BTEB2, a Krüppel-like transcription factor, regulates expression of the SMemb/Nonmuscle myosin heavy chain B (SMemb/NMHC-B) gene.
about
Mammalian Krüppel-like factors in health and diseasesPositive and negative regulation of the cardiovascular transcription factor KLF5 by p300 and the oncogenic regulator SET through interaction and acetylation on the DNA-binding domainIntestinal-enriched Krüppel-like factor (Krüppel-like factor 5) is a positive regulator of cellular proliferationLipoprotein(a) promotes smooth muscle cell proliferation and dedifferentiation in atherosclerotic lesions of human apo(a) transgenic rabbitsPhosphorylation of Kruppel-like factor 5 (KLF5/IKLF) at the CBP interaction region enhances its transactivation functionThe double life of KLF5: Opposing roles in regulation of gene-expression, cellular function, and transformationRosiglitzone suppresses angiotensin II-induced production of KLF5 and cell proliferation in rat vascular smooth muscle cellsDifferential expression of microRNAs in different disease states.Immunohistochemical study of surgically excised choroidal neovascular membranesSignificance of the transcription factor KLF5 in cardiovascular remodelingHuman Kruppel-like factor 5 is a target of the E3 ubiquitin ligase WWP1 for proteolysis in epithelial cellsRole of complement 3a in the growth of mesangial cells from stroke-prone spontaneously hypertensive ratsRole of complement 3a in the synthetic phenotype and angiotensin II-production in vascular smooth muscle cells from spontaneously hypertensive ratsBlood lipid mediator sphingosine 1-phosphate potently stimulates platelet-derived growth factor-A and -B chain expression through S1P1-Gi-Ras-MAPK-dependent induction of Kruppel-like factor 5Intestinal tumor progression is associated with altered function of KLF5Positive- and negative-acting Kruppel-like transcription factors bind a transforming growth factor beta control element required for expression of the smooth muscle cell differentiation marker SM22alpha in vivo.Molecular mechanisms of decreased smooth muscle differentiation marker expression after vascular injury.CArG elements control smooth muscle subtype-specific expression of smooth muscle myosin in vivo5' CArG degeneracy in smooth muscle alpha-actin is required for injury-induced gene suppression in vivo.Multiple repressor pathways contribute to phenotypic switching of vascular smooth muscle cells.Protein inhibitor of activated STAT1 interacts with and up-regulates activities of the pro-proliferative transcription factor Krüppel-like factor 5.Perfusion of veins at arterial pressure increases the expression of KLF5 and cell cycle genes in smooth muscle cellsDirect targets of Klf5 transcription factor contribute to the maintenance of mouse embryonic stem cell undifferentiated state.The biology of the mammalian Krüppel-like family of transcription factors.Sp1 and krüppel-like factor family of transcription factors in cell growth regulation and cancer.Krüppel-like factors: three fingers in many pies.The deacetylase HDAC1 negatively regulates the cardiovascular transcription factor Krüppel-like factor 5 through direct interaction.Origin and differentiation of human and murine stroma.Lower urinary tract physiology and pharmacology.Microarray and biochemical analysis of lovastatin-induced apoptosis of squamous cell carcinomasRegulation of smooth muscle phenotype.Myosin II isoform switching mediates invasiveness after TGF-β-induced epithelial-mesenchymal transitionKruppel-like factor 5 (KLF5) is critical for conferring uterine receptivity to implantation.Analysis of uterine gene expression in interleukin-15 knockout mice reveals uterine natural killer cells do not play a major role in decidualization and associated angiogenesisRoles of Krüpel-like factor 4 in normal homeostasis, cancer and stem cells.The role of Krüppel-like factors in the reprogramming of somatic cells to induced pluripotent stem cells.Essential role of KLF5 transcription factor in cell proliferation and differentiation and its implications for human diseases.TT Mutant Homozygote of Kruppel-like Factor 5 Is a Key Factor for Increasing Basal Metabolic Rate and Resting Metabolic Rate in Korean Elementary School Children.Microarray analysis reveals novel gene expression changes associated with erectile dysfunction in diabetic rats.Role of Krüppel-like factor 4 in phenotypic switching and proliferation of vascular smooth muscle cells.
P2860
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P2860
BTEB2, a Krüppel-like transcription factor, regulates expression of the SMemb/Nonmuscle myosin heavy chain B (SMemb/NMHC-B) gene.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
BTEB2, a Krüppel-like transcri ...... y chain B (SMemb/NMHC-B) gene.
@ast
BTEB2, a Krüppel-like transcri ...... y chain B (SMemb/NMHC-B) gene.
@en
type
label
BTEB2, a Krüppel-like transcri ...... y chain B (SMemb/NMHC-B) gene.
@ast
BTEB2, a Krüppel-like transcri ...... y chain B (SMemb/NMHC-B) gene.
@en
prefLabel
BTEB2, a Krüppel-like transcri ...... y chain B (SMemb/NMHC-B) gene.
@ast
BTEB2, a Krüppel-like transcri ...... y chain B (SMemb/NMHC-B) gene.
@en
P2093
P356
P1433
P1476
BTEB2, a Krüppel-like transcri ...... y chain B (SMemb/NMHC-B) gene.
@en
P2093
Kawai-Kowase K
Kurabayashi M
Shimomura Y
Watanabe M
P304
P356
10.1161/01.RES.85.2.182
P577
1999-07-01T00:00:00Z