Bruton's tyrosine kinase regulates immunoglobulin promoter activation in association with the transcription factor Bright.
about
Attenuation of PTEN increases p21 stability and cytosolic localization in kidney cancer cells: a potential mechanism of apoptosis resistance.Signalling of the BCR is regulated by a lipid rafts-localised transcription factor, Bright.Characterization of a new ARID family transcription factor (Brightlike/ARID3C) that co-activates Bright/ARID3A-mediated immunoglobulin gene transcriptionInduction of immunoglobulin heavy-chain transcription through the transcription factor Bright requires TFII-IBTK inhibitors in chronic lymphocytic leukemia: a glimpse to the future.The ARID family transcription factor bright is required for both hematopoietic stem cell and B lineage development.The herpes simplex virus immediate-early ubiquitin ligase ICP0 induces degradation of the ICP0 repressor protein E2FBP1.Lin28b promotes fetal B lymphopoiesis through the transcription factor Arid3aThe transcription factor Bright plays a role in marginal zone B lymphocyte development and autoantibody production.Bruton's tyrosine kinase revealed as a negative regulator of Wnt-beta-catenin signaling.Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.Clinical potential of targeting Bruton's tyrosine kinase.The Bright Side of Hematopoiesis: Regulatory Roles of ARID3a/Bright in Human and Mouse Hematopoiesis.Bruton tyrosine kinase (BTK) and its role in B-cell malignancy.The Transcription Factor ARID3a Is Important for In Vitro Differentiation of Human Hematopoietic Progenitors.Perspectives on fetal derived CD5+ B1 B cellsEnhanced Bruton's Tyrosine Kinase Activity in Peripheral Blood B Lymphocytes From Patients With Autoimmune Disease.Anti-nuclear antibody production and autoimmunity in transgenic mice that overexpress the transcription factor Bright.Construction of human activity-based phosphorylation networks.Differential expression of the transcription factor ARID3a in lupus patient hematopoietic progenitor cells.Loss of Bright/ARID3a function promotes developmental plasticity.A regulated nucleocytoplasmic shuttle contributes to Bright's function as a transcriptional activator of immunoglobulin genes.Bright/ARID3A contributes to chromatin accessibility of the immunoglobulin heavy chain enhancer
P2860
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P2860
Bruton's tyrosine kinase regulates immunoglobulin promoter activation in association with the transcription factor Bright.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մարտին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Bruton's tyrosine kinase regul ...... e transcription factor Bright.
@ast
Bruton's tyrosine kinase regul ...... e transcription factor Bright.
@en
type
label
Bruton's tyrosine kinase regul ...... e transcription factor Bright.
@ast
Bruton's tyrosine kinase regul ...... e transcription factor Bright.
@en
prefLabel
Bruton's tyrosine kinase regul ...... e transcription factor Bright.
@ast
Bruton's tyrosine kinase regul ...... e transcription factor Bright.
@en
P2093
P2860
P1476
Bruton's tyrosine kinase regul ...... e transcription factor Bright.
@en
P2093
Carol F Webb
David J Rawlings
Jamee C Nixon
Jaya Rajaiya
Melissa Hatfield
P2860
P304
P356
10.1128/MCB.25.6.2073-2084.2005
P407
P577
2005-03-01T00:00:00Z