Dual autonomous mitochondrial cell death pathways are activated by Nix/BNip3L and induce cardiomyopathy. - Wikidata - awgldk - TriplyDB

Dual autonomous mitochondrial cell death pathways are activated by Nix/BNip3L and induce cardiomyopathy.

Dual autonomous mitochondrial cell death pathways are activated by Nix/BNip3L and induce cardiomyopathy.

http://www.wikidata.org/entity/Q33927271

about

Cell death and survival through the endoplasmic reticulum-mitochondrial axis Molecular mechanisms that differentiate apoptosis from programmed necrosis Programmed necrosis in acute kidney injury Mitoconfusion: noncanonical functioning of dynamism factors in static mitochondria of the heart Functional implications of mitofusin 2-mediated mitochondrial-SR tethering Mitochondrial fusion and fission proteins as novel therapeutic targets for treating cardiovascular disease Bax and Bak function as the outer membrane component of the mitochondrial permeability pore in regulating necrotic cell death in mice Molecular mechanisms of mitochondrial autophagy/mitophagy in the heart Tcf19 is a novel islet factor necessary for proliferation and survival in the INS-1 β-cell line Naringenin prevents high glucose-induced mitochondria-mediated apoptosis involving AIF, Endo-G and caspases A conserved MADS-box phosphorylation motif regulates differentiation and mitochondrial function in skeletal, cardiac, and smooth muscle cells Myocardial MiR-30 downregulation triggered by doxorubicin drives alterations in β-adrenergic signaling and enhances apoptosis Nix Nought Nothing: fairy tale or real deal.Protective role of morin, a flavonoid, against high glucose induced oxidative stress mediated apoptosis in primary rat hepatocytes.Apoptotic cell death "Nixed" by an ER-mitochondrial necrotic pathway.Super-suppression of mitochondrial reactive oxygen species signaling impairs compensatory autophagy in primary mitophagic cardiomyopathy.Two close, too close: sarcoplasmic reticulum-mitochondrial crosstalk and cardiomyocyte fate Loss of Nix in Pdx1-deficient mice prevents apoptotic and necrotic β cell death and diabetes Mitochondrial genome linearization is a causative factor for cardiomyopathy in mice and Drosophila.Cardiac muscle regeneration: lessons from development.Bnip3 impairs mitochondrial bioenergetics and stimulates mitochondrial turnover Role of the mitochondrion in programmed necrosis The protective role of 5-HMF against hypoxic injury.Mechanisms and biology of B-cell leukemia/lymphoma 2/adenovirus E1B interacting protein 3 and Nip-like protein X.The mitochondrial dynamism-mitophagy-cell death interactome: multiple roles performed by members of a mitochondrial molecular ensemble.Identification of differentially expressed transcripts and pathways in blood one week and six months following implant of left ventricular assist devices.Mitochondrial fission and fusion factors reciprocally orchestrate mitophagic culling in mouse hearts and cultured fibroblasts Mechanisms of non-apoptotic programmed cell death in diabetes and heart failure.MicroRNA-214 protects the mouse heart from ischemic injury by controlling Ca²⁺ overload and cell death.Bcl-2 family in inter-organelle modulation of calcium signaling; roles in bioenergetics and cell survival.Programmed Necrosis: A Prominent Mechanism of Cell Death following Neonatal Brain Injury Dichotomy between RIP1- and RIP3-mediated necroptosis in tumor necrosis factor-α-induced shock.Repetitive stimulation of autophagy-lysosome machinery by intermittent fasting preconditions the myocardium to ischemia-reperfusion injury.BNip3 regulates mitochondrial function and lipid metabolism in the liver.A short linear motif in BNIP3L (NIX) mediates mitochondrial clearance in reticulocytes.Modulation of serines 17 and 24 in the LC3-interacting region of Bnip3 determines pro-survival mitophagy versus apoptosis.An involvement of oxidative stress in endoplasmic reticulum stress and its associated diseases.A nucleus-targeted alternately spliced Nix/Bnip3L protein isoform modifies nuclear factor κB (NFκB)-mediated cardiac transcription.Autophagy and mitophagy in the myocardium: therapeutic potential and concerns.Mitochondrial dysfunction and mitophagy: the beginning and end to diabetic nephropathy?

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P2860

Dual autonomous mitochondrial cell death pathways are activated by Nix/BNip3L and induce cardiomyopathy.

http://www.wikidata.org/entity/Q33927271

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Dual autonomous mitochondrial ...... p3L and induce cardiomyopathy.

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Dual autonomous mitochondrial ...... p3L and induce cardiomyopathy.

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type

label

Dual autonomous mitochondrial ...... p3L and induce cardiomyopathy.

@ast

Dual autonomous mitochondrial ...... p3L and induce cardiomyopathy.

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prefLabel

Dual autonomous mitochondrial ...... p3L and induce cardiomyopathy.

@ast

Dual autonomous mitochondrial ...... p3L and induce cardiomyopathy.

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Proceedings of the National Academy of Sciences of the United States of America

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Dual autonomous mitochondrial ...... p3L and induce cardiomyopathy.

@en

P2093

Abhinav Diwan

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Emily H-Y Cheng

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Gerald W Dorn

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Scot J Matkovich

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William Lewis

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Yun Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Nix is a selective autophagy receptor for mitochondrial clearance

BNIP3 and genetic control of necrosis-like cell death through the mitochondrial permeability transition pore

The E1B 19K/Bcl-2-binding protein Nip3 is a dimeric mitochondrial protein that activates apoptosis

Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stress

Cell death: critical control points

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

Cell death in the nervous system

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

Essential role for Nix in autophagic maturation of erythroid cells

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