about
The effect of Rho kinase inhibition on long-term keratinocyte proliferation is rapid and conditional.The mitotic chromosome binding activity of the papillomavirus E2 protein correlates with interaction with the cellular chromosomal protein, Brd4.The Ancient Evolutionary History of PolyomavirusesThe Role of the DNA Damage Response throughout the Papillomavirus Life CycleThe papillomavirus E1 helicase activates a cellular DNA damage response in viral replication fociHuman keratinocytes are efficiently immortalized by a Rho kinase inhibitorThe Papillomavirus Episteme: a central resource for papillomavirus sequence data and analysis.Papillomavirus genomes associate with BRD4 to replicate at fragile sites in the host genome.Conditional mutations in the mitotic chromosome binding function of the bovine papillomavirus type 1 E2 protein.Proteasome-mediated degradation of the papillomavirus E2-TA protein is regulated by phosphorylation and can modulate viral genome copy number.Reconstitution of papillomavirus E2-mediated plasmid maintenance in Saccharomyces cerevisiae by the Brd4 bromodomain protein.Interaction of bovine papillomavirus E2 protein with Brd4 stabilizes its association with chromatin.ROCK inhibitor and feeder cells induce the conditional reprogramming of epithelial cellsVariations in the association of papillomavirus E2 proteins with mitotic chromosomesComplete genome sequence of a tenth human polyomavirus.Snapshots: chromatin control of viral infection.Brd4: tethering, segregation and beyond.Embryonic mesoderm and endoderm induction requires the actions of non-embryonic Nodal-related ligands and Mxtx2The Promise of Proteomics in the Study of Oncogenic Viruses.PSORS2 is due to mutations in CARD14.Papillomaviruses use recombination-dependent replication to vegetatively amplify their genomes in differentiated cellsBrd4 is displaced from HPV replication factories as they expand and amplify viral DNABrd4 is required for e2-mediated transcriptional activation but not genome partitioning of all papillomaviruses.Replication and partitioning of papillomavirus genomes.A proteomic approach to discover and compare interacting partners of papillomavirus E2 proteins from diverse phylogenetic groupsThe role of integration in oncogenic progression of HPV-associated cancersProduction of infectious bovine papillomavirus from cloned viral DNA by using an organotypic raft/xenograft techniqueCurrent understanding of the role of the Brd4 protein in the papillomavirus lifecycle.Papillomavirus E2 proteins and the host BRD4 protein associate with transcriptionally active cellular chromatin.The papillomavirus E2 proteinsSp100 provides intrinsic immunity against human papillomavirus infection.Hitchhiking on host chromatin: how papillomaviruses persist.Mechanisms and strategies of papillomavirus replication.Phosphorylation regulates binding of the human papillomavirus type 8 E2 protein to host chromosomes.The human papillomavirus type 8 E2 tethering protein targets the ribosomal DNA loci of host mitotic chromosomes.Oncogenic human papillomaviruses.Casein Kinase II phosphorylation-induced conformational switch triggers degradation of the papillomavirus E2 protein.Interaction of the papillomavirus E2 protein with mitotic chromosomes.The transactivation and DNA binding domains of the BPV-1 E2 protein have different roles in cooperative origin binding with the E1 protein.Binding of bovine papillomavirus E1 to the origin is not sufficient for DNA replication.
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description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Alison A McBride
@nl
Alison A McBride
@sl
Alison A. McBride
@en
Alison A. McBride
@es
Alison McBride
@fr
type
label
Alison A McBride
@nl
Alison A McBride
@sl
Alison A. McBride
@en
Alison A. McBride
@es
Alison McBride
@fr
prefLabel
Alison A McBride
@nl
Alison A McBride
@sl
Alison A. McBride
@en
Alison A. McBride
@es
Alison McBride
@fr
P106
P1153
7102633364
P21
P31
P496
0000-0001-5607-5157