The IL-1 receptor and Rho directly associate to drive cell activation in inflammation.
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Transactivation by the p65 subunit of NF-kappaB in response to interleukin-1 (IL-1) involves MyD88, IL-1 receptor-associated kinase 1, TRAF-6, and Rac1Pleiotropic effects of statinsTemporal and spatial patterns of ovarian gene transcription following an ovulatory dose of gonadotropin in the ratA gene expression signature of confinement in peripheral blood of red wolves (Canis rufus).Contribution of small GTPase Rho and its target protein rock in a murine model of lung fibrosis.Use of serial analysis of gene expression to generate kidney expression libraries.Local thermal injury elicits immediate dynamic behavioural responses by corneal Langerhans cells.Changes in surface topologies of chondrocytes subjected to mechanical forces: an AFM analysis.A novel polymorphism in the toll-like receptor 2 gene and its potential association with staphylococcal infection.Pharmacology of 3-hydroxy-3-methylglutaryl-coenzyme A reductase inhibitors (statins), including rosuvastatin and pitavastatin.Manipulating actin dynamics affects human in vitro decidualization.Dihydroartemisinin supresses inflammation and fibrosis in bleomycine-induced pulmonary fibrosis in rats.Chronic inflammation in cancer development.Fasudil, a Rho-kinase inhibitor, attenuates bleomycin-induced pulmonary fibrosis in miceA comprehensive map of the toll-like receptor signaling network.Regulation of actin dynamics and protein trafficking during spermatogenesis--insights into a complex process.Evaluation of atorvastatin and simvastatin for treatment of multiple sclerosis.Astrocytes from acyclic female rats exhibit lowered capacity for neuronal differentiation.Inflammatory cytokines in vascular dysfunction and vascular disease.Interleukin-1beta causes acute lung injury via alphavbeta5 and alphavbeta6 integrin-dependent mechanisms.The biology of interleukin-1: emerging concepts in the regulation of the actin cytoskeleton and cell junction dynamics.Quantitative phosphoproteomic analysis of IL-33-mediated signalingGene therapy: a potential approach for cancer pain.Interleukin-1 stimulates glutamate uptake in glial cells by accelerating membrane trafficking of Na+/K+-ATPase via actin depolymerizationPneumococci induced TLR- and Rac1-dependent NF-kappaB-recruitment to the IL-8 promoter in lung epithelial cells.Perturbation of actin dynamics induces NF-kappaB activation in myelomonocytic cells through an NADPH oxidase-dependent pathway.The gene for a new brain specific RhoA exchange factor maps to the highly unstable chromosomal region 1p36.2-1p36.3.Rac1 regulates interleukin 1-induced nuclear factor kappaB activation in an inhibitory protein kappaBalpha-independent manner by enhancing the ability of the p65 subunit to transactivate gene expression.Oxygen radicals elicit paralysis and collapse of spinal cord neuron growth cones upon exposure to proinflammatory cytokines.Botulinum toxin type-A therapy in cluster headache: a novel molecular mechanism.Signal transduction pathways of IL-1beta-mediated iNOS in pulmonary vascular smooth muscle cells.A WT1 co-regulator controls podocyte phenotype by shuttling between adhesion structures and nucleus.Rho inhibition decreases TNF-induced endothelial MAPK activation and monolayer permeability.Statins in multiple sclerosis: a new therapeutic option?Natural isoprenoids are able to reduce inflammation in a mouse model of mevalonate kinase deficiency.Novel use of botulinum toxin to ameliorate arthrofibrosis: an experimental study in rabbits.Disruption of the actin cytoskeleton regulates cytokine-induced iNOS expression.Phenotypic modulation of cultured bladder smooth muscle cells and the expression of inducible nitric oxide synthase.Genomic regions underlying uniformity of yearling weight in Nellore cattle evaluated under different response variables
P2860
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P2860
The IL-1 receptor and Rho directly associate to drive cell activation in inflammation.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
name
The IL-1 receptor and Rho directly associate to drive cell activation in inflammation.
@ast
The IL-1 receptor and Rho directly associate to drive cell activation in inflammation.
@en
type
label
The IL-1 receptor and Rho directly associate to drive cell activation in inflammation.
@ast
The IL-1 receptor and Rho directly associate to drive cell activation in inflammation.
@en
prefLabel
The IL-1 receptor and Rho directly associate to drive cell activation in inflammation.
@ast
The IL-1 receptor and Rho directly associate to drive cell activation in inflammation.
@en
P2093
P2860
P356
P1476
The IL-1 receptor and Rho directly associate to drive cell activation in inflammation.
@en
P2093
A Shirvaikar
J R Schelling
M Konieczkowski
P2860
P304
P356
10.1172/JCI5754
P407
P577
1999-06-01T00:00:00Z