BMP receptor IA is required in mammalian neural crest cells for development of the cardiac outflow tract and ventricular myocardium
about
Transcription factor genes Smad4 and Gata4 cooperatively regulate cardiac valve development. [corrected]Identification of receptors and signaling pathways for orphan bone morphogenetic protein/growth differentiation factor ligands based on genomic analysesFactors controlling cardiac neural crest cell migrationMesenchymal Stem Cells for Cardiac Regenerative Therapy: Optimization of Cell Differentiation StrategyThe neural crest in cardiac congenital anomaliesCurrent perspectives of the signaling pathways directing neural crest inductionTbx1 regulates the BMP-Smad1 pathway in a transcription independent manner.Murine Jagged1/Notch signaling in the second heart field orchestrates Fgf8 expression and tissue-tissue interactions during outflow tract developmentDefective ALK5 signaling in the neural crest leads to increased postmigratory neural crest cell apoptosis and severe outflow tract defectsAtrioventricular cushion transformation is mediated by ALK2 in the developing mouse heart.A heart-hand syndrome gene: Tfap2b plays a critical role in the development and remodeling of mouse ductus arteriosus and limb patterningBMP signaling regulates sympathetic nervous system development through Smad4-dependent and -independent pathwaysBMP type II receptor regulates positioning of outflow tract and remodeling of atrioventricular cushion during cardiogenesisCanonical Wnt signaling functions in second heart field to promote right ventricular growthBMP4 is required in the anterior heart field and its derivatives for endocardial cushion remodeling, outflow tract septation, and semilunar valve developmentOutflow tract cushions perform a critical valve-like function in the early embryonic heart requiring BMPRIA-mediated signaling in cardiac neural crestAn in vivo reporter of BMP signaling in organogenesis reveals targets in the developing kidney.Precardiac deletion of Numb and Numblike reveals renewal of cardiac progenitorsA purified population of multipotent cardiovascular progenitors derived from primate pluripotent stem cells engrafts in postmyocardial infarcted nonhuman primates.Bmp2 and Bmp4 genetically interact to support multiple aspects of mouse development including functional heart development.Neural crest cell signaling pathways critical to cranial bone development and pathology.Genetic interaction between Bmp2 and Bmp4 reveals shared functions during multiple aspects of mouse organogenesis.BMPRIA mediated signaling is essential for temporomandibular joint development in mice.Trigenic neural crest-restricted Smad7 over-expression results in congenital craniofacial and cardiovascular defects.A Tlx2-Cre mouse line uncovers essential roles for hand1 in extraembryonic and lateral mesoderm.Bmpr1a signaling plays critical roles in palatal shelf growth and palatal bone formation.Redundant and dosage sensitive requirements for Fgf3 and Fgf10 in cardiovascular developmentPax3 is essential for normal cardiac neural crest morphogenesis but is not required during migration nor outflow tract septation.TGFβ superfamily signaling in the neural crest lineage.Combinatorial transcriptional interaction within the cardiac neural crest: a pair of HANDs in heart formation.Defective decapentaplegic signaling results in heart overgrowth and reduced cardiac output in DrosophilaGenetic pathways to mammalian heart development: Recent progress from manipulation of the mouse genome.Cooperation between the PDGF receptors in cardiac neural crest cell migration.Characterization of transcription factor AP-2 β mutations involved in familial isolated patent ductus arteriosus suggests haploinsufficiency.cKit+ cardiac progenitors of neural crest origin.Distinct roles of Wnt/beta-catenin and Bmp signaling during early cardiogenesis.Identification of exercise capacity QTL using association mapping in inbred miceNeural crest stem cell maintenance by combinatorial Wnt and BMP signaling.Maternal diabetes induces congenital heart defects in mice by altering the expression of genes involved in cardiovascular developmentReduced bone morphogenetic protein receptor type 1A signaling in neural-crest-derived cells causes facial dysmorphism.
P2860
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P2860
BMP receptor IA is required in mammalian neural crest cells for development of the cardiac outflow tract and ventricular myocardium
description
2004 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մայիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2004
@ast
im Mai 2004 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2004/05/01)
@sk
vědecký článek publikovaný v roce 2004
@cs
wetenschappelijk artikel (gepubliceerd op 2004/05/01)
@nl
наукова стаття, опублікована в травні 2004
@uk
مقالة علمية (نشرت في مايو 2004)
@ar
name
BMP receptor IA is required in ...... act and ventricular myocardium
@ast
BMP receptor IA is required in ...... act and ventricular myocardium
@en
BMP receptor IA is required in ...... act and ventricular myocardium
@nl
type
label
BMP receptor IA is required in ...... act and ventricular myocardium
@ast
BMP receptor IA is required in ...... act and ventricular myocardium
@en
BMP receptor IA is required in ...... act and ventricular myocardium
@nl
prefLabel
BMP receptor IA is required in ...... act and ventricular myocardium
@ast
BMP receptor IA is required in ...... act and ventricular myocardium
@en
BMP receptor IA is required in ...... act and ventricular myocardium
@nl
P2093
P2860
P921
P356
P1433
P1476
BMP receptor IA is required in ...... act and ventricular myocardium
@en
P2093
Erik N. Meyers
John Klingensmith
Murim Choi
Rolf W. Stottmann
Yuji Mishina
P2860
P304
P356
10.1242/DEV.01086
P407
P577
2004-05-01T00:00:00Z