Targeted recombination within the spike gene of murine coronavirus mouse hepatitis virus-A59: Q159 is a determinant of hepatotropism.
about
Molecular mechanisms of severe acute respiratory syndrome (SARS)Mechanisms of coronavirus cell entry mediated by the viral spike proteinVirus-like particles of SARS-like coronavirus formed by membrane proteins from different origins demonstrate stimulating activity in human dendritic cells.Demyelination determinants map to the spike glycoprotein gene of coronavirus mouse hepatitis virusPathogenesis of chimeric MHV4/MHV-A59 recombinant viruses: the murine coronavirus spike protein is a major determinant of neurovirulence.Retargeting of coronavirus by substitution of the spike glycoprotein ectodomain: crossing the host cell species barrierDevelopment of a transgenic mouse model susceptible to human coronavirus 229E.Murine coronavirus evolution in vivo: functional compensation of a detrimental amino acid substitution in the receptor binding domain of the spike glycoprotein.Murine coronavirus spike protein determines the ability of the virus to replicate in the liver and cause hepatitis.Variations in disparate regions of the murine coronavirus spike protein impact the initiation of membrane fusion.Contributions of the viral genetic background and a single amino acid substitution in an immunodominant CD8+ T-cell epitope to murine coronavirus neurovirulence.A mechanism of virus-induced demyelination.Pathogenesis of murine coronavirus in the central nervous system.Coronavirus pathogenesis and the emerging pathogen severe acute respiratory syndrome coronavirus.Switching species tropism: an effective way to manipulate the feline coronavirus genome.Murine coronavirus-induced hepatitis: JHM genetic background eliminates A59 spike-determined hepatotropismAnimal origins of the severe acute respiratory syndrome coronavirus: insight from ACE2-S-protein interactions.Identification and characterization of a novel alpaca respiratory coronavirus most closely related to the human coronavirus 229E.A review of genetic methods and models for analysis of coronavirus-induced severe pneumonitis.The E protein is a multifunctional membrane protein of SARS-CoV.The N-terminal domain of the murine coronavirus spike glycoprotein determines the CEACAM1 receptor specificity of the virus strain.Targeted recombination demonstrates that the spike gene of transmissible gastroenteritis coronavirus is a determinant of its enteric tropism and virulence.Conformational changes in the spike glycoprotein of murine coronavirus are induced at 37 degrees C either by soluble murine CEACAM1 receptors or by pH 8.Retroviral vectors pseudotyped with severe acute respiratory syndrome coronavirus S protein.TNF receptor 1 mediates dendritic cell maturation and CD8 T cell response through two distinct mechanisms.
P2860
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P2860
Targeted recombination within the spike gene of murine coronavirus mouse hepatitis virus-A59: Q159 is a determinant of hepatotropism.
description
1998 nî lūn-bûn
@nan
1998 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Targeted recombination within ...... determinant of hepatotropism.
@ast
Targeted recombination within ...... determinant of hepatotropism.
@en
type
label
Targeted recombination within ...... determinant of hepatotropism.
@ast
Targeted recombination within ...... determinant of hepatotropism.
@en
prefLabel
Targeted recombination within ...... determinant of hepatotropism.
@ast
Targeted recombination within ...... determinant of hepatotropism.
@en
P2093
P2860
P1433
P1476
Targeted recombination within ...... a determinant of hepatotropism
@en
P2093
I Leparc-Goffart
J Phillips
S T Hingley
P2860
P304
P577
1998-12-01T00:00:00Z