Control of Epstein-Barr virus reactivation by activated CD40 and viral latent membrane protein 1. - Wikidata - awgldk - TriplyDB

Control of Epstein-Barr virus reactivation by activated CD40 and viral latent membrane protein 1.

Control of Epstein-Barr virus reactivation by activated CD40 and viral latent membrane protein 1.

http://www.wikidata.org/entity/Q33898010

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Signal Transduction and Transcription Factor Modification during Reactivation of Epstein-Barr Virus from Latency Epstein-Barr virus LMP2A signaling in statu nascendi mimics a B cell antigen receptor-like activation signal IL-21 imposes a type II EBV gene expression on type III and type I B cells by the repression of C- and activation of LMP-1-promoter.Hyperphosphorylation of EBNA2 by Epstein-Barr virus protein kinase suppresses transactivation of the LMP1 promoter.Establishment and maintenance of long-term murine gammaherpesvirus 68 latency in B cells in the absence of CD40.PARP1 restricts Epstein Barr Virus lytic reactivation by binding the BZLF1 promoter Suppression of tetradecanoyl phorbol acetate-induced lytic reactivation of Kaposi's sarcoma-associated herpesvirus by K1 signal transduction Latent membrane protein 1 inhibits Epstein-Barr virus lytic cycle induction and progress via different mechanisms.Terminal differentiation into plasma cells initiates the replicative cycle of Epstein-Barr virus in vivo.Down-regulation of BLIMP1α by the EBV oncogene, LMP-1, disrupts the plasma cell differentiation program and prevents viral replication in B cells: implications for the pathogenesis of EBV-associated B-cell lymphomas.Suppression of the LMP2A target gene, EGR-1, protects Hodgkin's lymphoma cells from entry to the EBV lytic cycle.Theodore E. Woodward Award: development of novel, EBV-targeted therapies for EBV-positive tumors Molecular parameters for precise diagnosis of asymptomatic Epstein-Barr virus reactivation in healthy carriers NF-kappaB inhibits gammaherpesvirus lytic replication Long-term latent murine Gammaherpesvirus 68 infection is preferentially found within the surface immunoglobulin D-negative subset of splenic B cells in vivo.Maintenance of Epstein-Barr Virus Latent Status by a Novel Mechanism, Latent Membrane Protein 1-Induced Interleukin-32, via the Protein Kinase Cδ Pathway.LMP1-Induced Sumoylation Influences the Maintenance of Epstein-Barr Virus Latency through KAP1 Epstein-Barr virus latent membrane protein 2A is a B-cell receptor mimic and essential for B-cell survival An Epstein-Barr Virus (EBV) mutant with enhanced BZLF1 expression causes lymphomas with abortive lytic EBV infection in a humanized mouse model.Transcriptome changes induced by Epstein-Barr virus LMP1 and LMP2A in transgenic lymphocytes and lymphoma.The cellular ataxia telangiectasia-mutated kinase promotes epstein-barr virus lytic reactivation in response to multiple different types of lytic reactivation-inducing stimuli Systematic mutagenesis of the murine gammaherpesvirus 68 M2 protein identifies domains important for chronic infection.The B-cell-specific transcription factor and master regulator Pax5 promotes Epstein-Barr virus latency by negatively regulating the viral immediate early protein BZLF1.Epstein-Barr virus in systemic autoimmune diseases Assessment of the effect of TLR7/8, TLR9 agonists and CD40 ligand on the transformation efficiency of Epstein-Barr virus in human B lymphocytes by limiting dilution assay.Induction of Epstein-Barr virus latent membrane protein 1 by a lytic transactivator Rta.Dissecting the contribution of EBNA3C domains important for EBV-induced B-cell growth and proliferation.Tumor Suppressor p53 Stimulates the Expression of Epstein-Barr Virus Latent Membrane Protein 1.The role of oxidative stress in EBV lytic reactivation, radioresistance and the potential preventive and therapeutic implications.Arsenic mediated disruption of promyelocytic leukemia protein nuclear bodies induces ganciclovir susceptibility in Epstein-Barr positive epithelial cells.Promoter sequences required for reactivation of Epstein-Barr virus from latency Unexpected absence of the Epstein-Barr virus (EBV) lyLMP-1 open reading frame in tumor virus isolates: lack of correlation between Met129 status and EBV strain identity.Epstein-Barr virus inhibits Kaposi's sarcoma-associated herpesvirus lytic replication in primary effusion lymphomas.CD95 Signaling Inhibits B Cell Receptor-Mediated Gammaherpesvirus Replication in Apoptosis-Resistant B Lymphoma Cells.Epstein-Barr virus latency type and spontaneous reactivation predict lytic induction levels.Epstein-Barr virus latent membrane protein 1 (LMP-1) half-life in epithelial cells is down-regulated by lytic LMP-1.Epstein-Barr virus transforming protein LMP1 plays a critical role in virus production.ZEB1 and c-Jun levels contribute to the establishment of highly lytic Epstein-Barr virus infection in gastric AGS cells.The latent membrane protein 1 of Epstein-Barr virus establishes an antiviral state via induction of interferon-stimulated genes.EBV Infection and Glucose Metabolism in Nasopharyngeal Carcinoma.

