A molecular signature of gastric metaplasia arising in response to acute parietal cell loss
about
Targeted deletion of Kcne2 causes gastritis cystica profunda and gastric neoplasiaThe origin of pre-neoplastic metaplasia in the stomach: chief cells emerge from the MistMature chief cells are cryptic progenitors for metaplasia in the stomachThe gastric epithelial progenitor cell niche and differentiation of the zymogenic (chief) cell lineage.Role of Helicobacter pylori infection in gastric carcinogenesis: Current knowledge and future directionsHelicobacter pylori and microRNAs: Relation with innate immunity and progression of preneoplastic conditionsReserve stem cells: Differentiated cells reprogram to fuel repair, metaplasia, and neoplasia in the adult gastrointestinal tractSilencing Mist1 Gene Expression Is Essential for Recovery from Acute PancreatitisHip1r is expressed in gastric parietal cells and is required for tubulovesicle formation and cell survival in miceAutoimmune atrophic gastritis: current perspectives.Epithelial Regeneration After Gastric Ulceration Causes Prolonged Cell-Type AlterationsLoss of parietal cell expression of Sonic hedgehog induces hypergastrinemia and hyperproliferation of surface mucous cells.Gene expression profiling of metaplastic lineages identifies CDH17 as a prognostic marker in early stage gastric cancer.Transcriptome signatures in Helicobacter pylori-infected mucosa identifies acidic mammalian chitinase loss as a corpus atrophy markerAltered expression of a putative progenitor cell marker DCAMKL1 in the rat gastric mucosa in regeneration, metaplasia and dysplasia.Metaplasia in the Stomach Arises From Gastric Chief CellsThe transcription factor MIST1 is a novel human gastric chief cell marker whose expression is lost in metaplasia, dysplasia, and carcinoma.Metachronous gastric cancer after successful Helicobacter pylori eradication.Gastric epithelial stem cellsABCC11/MRP8 Expression in the Gastrointestinal Tract and a Novel Role for Pepsinogen Secretion.Heterogeneity in mouse spasmolytic polypeptide-expressing metaplasia lineages identifies markers of metaplastic progression.Establishment of novel in vitro mouse chief cell and SPEM cultures identifies MAL2 as a marker of metaplasia in the stomach.TFF2 mRNA transcript expression marks a gland progenitor cell of the gastric oxyntic mucosa.miR-30-HNF4γ and miR-194-NR2F2 regulatory networks contribute to the upregulation of metaplasia markers in the stomachFibroblast growth factor 10-fibroblast growth factor receptor 2b mediated signaling is not required for adult glandular stomach homeostasis.Expression and diagnostic value of HE4 in pancreatic adenocarcinoma.[Continual self-renewal of the gastric epithelium by cell differentiation: implications for carcinogenesis].Proteomic profiling of paraffin-embedded samples identifies metaplasia-specific and early-stage gastric cancer biomarkers.Identification of alanyl aminopeptidase (CD13) as a surface marker for isolation of mature gastric zymogenic chief cells.Human epididymis protein 4 is up-regulated in gastric and pancreatic adenocarcinomasLoss of anterior gradient 2 (Agr2) expression results in hyperplasia and defective lineage maturation in the murine stomachGastric tumor development in Smad3-deficient mice initiates from forestomach/glandular transition zone along the lesser curvature.The ubiquitin ligase Mindbomb 1 coordinates gastrointestinal secretory cell maturationAutoimmune gastritis mediated by CD4+ T cells promotes the development of gastric cancer.The hyaluronic acid receptor CD44 coordinates normal and metaplastic gastric epithelial progenitor cell proliferationLoss of HGF activator inhibits foveolar hyperplasia induced by oxyntic atrophy without altering gastrin levels.Hepatocyte growth factor regulates the development of highly pure cultured chief cells from rat stomach by stimulating chief cell proliferation in vitroInducible activation of Cre recombinase in adult mice causes gastric epithelial atrophy, metaplasia, and regenerative changes in the absence of "floxed" alleles.p40phox expression regulates neutrophil recruitment and function during the resolution phase of intestinal inflammation.How form follows functional genomics: gene expression profiling gastric epithelial cells with a particular discourse on the parietal cell
P2860
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P2860
A molecular signature of gastric metaplasia arising in response to acute parietal cell loss
description
2007 nî lūn-bûn
@nan
2007 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
A molecular signature of gastr ...... se to acute parietal cell loss
@ast
A molecular signature of gastr ...... se to acute parietal cell loss
@en
A molecular signature of gastr ...... e to acute parietal cell loss.
@nl
type
label
A molecular signature of gastr ...... se to acute parietal cell loss
@ast
A molecular signature of gastr ...... se to acute parietal cell loss
@en
A molecular signature of gastr ...... e to acute parietal cell loss.
@nl
prefLabel
A molecular signature of gastr ...... se to acute parietal cell loss
@ast
A molecular signature of gastr ...... se to acute parietal cell loss
@en
A molecular signature of gastr ...... e to acute parietal cell loss.
@nl
P2093
P2860
P1433
P1476
A molecular signature of gastr ...... se to acute parietal cell loss
@en
P2093
Bonnie J Lafleur
James R Goldenring
Janice A Williams
Jason C Mills
Koji Nozaki
Masako Ogawa
Stephen F Konieczny
P2860
P304
P356
10.1053/J.GASTRO.2007.11.058
P407
P577
2007-12-04T00:00:00Z