox signaling media ...... poptosis and phenotypic switch

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Nox4 and redox signaling media ...... poptosis and phenotypic switch

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Cell Death and Disease

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Nox4 and redox signaling media ...... poptosis and phenotypic switch

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F Yan

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G J Dusting

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H M Peshavariya

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P2860

Cyclic stretch stimulates vascular smooth muscle cell alignment by redox-dependent activation of Notch3

Transforming growth factor-beta1 (TGF-beta)-induced apoptosis of prostate cancer cells involves Smad7-dependent activation of p38 by TGF-beta-activated kinase 1 and mitogen-activated protein kinase kinase 3

Reactive oxygen species, cellular redox systems, and apoptosis

Upregulation of the NADPH oxidase NOX4 by TGF-beta in hepatocytes is required for its pro-apoptotic activity

ALK5- and TGFBR2-independent role of ALK1 in the pathogenesis of hereditary hemorrhagic telangiectasia type 2

Dll4 signalling through Notch1 regulates formation of tip cells during angiogenesis

Nox4 overexpression activates reactive oxygen species and p38 MAPK in human endothelial cells.

NADPH oxidase 1 modulates WNT and NOTCH1 signaling to control the fate of proliferative progenitor cells in the colon

Elevated systemic TGF-beta impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(-/-) mice

NOX4/NADPH oxidase expression is increased in pulmonary fibroblasts from patients with idiopathic pulmonary fibrosis and mediates TGFbeta1-induced fibroblast differentiation into myofibroblasts.

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10.1038/CDDIS.2013.551

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