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P2860

Control of Epstein-Barr virus reactivation by activated CD40 and viral latent membrane protein 1.

http://www.wikidata.org/entity/Q33898010

description

2001 nî lūn-bûn

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2001 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2001 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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name

Control of Epstein-Barr virus ...... ral latent membrane protein 1.

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Control of Epstein-Barr virus ...... ral latent membrane protein 1.

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Control of Epstein-Barr virus ...... ral latent membrane protein 1.

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Control of Epstein-Barr virus ...... ral latent membrane protein 1.

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Control of Epstein-Barr virus ...... ral latent membrane protein 1.

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Control of Epstein-Barr virus ...... ral latent membrane protein 1.

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P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Control of Epstein-Barr virus ...... ral latent membrane protein 1.

@en

P2093

Barbara Adler

http://www.w3.org/2001/XMLSchema#string

Barbara Baier

http://www.w3.org/2001/XMLSchema#string

Bettina Kempkes

http://www.w3.org/2001/XMLSchema#string

Eveline Schaadt

http://www.w3.org/2001/XMLSchema#string

Ursula Zimber-Strobl

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P2860

The CD40 antigen and its ligand

Epstein-Barr virus latent membrane protein transactivates the human immunodeficiency virus type 1 long terminal repeat through induction of NF-kappa B activity.

Epstein-Barr virus latent membrane protein-1 triggers AP-1 activity via the c-Jun N-terminal kinase cascade.

Epstein-Barr virus-mediated B-cell proliferation is dependent upon latent membrane protein 1, which simulates an activated CD40 receptor

Latent membrane protein 1 of Epstein-Barr virus interacts with JAK3 and activates STAT proteins

Latent membrane protein 1 of Epstein-Barr virus mimics a constitutively active receptor molecule.

Epstein-Barr virus, infectious mononucleosis, and posttransplant lymphoproliferative disorders.

An integral membrane protein (LMP2) blocks reactivation of Epstein-Barr virus from latency following surface immunoglobulin crosslinking.

Epstein-Barr virus latent membrane protein (LMP1) is not sufficient to maintain proliferation of B cells but both it and activated CD40 can prolong their survival.

Stimulation via CD40 can substitute for CD4 T cell function in preventing reactivation of a latent herpesvirus.

P304

437-442

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scholarly article

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10.1073/PNAS.221439999

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P433

1

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99

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P50

Georg W Bornkamm

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2001-12-18T00:00:00Z

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P5875

11605735

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P698

11752411

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P921

Epstein–Barr virus

P932

117578

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