Mouse hepatitis coronavirus A59 nucleocapsid protein is a type I interferon antagonist.
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Regulation of Stress Responses and Translational Control by CoronavirusCoronavirus virulence genes with main focus on SARS-CoV envelope geneCoronavirus gene 7 counteracts host defenses and modulates virus virulenceCoronavirus Infection Modulates the Unfolded Protein Response and Mediates Sustained Translational RepressionMurine Coronavirus Mouse Hepatitis Virus Is Recognized by MDA5 and Induces Type I Interferon in Brain Macrophages/MicrogliaMouse hepatitis coronavirus nucleocapsid phosphorylation.Coronavirus non-structural protein 1 is a major pathogenicity factor: implications for the rational design of coronavirus vaccines.Suppression of the interferon and NF-κB responses by severe fever with thrombocytopenia syndrome virus.Detection and evaluation of immunofunction of patients with severe fever with thrombocytopenia syndrome.Type I interferon receptor-independent and -dependent host transcriptional responses to mouse hepatitis coronavirus infection in vivoRNase L mediated protection from virus induced demyelination.Viral and Cellular mRNA Translation in Coronavirus-Infected Cells.Coronaviruses post-SARS: update on replication and pathogenesis.Coronavirus infection, ER stress, apoptosis and innate immunityDeubiquitinating and interferon antagonism activities of coronavirus papain-like proteases.Murine coronavirus induces type I interferon in oligodendrocytes through recognition by RIG-I and MDA5.Mechanisms of severe acute respiratory syndrome pathogenesis and innate immunomodulation.Pathogenesis of murine coronavirus in the central nervous system.Accessory protein 5a is a major antagonist of the antiviral action of interferon against murine coronavirus.Porcine epidemic diarrhea virus nucleocapsid protein antagonizes beta interferon production by sequestering the interaction between IRF3 and TBK1.Genetic determinants of mouse hepatitis virus strain 1 pneumovirulence.Autocrine interferon priming in macrophages but not dendritic cells results in enhanced cytokine and chemokine production after coronavirus infectionCell-type-specific type I interferon antagonism influences organ tropism of murine coronavirusA Kinome-Wide Small Interfering RNA Screen Identifies Proviral and Antiviral Host Factors in Severe Acute Respiratory Syndrome Coronavirus Replication, Including Double-Stranded RNA-Activated Protein Kinase and Early Secretory Pathway Proteins.The Nucleocapsid Protein of Coronaviruses Acts as a Viral Suppressor of RNA Silencing in Mammalian CellsInhibition of the alpha/beta interferon response by mouse hepatitis virus at multiple levels.Identification of mouse hepatitis coronavirus A59 nucleocapsid protein phosphorylation sitesProtective role of Toll-like Receptor 3-induced type I interferon in murine coronavirus infection of macrophages.Recent progress in studies of arterivirus- and coronavirus-host interactionsMiddle East Respiratory Coronavirus Accessory Protein 4a Inhibits PKR-Mediated Antiviral Stress ResponsesType I interferons are essential in controlling neurotropic coronavirus infection irrespective of functional CD8 T cells.SARS coronavirus and innate immunity.Severe acute respiratory syndrome coronavirus protein 6 accelerates murine hepatitis virus infections by more than one mechanism.The structural and accessory proteins M, ORF 4a, ORF 4b, and ORF 5 of Middle East respiratory syndrome coronavirus (MERS-CoV) are potent interferon antagonistsSevere acute respiratory syndrome coronavirus protein 6 is required for optimal replication.Alphacoronavirus protein 7 modulates host innate immune response.Organ-specific attenuation of murine hepatitis virus strain A59 by replacement of catalytic residues in the putative viral cyclic phosphodiesterase ns2.Middle East respiratory syndrome coronavirus accessory protein 4a is a type I interferon antagonist.Mouse hepatitis virus infection of the CNS: a model for defense, disease, and repair.Reverse genetic characterization of the natural genomic deletion in SARS-Coronavirus strain Frankfurt-1 open reading frame 7b reveals an attenuating function of the 7b protein in-vitro and in-vivo.
P2860
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P2860
Mouse hepatitis coronavirus A59 nucleocapsid protein is a type I interferon antagonist.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Mouse hepatitis coronavirus A59 nucleocapsid protein is a type I interferon antagonist.
@en
type
label
Mouse hepatitis coronavirus A59 nucleocapsid protein is a type I interferon antagonist.
@en
prefLabel
Mouse hepatitis coronavirus A59 nucleocapsid protein is a type I interferon antagonist.
@en
P2093
P2860
P356
P1433
P1476
Mouse hepatitis coronavirus A59 nucleocapsid protein is a type I interferon antagonist.
@en
P2093
Bertram L Jacobs
Brenda G Hogue
Jeffrey O Langland
Kevin Hauns
P2860
P304
P356
10.1128/JVI.01634-06
P407
P577
2006-12-20T00:00:00Z