Enhancer requirement for murine cytomegalovirus growth and genetic complementation by the human cytomegalovirus enhancer.
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Elimination of ie1 significantly attenuates murine cytomegalovirus virulence but does not alter replicative capacity in cell cultureThe human cytomegalovirus major immediate-early distal enhancer region is required for efficient viral replication and immediate-early gene expression.Identification of a boundary domain adjacent to the potent human cytomegalovirus enhancer that represses transcription of the divergent UL127 promoter.In vivo replication of recombinant murine cytomegalovirus driven by the paralogous major immediate-early promoter-enhancer of human cytomegalovirusSystematic excision of vector sequences from the BAC-cloned herpesvirus genome during virus reconstitution.Patchwork pattern of transcriptional reactivation in the lungs indicates sequential checkpoints in the transition from murine cytomegalovirus latency to recurrenceA strong negative transcriptional regulatory region between the human cytomegalovirus UL127 gene and the major immediate-early enhancer.The major immediate-early gene ie3 of mouse cytomegalovirus is essential for viral growth.Dominance of virus over host factors in cross-species activation of human cytomegalovirus early gene expression.Reactivation of the human cytomegalovirus major immediate-early regulatory region and viral replication in embryonal NTera2 cells: role of trichostatin A, retinoic acid, and deletion of the 21-base-pair repeats and modulator.Rat cytomegalovirus major immediate-early enhancer switching results in altered growth characteristics.Random, asynchronous, and asymmetric transcriptional activity of enhancer-flanking major immediate-early genes ie1/3 and ie2 during murine cytomegalovirus latency in the lungs.Enhancerless cytomegalovirus is capable of establishing a low-level maintenance infection in severely immunodeficient host tissues but fails in exponential growth.Requirement of multiple cis-acting elements in the human cytomegalovirus major immediate-early distal enhancer for viral gene expression and replication.The human cytomegalovirus major immediate-early enhancer determines the efficiency of immediate-early gene transcription and viral replication in permissive cells at low multiplicity of infection.An essential role of the enhancer for murine cytomegalovirus in vivo growth and pathogenesis.Cloning of the full-length rhesus cytomegalovirus genome as an infectious and self-excisable bacterial artificial chromosome for analysis of viral pathogenesis.Viral enhancer mimicry of host innate-immune promoters.A temporal gate for viral enhancers to co-opt Toll-like-receptor transcriptional activation pathways upon acute infectionReversible inhibition of murine cytomegalovirus replication by gamma interferon (IFN-γ) in primary macrophages involves a primed type I IFN-signaling subnetwork for full establishment of an immediate-early antiviral state.Immediate-Early (IE) gene regulation of cytomegalovirus: IE1- and pp71-mediated viral strategies against cellular defensesIn vivo competence of murine cytomegalovirus under the control of the human cytomegalovirus major immediate-early enhancer in the establishment of latency and reactivation.Neutrality of the canonical NF-kappaB-dependent pathway for human and murine cytomegalovirus transcription and replication in vitro.Role of the proximal enhancer of the major immediate-early promoter in human cytomegalovirus replication.Mouse cytomegalovirus early M112/113 proteins control the repressive effect of IE3 on the major immediate-early promoter.Regulation of the transcription and replication cycle of human cytomegalovirus is insensitive to genetic elimination of the cognate NF-kappaB binding sites in the enhancer.Generation of mutant murine cytomegalovirus strains from overlapping cosmid and plasmid clones.Cloning of the human cytomegalovirus (HCMV) genome as an infectious bacterial artificial chromosome in Escherichia coli: a new approach for construction of HCMV mutants.The activator protein 1 binding motifs within the human cytomegalovirus major immediate-early enhancer are functionally redundant and act in a cooperative manner with the NF-{kappa}B sites during acute infectionBile Acids Act as Soluble Host Restriction Factors Limiting Cytomegalovirus Replication in Hepatocytes.Murine cytomegalovirus major immediate-early enhancer region operating as a genetic switch in bidirectional gene pair transcription.Temporal profiling of the coding and noncoding murine cytomegalovirus transcriptomes.
P2860
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P2860
Enhancer requirement for murine cytomegalovirus growth and genetic complementation by the human cytomegalovirus enhancer.
description
1998 nî lūn-bûn
@nan
1998 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Enhancer requirement for murin ...... uman cytomegalovirus enhancer.
@ast
Enhancer requirement for murin ...... uman cytomegalovirus enhancer.
@en
type
label
Enhancer requirement for murin ...... uman cytomegalovirus enhancer.
@ast
Enhancer requirement for murin ...... uman cytomegalovirus enhancer.
@en
prefLabel
Enhancer requirement for murin ...... uman cytomegalovirus enhancer.
@ast
Enhancer requirement for murin ...... uman cytomegalovirus enhancer.
@en
P2093
P2860
P1433
P1476
Enhancer requirement for murin ...... uman cytomegalovirus enhancer.
@en
P2093
U H Koszinowski
P2860
P304
P577
1998-11-01T00:00:00Z