Preconditioning and postconditioning: the essential role of the mitochondrial permeability transition pore.
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Beyond Preconditioning: Postconditioning as an Alternative Technique in the Prevention of Liver Ischemia-Reperfusion InjuryChemical Conditioning as an Approach to Ischemic Stroke Tolerance: Mitochondria as the TargetMitochondrial dynamics in cardiovascular health and diseaseRole of the MPTP in conditioning the heart - translatability and mechanismMitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioningMitochondrial fusion and fission proteins as novel therapeutic targets for treating cardiovascular diseaseProtein Oxidative Modifications: Beneficial Roles in Disease and HealthAttenuation of skeletal muscle and renal injury to the lower limb following ischemia-reperfusion using mPTP inhibitor NIM-811Pulmonary diesel particulate increases susceptibility to myocardial ischemia/reperfusion injury via activation of sensory TRPV1 and β1 adrenoreceptors.Endothelial NOS activity and myocardial oxygen metabolism define the salvageable ischemic time window for ischemic postconditioning.Cyclosporine A at reperfusion reduces infarct size in pigsMechanism for resveratrol-induced cardioprotection against reperfusion injury involves glycogen synthase kinase 3beta and mitochondrial permeability transition pore.The cardioprotective effect of necrostatin requires the cyclophilin-D component of the mitochondrial permeability transition poreType 2 diabetic obese db/db mice are refractory to myocardial ischaemic post-conditioning in vivo: potential role for Hsp20, F1-ATPase δ and Echs1Pharmacological postconditioning with tanshinone IIA attenuates myocardial ischemia-reperfusion injury in rats by activating the phosphatidylinositol 3-kinase pathwayMorphine induces preconditioning via activation of mitochondrial K(Ca) channelsThe novel adipocytokine visfatin exerts direct cardioprotective effectsAutophagy in health and disease. 5. Mitophagy as a way of lifeMitochondrial cyclophilin-D as a critical mediator of ischaemic preconditioning.NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in ratsRespiratory uncoupling by increased H(+) or K(+) flux is beneficial for heart mitochondrial turnover of reactive oxygen species but not for permeability transitionInhibitory effect of Curcuma purpurascens BI. rhizome on HT-29 colon cancer cells through mitochondrial-dependent apoptosis pathwayIschemia/reperfusion injury and cardioprotective mechanisms: Role of mitochondria and reactive oxygen speciesClinical effect of postconditioning in ST-elevation myocardial infarction patients treated with primary percutaneous coronary intervention: a meta-analysis of randomized controlled trials.Mechanisms underlying acute protection from cardiac ischemia-reperfusion injuryDistinct mPTP activation mechanisms in ischaemia-reperfusion: contributions of Ca2+, ROS, pH, and inorganic polyphosphate.Circadian dependence of infarct size and left ventricular function after ST elevation myocardial infarction.Protective effect of olmesartan against cardiac ischemia/reperfusion injury in spontaneously hypertensive ratsEndogenous cardioprotection by ischaemic postconditioning and remote conditioning.A novel role for mitochondrial sphingosine-1-phosphate produced by sphingosine kinase-2 in PTP-mediated cell survival during cardioprotection.Cyclosporin A and cardioprotection: from investigative tool to therapeutic agent.The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology.Cyclosporin variably and inconsistently reduces infarct size in experimental models of reperfused myocardial infarction: a systematic review and meta-analysis.Physiological and pathological roles of the mitochondrial permeability transition pore in the heart.Possible Role of Interaction between PPARα and Cyclophilin D in Cardioprotection of AMPK against In Vivo Ischemia-Reperfusion in RatsSoluble guanylate cyclase activation during ischemic injury in mice protects against postischemic inflammation at the mitochondrial level.Aging and cardioprotection.The paradigm of postconditioning to protect the heart.Mitochondrial therapeutics for cardioprotection.Cyclophilin D is required for mitochondrial removal by autophagy in cardiac cells
P2860
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P2860
Preconditioning and postconditioning: the essential role of the mitochondrial permeability transition pore.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Preconditioning and postcondit ...... permeability transition pore.
@ast
Preconditioning and postcondit ...... permeability transition pore.
@en
type
label
Preconditioning and postcondit ...... permeability transition pore.
@ast
Preconditioning and postcondit ...... permeability transition pore.
@en
prefLabel
Preconditioning and postcondit ...... permeability transition pore.
@ast
Preconditioning and postcondit ...... permeability transition pore.
@en
P2860
P50
P1476
Preconditioning and postcondit ...... permeability transition pore.
@en
P2093
Shiang Y Lim
P2860
P304
P356
10.1016/J.CARDIORES.2007.04.022
P577
2007-05-04T00:00:00Z