Signalling via the reperfusion injury signalling kinase (RISK) pathway links closure of the mitochondrial permeability transition pore to cardioprotection.
about
Role of the MPTP in conditioning the heart - translatability and mechanismIschaemic conditioning and targeting reperfusion injury: a 30 year voyage of discoveryAttenuation of doxorubicin-induced cardiotoxicity by mdivi-1: a mitochondrial division/mitophagy inhibitorMultiple abnormalities of myocardial insulin signaling in a porcine model of diet-induced obesity.Mitochondrial integrity: preservation through Akt/Pim-1 kinase signaling in the cardiomyocyteThe novel adipocytokine visfatin exerts direct cardioprotective effectsProtective effects of panax notoginseng saponins on cardiovascular diseases: a comprehensive overview of experimental studies.Induction of the matricellular protein CCN1 through RhoA and MRTF-A contributes to ischemic cardioprotection.The role of mitochondria in protection of the heart by preconditioning.Loss of PINK1 increases the heart's vulnerability to ischemia-reperfusion injury.Cardioprotective effects of mineralocorticoid receptor antagonists at reperfusion.Hydrogen sulfide provides cardioprotection against myocardial/ischemia reperfusion injury in the diabetic state through the activation of the RISK pathwayRole of Endothelial Cells in Myocardial Ischemia-Reperfusion Injury.Leptin, the obesity-associated hormone, exhibits direct cardioprotective effectsHyperglycemia raises the threshold of levosimendan- but not milrinone-induced postconditioning in rat heartsRole of Akt and Ca2+ on cell permeabilization via connexin43 hemichannels induced by metabolic inhibition.Vagal nerve stimulation started just prior to reperfusion limits infarct size and no-reflowA novel role for mitochondrial sphingosine-1-phosphate produced by sphingosine kinase-2 in PTP-mediated cell survival during cardioprotection.Preconditioning and postconditioning: the essential role of the mitochondrial permeability transition pore.Marked hyperglycemia attenuates anesthetic preconditioning in human-induced pluripotent stem cell-derived cardiomyocytes.Astragaloside IV inhibits oxidative stress-induced mitochondrial permeability transition pore opening by inactivating GSK-3β via nitric oxide in H9c2 cardiac cells.From Protecting the Heart to Improving Athletic Performance - the Benefits of Local and Remote Ischaemic Preconditioning.Mitochondria as a drug target in ischemic heart disease and cardiomyopathyOverexpression of uncoupling protein 2 inhibits the high glucose-induced apoptosis of human umbilical vein endothelial cells.Roles of endothelial nitric oxide synthase (eNOS) and mitochondrial permeability transition pore (MPTP) in epoxyeicosatrienoic acid (EET)-induced cardioprotection against infarction in intact rat hearts.Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase CMyocardial AKT: the omnipresent nexus.Preconditioning and postconditioning: new strategies for cardioprotection.Ischemic preconditioning: protection against myocardial necrosis and apoptosis.Nuclear and mitochondrial signalling Akts in cardiomyocytesAkt mediated mitochondrial protection in the heart: metabolic and survival pathways to the rescue.Hyperglycemia-induced inhibition of DJ-1 expression compromised the effectiveness of ischemic postconditioning cardioprotection in rats.Activation of ALDH2 with Low Concentration of Ethanol Attenuates Myocardial Ischemia/Reperfusion Injury in Diabetes Rat ModelIsoflurane decreases death of human embryonic stem cell-derived, transcriptional marker Nkx2.5(+) cardiac progenitor cells.Cardioprotective growth factors.JAK redux: a second look at the regulation and role of JAKs in the heart.Tauroursodeoxycholic acid inhibits endoplasmic reticulum stress, blocks mitochondrial permeability transition pore opening, and suppresses reperfusion injury through GSK-3ß in cardiac H9c2 cells.Inhibition of mitochondrial membrane permeability as a putative pharmacological target for cardioprotection.DJ-1 protects against cell death following acute cardiac ischemia-reperfusion injury.Insulin says NO to cardiovascular disease.
P2860
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P2860
Signalling via the reperfusion injury signalling kinase (RISK) pathway links closure of the mitochondrial permeability transition pore to cardioprotection.
description
2005 nî lūn-bûn
@nan
2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Signalling via the reperfusion ...... tion pore to cardioprotection.
@ast
Signalling via the reperfusion ...... tion pore to cardioprotection.
@en
type
label
Signalling via the reperfusion ...... tion pore to cardioprotection.
@ast
Signalling via the reperfusion ...... tion pore to cardioprotection.
@en
prefLabel
Signalling via the reperfusion ...... tion pore to cardioprotection.
@ast
Signalling via the reperfusion ...... tion pore to cardioprotection.
@en
P50
P1476
Signalling via the reperfusion ...... tion pore to cardioprotection.
@en
P304
P356
10.1016/J.BIOCEL.2005.09.017
P577
2005-10-21T00:00:00Z