Effects of synaptic modulation on beta-amyloid, synaptophysin, and memory performance in Alzheimer's disease transgenic mice. - Wikidata - awgldk - TriplyDB

Effects of synaptic modulation on beta-amyloid, synaptophysin, and memory performance in Alzheimer's disease transgenic mice.

Effects of synaptic modulation on beta-amyloid, synaptophysin, and memory performance in Alzheimer's disease transgenic mice.

http://www.wikidata.org/entity/Q30497246

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Lesion of the subiculum reduces the spread of amyloid beta pathology to interconnected brain regions in a mouse model of Alzheimer's disease Connectivity of Pathology: The Olfactory System as a Model for Network-Driven Mechanisms of Alzheimer's Disease Pathogenesis Use of Benzodiazepines in Alzheimer's Disease: A Systematic Review of Literature The multifaceted nature of amyloid precursor protein and its proteolytic fragments: friends and foes Trafficking and proteolytic processing of APP Aβ induces astrocytic glutamate release, extrasynaptic NMDA receptor activation, and synaptic loss The type of Aβ-related neuronal degeneration differs between amyloid precursor protein (APP23) and amyloid β-peptide (APP48) transgenic mice Amyloid proteotoxicity initiates an inflammatory response blocked by cannabinoids.Back to the future with phenotypic screening Pyroglutamate-amyloid-β and glutaminyl cyclase are colocalized with amyloid-β in secretory vesicles and undergo activity-dependent, regulated secretion.Abnormal vibrissa-related behavior and loss of barrel field inhibitory neurons in 5xFAD transgenics.E3 ubiquitin ligase Siah-1 downregulates synaptophysin expression under high glucose and hypoxia.Il10 deficiency rebalances innate immunity to mitigate Alzheimer-like pathology.Impaired β-amyloid secretion in Alzheimer's disease pathogenesis.Amyloid β: linking synaptic plasticity failure to memory disruption in Alzheimer's disease Synaptic activity and Alzheimer's disease: a critical update.Critical role of intraneuronal Aβ in Alzheimer's disease: technical challenges in studying intracellular Aβ.The protection of acetylcholinesterase inhibitor on β-amyloid-induced the injury of neurite outgrowth via regulating axon guidance related genes expression in neuronal cells.Modulation of 5-lipoxygenase in proteotoxicity and Alzheimer's disease.Early enriched environment exposure protects spatial memory and accelerates amyloid plaque formation in APP(Swe)/PS1(L166P) mice.Consequences of cancer treatments on adult hippocampal neurogenesis: implications for cognitive function and depressive symptoms The influence of synaptic activity on neuronal health.New therapeutic targets in Alzheimer's disease: brain deregulation of calcium and zinc.Convergence of synapses, endosomes, and prions in the biology of neurodegenerative diseases.β-Amyloid peptides and amyloid plaques in Alzheimer's disease.Synaptic activity protects against AD and FTD-like pathology via autophagic-lysosomal degradation.The Luminescent Oligothiophene p-FTAA Converts Toxic Aβ1-42 Species into Nontoxic Amyloid Fibers with Altered Properties.Rapid β-amyloid deposition and cognitive impairment after cholinergic denervation in APP/PS1 mice.Effects of selegiline, a monoamine oxidase B inhibitor, on differentiation of P19 embryonal carcinoma stem cells, into neuron-like cells.Regulation of Synaptic Amyloid-β Generation through BACE1 Retrograde Transport in a Mouse Model of Alzheimer's Disease.Aβ accumulation causes MVB enlargement and is modelled by dominant negative VPS4A.Intracellular distribution of amyloid beta peptide and its relationship to the lysosomal system.Intermittent hypoxia training: Powerful, non-invasive cerebroprotection against ethanol withdrawal excitotoxicity.Effects of repetitive exposure to anesthetics and analgesics in the Tg2576 mouse Alzheimer's model.Rescue of long-range circuit dysfunction in Alzheimer's disease models.Brain activity and Alzheimer's disease: a complex relationship.Intraneuronal Aβ accumulation, amyloid plaques, and synapse pathology in Alzheimer's disease.Mitochondrial Dysfunction Triggers Synaptic Deficits via Activation of p38 MAP Kinase Signaling in Differentiated Alzheimer's Disease Trans-Mitochondrial Cybrid Cells.Synaptophysin and the dopaminergic system in hippocampus are involved in the protective effect of rutin against trimethyltin-induced learning and memory impairment.Trans-Synaptic Spread of Amyloid-β in Alzheimer's Disease: Paths to β-Amyloidosis.

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Effects of synaptic modulation on beta-amyloid, synaptophysin, and memory performance in Alzheimer's disease transgenic mice.

http://www.wikidata.org/entity/Q30497246

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2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Effects of synaptic modulation ...... mer's disease transgenic mice.

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Effects of synaptic modulation ...... mer's disease transgenic mice.

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Effects of synaptic modulation ...... mer's disease transgenic mice.

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Effects of synaptic modulation ...... mer's disease transgenic mice.

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Effects of synaptic modulation ...... mer's disease transgenic mice.

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JNEUROSCI.3383-10.2010

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Journal of Neuroscience

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Effects of synaptic modulation ...... imer's disease transgenic mice

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Davide Tampellini

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Fangmin Yu

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Magali Dumont

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Michael T Lin

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Zhenyong Huang

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P2860

Days to criterion as an indicator of toxicity associated with human Alzheimer amyloid-beta oligomers

Amyloid-beta dynamics are regulated by orexin and the sleep-wake cycle

Physical basis of cognitive alterations in Alzheimer's disease: synapse loss is the major correlate of cognitive impairment

Alzheimer's disease is a synaptic failure

Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction

Molecular, structural, and functional characterization of Alzheimer's disease: evidence for a relationship between default activity, amyloid, and memory

APP processing and synaptic function

High-level neuronal expression of abeta 1-42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation

Aberrant excitatory neuronal activity and compensatory remodeling of inhibitory hippocampal circuits in mouse models of Alzheimer's disease

Reduction of oxidative stress, amyloid deposition, and memory deficit by manganese superoxide dismutase overexpression in a transgenic mouse model of Alzheimer's disease.

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10.1523/JNEUROSCI.3383-10.2010

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Gunnar K. Gouras

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2010-10-01T00:00:00Z

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20980585

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Alzheimer's disease

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2972675

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