Comprehensive follow-up of the first genome-wide association study of multiple sclerosis identifies KIF21B and TMEM39A as susceptibility loci.
about
The genetic association of variants in CD6, TNFRSF1A and IRF8 to multiple sclerosis: a multicenter case-control studyRevealing the genetic basis of multiple sclerosis: are we there yet?Chronic cerebro-spinal venous insufficiency (CCSVI) and multiple sclerosisDiverse convergent evidence in the genetic analysis of complex disease: coordinating omic, informatic, and experimental evidence to better identify and validate risk factorsTRIM3 regulates the motility of the kinesin motor protein KIF21BPolymorphisms in the receptor tyrosine kinase MERTK gene are associated with multiple sclerosis susceptibilityA polymorphism in the HLA-DPB1 gene is associated with susceptibility to multiple sclerosisGenome-wide meta-analysis identifies novel multiple sclerosis susceptibility loci.Genome-wide association study of severity in multiple sclerosis.Identification of Orch3, a locus controlling dominant resistance to autoimmune orchitis, as kinesin family member 1CAn inflammatory bowel disease-risk variant in INAVA decreases pattern recognition receptor-induced outcomesThe effect of single nucleotide polymorphisms from genome wide association studies in multiple sclerosis on gene expression.Association of novel polymorphisms in TMEM39A gene with systemic lupus erythematosus in a Chinese Han population.Synthesis-View: visualization and interpretation of SNP association results for multi-cohort, multi-phenotype data and meta-analysis.Genetic variation in the IL7RA/IL7 pathway increases multiple sclerosis susceptibility.Preliminary Study on the Role of TMEM39A Gene in Multiple Sclerosis.Evidence for CRHR1 in multiple sclerosis using supervised machine learning and meta-analysis in 12,566 individuals.Identity-by-descent mapping to detect rare variants conferring susceptibility to multiple sclerosisA noise-reduction GWAS analysis implicates altered regulation of neurite outgrowth and guidance in autism.Association of interleukin 7 receptor gene polymorphism rs6897932 with multiple sclerosis patients in Khuzestan.Multiple sclerosis susceptibility genes: associations with relapse severity and recovery.Association of multiple sclerosis susceptibility variants and early attack location in the CNSInterrogating the complex role of chromosome 16p13.13 in multiple sclerosis susceptibility: independent genetic signals in the CIITA-CLEC16A-SOCS1 gene complexAssociation analyses confirm five susceptibility loci for systemic lupus erythematosus in the Han Chinese population.Autoimmune risk loci of IL12RB2, IKZF1, XKR6, TMEM39A and CSK in Chinese patients with systemic lupus erythematosus.Identification of IRF8, TMEM39A, and IKZF3-ZPBP2 as susceptibility loci for systemic lupus erythematosus in a large-scale multiracial replication study.Serum Leukocyte Immunoglobulin-Like Receptor A3 (LILRA3) Is Increased in Patients with Multiple Sclerosis and Is a Strong Independent Indicator of Disease Severity; 6.7kbp LILRA3 Gene Deletion Is Not Associated with Diseases SusceptibilityXVI European Charcot Foundation lecture: nutrition and environment: can MS be prevented?The 2D:4D ratio, a proxy for prenatal androgen levels, differs in men with and without MS.Progress in multiple sclerosis genetics.Aggregation of multiple sclerosis genetic risk variants in multiple and single case families.Variation within DNA repair pathway genes and risk of multiple sclerosis.De Novo Mutations of RERE Cause a Genetic Syndrome with Features that Overlap Those Associated with Proximal 1p36 Deletions.GWAS analysis implicates NF-κB-mediated induction of inflammatory T cells in multiple sclerosis.Genome-wide association study for acute otitis media in children identifies FNDC1 as disease contributing gene.Recognition of Transmembrane Protein 39A as a Tumor-Specific Marker in Brain Tumor.The Kinesin KIF21B Regulates Microtubule Dynamics and Is Essential for Neuronal Morphology, Synapse Function, and Learning and MemoryMultiple sclerosis etiology: beyond genes and environment.Revisiting the T-cell receptor alpha/delta locus and possible associations with multiple sclerosis.Correcting away the hidden heritability.
P2860
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P2860
Comprehensive follow-up of the first genome-wide association study of multiple sclerosis identifies KIF21B and TMEM39A as susceptibility loci.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Comprehensive follow-up of the ...... MEM39A as susceptibility loci.
@ast
Comprehensive follow-up of the ...... MEM39A as susceptibility loci.
@en
type
label
Comprehensive follow-up of the ...... MEM39A as susceptibility loci.
@ast
Comprehensive follow-up of the ...... MEM39A as susceptibility loci.
@en
prefLabel
Comprehensive follow-up of the ...... MEM39A as susceptibility loci.
@ast
Comprehensive follow-up of the ...... MEM39A as susceptibility loci.
@en
P2860
P921
P356
P1476
Comprehensive follow-up of the ...... MEM39A as susceptibility loci.
@en
P2093
International Multiple Sclerosis Genetics Consortium (IMSGC)
P2860
P304
P356
10.1093/HMG/DDP542
P577
2009-12-09T00:00:00Z