Titin-mediated control of cardiac myofibrillar function.
about
Chronic Rho-kinase inhibition improves left ventricular contractile dysfunction in early type-1 diabetes by increasing myosin cross-bridge extension.Molecule specific effects of PKA-mediated phosphorylation on rat isolated heart and cardiac myofibrillar functionIncreased Titin Compliance Reduced Length-Dependent Contraction and Slowed Cross-Bridge Kinetics in Skinned Myocardial Strips from Rbm (20ΔRRM) Mice.β-Arrestin mediates the Frank-Starling mechanism of cardiac contractilityCalcium sensitivity of residual force enhancement in rabbit skinned fibers.Myosin MgADP release rate decreases at longer sarcomere length to prolong myosin attachment time in skinned rat myocardium.
P2860
Titin-mediated control of cardiac myofibrillar function.
description
2013 nî lūn-bûn
@nan
2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Titin-mediated control of cardiac myofibrillar function.
@ast
Titin-mediated control of cardiac myofibrillar function.
@en
type
label
Titin-mediated control of cardiac myofibrillar function.
@ast
Titin-mediated control of cardiac myofibrillar function.
@en
prefLabel
Titin-mediated control of cardiac myofibrillar function.
@ast
Titin-mediated control of cardiac myofibrillar function.
@en
P2860
P1476
Titin-mediated control of cardiac myofibrillar function
@en
P2093
Laurin M Hanft
Marion L Greaser
P2860
P356
10.1016/J.ABB.2013.11.005
P407
P478
P577
2013-11-20T00:00:00Z