Hepatic FGF21 expression is induced at birth via PPARalpha in response to milk intake and contributes to thermogenic activation of neonatal brown fat. - Wikidata - awgldk - TriplyDB

Hepatic FGF21 expression is induced at birth via PPARalpha in response to milk intake and contributes to thermogenic activation of neonatal brown fat.

Hepatic FGF21 expression is induced at birth via PPARalpha in response to milk intake and contributes to thermogenic activation of neonatal brown fat.

http://www.wikidata.org/entity/Q33762194

about

Endocrine fibroblast growth factors 15/19 and 21: from feast to famine Fatty acid metabolism and the basis of brown adipose tissue function Non-sympathetic control of brown adipose tissue Fibroblast Growth Factor Signaling in Metabolic Regulation Metabolic Effects of FGF-21: Thermoregulation and Beyond FGF21 and Cardiac Physiopathology FGF21, energy expenditure and weight loss - How much brown fat do you need?Developmental influences on circuits programming susceptibility to obesity Mitochondrial fatty acid oxidation in obesity Inventing new medicines: The FGF21 story The brain and brown fat Stressed Liver and Muscle Call on Adipocytes with FGF21 Differential Role of Adipose Tissues in Obesity and Related Metabolic and Vascular Complications Brown adipose tissue activity as a target for the treatment of obesity/insulin resistance Obligate role for ketone body oxidation in neonatal metabolic homeostasis Pre and post-natal risk and determination of factors for child obesity FGF21 contributes to neuroendocrine control of female reproduction.Brown adipose tissue: Recent insights into development, metabolic function and therapeutic potential Traveling from the hypothalamus to the adipose tissue: The thermogenic pathway Reduced adiposity attenuates FGF21 mediated metabolic improvements in the Siberian hamster.A high-fat, ketogenic diet causes hepatic insulin resistance in mice, despite increasing energy expenditure and preventing weight gain Isoenergetic feeding of low carbohydrate-high fat diets does not increase brown adipose tissue thermogenic capacity in rats FGF21 acts centrally to induce sympathetic nerve activity, energy expenditure, and weight loss.Adaptive thermogenesis in adipocytes: is beige the new brown?Cellular mechanisms by which FGF21 improves insulin sensitivity in male mice Brown adipose tissue and thermogenesis.Nursing for 48 hours from birth supports porcine uterine gland development and endometrial cell compartment-specific gene expression.Disruption of thyroid hormone activation in type 2 deiodinase knockout mice causes obesity with glucose intolerance and liver steatosis only at thermoneutrality Surge in expression of carboxylesterase 1 during the post-neonatal stage enables a rapid gain of the capacity to activate the anti-influenza prodrug oseltamivir.Tissue-specific actions of the metabolic hormones FGF15/19 and FGF21.Thermogenic activation induces FGF21 expression and release in brown adipose tissue.The miR-379/miR-410 cluster at the imprinted Dlk1-Dio3 domain controls neonatal metabolic adaptation Nutritionally-induced catch-up growth Molecular characterization and mapping of Fgf21 gene in a foodfish species asian seabass.Placental fibroblast growth factor 21 is not altered in late-onset preeclampsia.Fibroblast growth factor 21 induces glucose transporter-1 expression through activation of the serum response factor/Ets-like protein-1 in adipocytes.Adipose tissue plasticity from WAT to BAT and in between.Parasitic helminths and their beneficial impact on type 1 and type 2 diabetes.FGF21 regulates PGC-1α and browning of white adipose tissues in adaptive thermogenesis.Central Fibroblast Growth Factor 21 Browns White Fat via Sympathetic Action in Male Mice.

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P2860

Hepatic FGF21 expression is induced at birth via PPARalpha in response to milk intake and contributes to thermogenic activation of neonatal brown fat.

http://www.wikidata.org/entity/Q33762194

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Hepatic FGF21 expression is in ...... ivation of neonatal brown fat.

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Hepatic FGF21 expression is in ...... ivation of neonatal brown fat.

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Hepatic FGF21 expression is in ...... ivation of neonatal brown fat.

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Hepatic FGF21 expression is in ...... ivation of neonatal brown fat.

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Hepatic FGF21 expression is in ...... ivation of neonatal brown fat.

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Hepatic FGF21 expression is in ...... ivation of neonatal brown fat.

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P356

J.CMET.2010.02.001

P1433

Cell Metabolism

P1476

Hepatic FGF21 expression is in ...... tivation of neonatal brown fat

@en

P2093

Elayne Hondares

http://www.w3.org/2001/XMLSchema#string

Frank J Gonzalez

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Meritxell Rosell

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Roser Iglesias

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P2860

FGF-21 as a novel metabolic regulator.

FGF21 induces PGC-1alpha and regulates carbohydrate and fatty acid metabolism during the adaptive starvation response

Metabolic imprinting: critical impact of the perinatal environment on the regulation of energy homeostasis

Activators of peroxisome proliferator-activated receptor-alpha induce the expression of the uncoupling protein-3 gene in skeletal muscle: a potential mechanism for the lipid intake-dependent activation of uncoupling protein-3 gene expression at birth

Hepatic fibroblast growth factor 21 is regulated by PPARalpha and is a key mediator of hepatic lipid metabolism in ketotic states

Endocrine regulation of the fasting response by PPARalpha-mediated induction of fibroblast growth factor 21

Fibroblast growth factor 21 corrects obesity in mice.

Free fatty acids link metabolism and regulation of the insulin-sensitizing fibroblast growth factor-21.

Fibroblast growth factor 21 controls glycemia via regulation of hepatic glucose flux and insulin sensitivity

Circulating fibroblast growth factor-21 is elevated in impaired glucose tolerance and type 2 diabetes and correlates with muscle and hepatic insulin resistance.

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206-212

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scholarly article

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10.1016/J.CMET.2010.02.001

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3

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11

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Francesc Villarroya

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2010-03-01T00:00:00Z

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41668876

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20197053

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2847690

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