The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
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Tmem100, an ALK1 receptor signaling-dependent gene essential for arterial endothelium differentiation and vascular morphogenesisRBP-Jkappa/SHARP recruits CtIP/CtBP corepressors to silence Notch target genesRepression of androgen receptor activity by HEYL, a third member of the Hairy/Enhancer-of-split-related family of Notch effectorsSegmental expression of Notch and Hairy genes in nephrogenesisSlug is a direct Notch target required for initiation of cardiac cushion cellularizationNotch signaling is required for maintaining stem-cell features of neuroprogenitor cells derived from human embryonic stem cellsHey basic helix-loop-helix transcription factors are repressors of GATA4 and GATA6 and restrict expression of the GATA target gene ANF in fetal heartsDifferential effects of the HESR/HEY transcription factor family on dopamine transporter reporter gene expression via variable number of tandem repeatsHey1 basic helix-loop-helix protein plays an important role in mediating BMP9-induced osteogenic differentiation of mesenchymal progenitor cellsDelta-like ligand 4 (Dll4) is induced by VEGF as a negative regulator of angiogenic sproutingKSHV-Mediated Angiogenesis in Tumor ProgressionFunctional and Biological Role of Endothelial Precursor Cells in Tumour Progression: A New Potential Therapeutic Target in Haematological MalignanciesMolecular control of endothelial cell behaviour during blood vessel morphogenesisStability and function of adult vasculature is sustained by Akt/Jagged1 signalling axis in endothelium.Physiological notch signaling maintains bone homeostasis via RBPjk and Hey upstream of NFATc1KCTD10 is involved in the cardiovascular system and Notch signaling during early embryonic developmentHaploinsufficiency of the NOTCH1 Receptor as a Cause of Adams-Oliver Syndrome With Variable Cardiac AnomaliesEndothelial expression of the Notch ligand Jagged1 is required for vascular smooth muscle developmentBiodistribution and toxicity of pegylated single wall carbon nanotubes in pregnant mice.Haploinsufficient lethality and formation of arteriovenous malformations in Notch pathway mutantsTarget gene analysis by microarrays and chromatin immunoprecipitation identifies HEY proteins as highly redundant bHLH repressorsArtery and vein size is balanced by Notch and ephrin B2/EphB4 during angiogenesisTargeted deletion of Hand2 in cardiac neural crest-derived cells influences cardiac gene expression and outflow tract developmentOverexpression of delta-like 4 induces arterialization and attenuates vessel formation in developing mouse embryosHey2 regulation by FGF provides a Notch-independent mechanism for maintaining pillar cell fate in the organ of CortiHeyL promotes neuronal differentiation of neural progenitor cellsSuppression of Notch signalling by the COUP-TFII transcription factor regulates vein identityEssential role of endothelial Notch1 in angiogenesisHes1 expression is reduced in Tbx1 null cells and is required for the development of structures affected in 22q11 deletion syndromeMammalian Llgl2 is necessary for proper branching morphogenesis during placental developmentHOXA13 Is essential for placental vascular patterning and labyrinth endothelial specificationEmbryonic arrest at midgestation and disruption of Notch signaling produced by the absence of both epsin 1 and epsin 2 in miceHeyL regulates the number of TrkC neurons in dorsal root gangliaThe essential haematopoietic transcription factor Scl is also critical for neuronal developmentDosage-sensitive requirement for mouse Dll4 in artery developmentOtt1 (Rbm15) is essential for placental vascular branching morphogenesis and embryonic development of the heart and spleenThe PIAS-like protein Zimp10 is essential for embryonic viability and proper vascular developmentAnkrd17, an ubiquitously expressed ankyrin factor, is essential for the vascular integrity during embryogenesisTranscription factor CHF1/Hey2 regulates coronary vascular maturationGeneChip analysis of human embryonic stem cell differentiation into hemangioblasts: an in silico dissection of mixed phenotypes.
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The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
description
2004 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2004
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im April 2004 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 2004/04/15)
@sk
vědecký článek publikovaný v roce 2004
@cs
wetenschappelijk artikel (gepubliceerd op 2004/04/15)
@nl
наукова стаття, опублікована у квітні 2004
@uk
مقالة علمية (نشرت في 15-4-2004)
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name
The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
@ast
The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
@en
The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
@nl
type
label
The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
@ast
The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
@en
The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
@nl
prefLabel
The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
@ast
The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
@en
The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
@nl
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P3181
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P1433
P1476
The Notch target genes Hey1 and Hey2 are required for embryonic vascular development
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P2093
Andreas Fischer
Manfred Maier
Nina Schumacher
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P304
P3181
P356
10.1101/GAD.291004
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2004-04-15T00:00:00Z