Diazoxide protects mitochondria from anoxic injury: implications for myopreservation.
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Mitochondrial transplantation for therapeutic useCardiomyocyte Remodeling in Atrial Fibrillation and Hibernating Myocardium: Shared Pathophysiologic Traits Identify Novel Treatment Strategies?Cardiac subsarcolemmal and interfibrillar mitochondria display distinct responsiveness to protection by diazoxideProtection of cardiac mitochondria by diazoxide and protein kinase C: implications for ischemic preconditioning.Peripheral channelopathies as targets for potassium channel openers.Potassium channel openers: therapeutic potential in cardiology and medicine.Intracoronary Delivery of Mitochondria to the Ischemic Heart for CardioprotectionPKCε promotes cardiac mitochondrial and metabolic adaptation to chronic hypobaric hypoxia by GSK3β inhibition.K(ATP) channel therapeutics at the bedside.Nitrite augments tolerance to ischemia/reperfusion injury via the modulation of mitochondrial electron transfer.Aging-induced alterations in gene transcripts and functional activity of mitochondrial oxidative phosphorylation complexes in the heartDiazoxide amelioration of myocardial injury and mitochondrial damage during cardiac surgery.Mechanism of Mitochondrial Connexin43's Protection of the Neurovascular Unit under Acute Cerebral Ischemia-Reperfusion Injury.Selective opening of mitochondrial ATP-sensitive potassium channels during surgically induced myocardial ischemia decreases necrosis and apoptosisOpening of the mitoKATP channel and decoupling of mitochondrial complex II and III contribute to the suppression of myocardial reperfusion hyperoxygenation.Multiplicity of effectors of the cardioprotective agent, diazoxide.Cardioprotection by metabolic shut-down and gradual wake-up.Inhibitors of succinate: quinone reductase/Complex II regulate production of mitochondrial reactive oxygen species and protect normal cells from ischemic damage but induce specific cancer cell death.Mitochondria from anoxia-tolerant animals reveal common strategies to survive without oxygen.Cellular and molecular events in ischemic preconditioning: potential therapeutic applications in cardioprotection.Beating oxygen: chronic anoxia exposure reduces mitochondrial F1FO-ATPase activity in turtle (Trachemys scripta) heart.Glucose-stimulated DNA synthesis through mammalian target of rapamycin (mTOR) is regulated by KATP channels: effects on cell cycle progression in rodent islets.Potassium channel openers protect cardiac mitochondria by attenuating oxidant stress at reoxygenation.Mechanisms by which opening the mitochondrial ATP- sensitive K(+) channel protects the ischemic heart.Opening of mitochondrial KATP channels enhances cardioprotection through the modulation of mitochondrial matrix volume, calcium accumulation, and respiration.K+-dependent regulation of matrix volume improves mitochondrial function under conditions mimicking ischemia-reperfusion.Transit and integration of extracellular mitochondria in human heart cells.Mitochondrial Transplantation in Myocardial Ischemia and Reperfusion Injury.Isoflurane preconditioning uncouples mitochondria and protects against hypoxia-reoxygenation.Myocardial preconditioning against ischemia-reperfusion injury is abolished in Zucker obese rats with insulin resistance.
P2860
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P2860
Diazoxide protects mitochondria from anoxic injury: implications for myopreservation.
description
2001 nî lūn-bûn
@nan
2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Diazoxide protects mitochondria from anoxic injury: implications for myopreservation.
@ast
Diazoxide protects mitochondria from anoxic injury: implications for myopreservation.
@en
type
label
Diazoxide protects mitochondria from anoxic injury: implications for myopreservation.
@ast
Diazoxide protects mitochondria from anoxic injury: implications for myopreservation.
@en
prefLabel
Diazoxide protects mitochondria from anoxic injury: implications for myopreservation.
@ast
Diazoxide protects mitochondria from anoxic injury: implications for myopreservation.
@en
P2093
P356
P1476
Diazoxide protects mitochondria from anoxic injury: implications for myopreservation.
@en
P2093
Holmuhamedov EL
Jahangir A
P304
P356
10.1067/MTC.2001.111421
P407
P577
2001-02-01T00:00:00Z