Super-suppression of mitochondrial reactive oxygen species signaling impairs compensatory autophagy in primary mitophagic cardiomyopathy. - Wikidata - awgldk - TriplyDB

Super-suppression of mitochondrial reactive oxygen species signaling impairs compensatory autophagy in primary mitophagic cardiomyopathy.

Super-suppression of mitochondrial reactive oxygen species signaling impairs compensatory autophagy in primary mitophagic cardiomyopathy.

http://www.wikidata.org/entity/Q33931661

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Mitochondrial Regulation of the Muscle Microenvironment in Critical Limb Ischemia Mitochondrial biogenesis and dynamics in the developing and diseased heart An experimental approach to study the function of mitochondria in cardiomyopathy Mitochondrial dynamism and heart disease: changing shape and shaping change Mending a broken heart: the role of mitophagy in cardioprotection Mitoconfusion: noncanonical functioning of dynamism factors in static mitochondria of the heart Functional implications of mitofusin 2-mediated mitochondrial-SR tethering Akap1 Deficiency Promotes Mitochondrial Aberrations and Exacerbates Cardiac Injury Following Permanent Coronary Ligation via Enhanced Mitophagy and Apoptosis Mitochondrial fusion and fission proteins as novel therapeutic targets for treating cardiovascular disease New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases Molecular mechanisms mediating mitochondrial dynamics and mitophagy and their functional roles in the cardiovascular system Mitophagy and heart failure Molecular mechanisms of mitochondrial autophagy/mitophagy in the heart Staying young at heart: autophagy and adaptation to cardiac aging Targeting mitochondrial dysfunction in the treatment of heart failure.European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Mitochondria and ageing: role in heart, skeletal muscle and adipose tissue.The mitochondrial dynamism-mitophagy-cell death interactome: multiple roles performed by members of a mitochondrial molecular ensemble.Mitochondrial fission and fusion factors reciprocally orchestrate mitophagic culling in mouse hearts and cultured fibroblasts Mitochondrial reactive oxygen species at the heart of the matter: new therapeutic approaches for cardiovascular diseases.Mitochondrial quality control: Easy come, easy go Interdependence of Parkin-Mediated Mitophagy and Mitochondrial Fission in Adult Mouse Hearts.Obesity-induced changes in kidney mitochondria and endoplasmic reticulum in the presence or absence of leptin Defective insulin signaling and mitochondrial dynamics in diabetic cardiomyopathy.Parkin-mediated mitophagy directs perinatal cardiac metabolic maturation in mice Mitochondrial Reactive Oxygen Species Mediate Cardiac Structural, Functional, and Mitochondrial Consequences of Diet-Induced Metabolic Heart Disease Nutrient-sensing mTORC1: Integration of metabolic and autophagic signals Hearts deficient in both Mfn1 and Mfn2 are protected against acute myocardial infarction.Far-infrared radiation protects viability in a cell model of Spinocerebellar Ataxia by preventing polyQ protein accumulation and improving mitochondrial function Mitochondrial fission/fusion and cardiomyopathy.Mitochondrial-Shaping Proteins in Cardiac Health and Disease - the Long and the Short of It!Expert consensus document: Mitochondrial function as a therapeutic target in heart failure How mitochondrial dynamism orchestrates mitophagy.Parkin-dependent mitophagy in the heart.Mitochondrial dynamics and cell death in heart failure.Central Parkin: The evolving role of Parkin in the heart.Autophagy capacity and sub-mitochondrial heterogeneity shape Bnip3-induced mitophagy regulation of apoptosis.Rays Sting: The Acute Cellular Effects of Ionizing Radiation Exposure.Mitochondrial Dynamics as a Therapeutic Target for Treating Cardiac Diseases.Autophagy as a regulator of cardiovascular redox homeostasis.

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P2860

Super-suppression of mitochondrial reactive oxygen species signaling impairs compensatory autophagy in primary mitophagic cardiomyopathy.

http://www.wikidata.org/entity/Q33931661

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Super-suppression of mitochond ...... ary mitophagic cardiomyopathy.

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Super-suppression of mitochond ...... ary mitophagic cardiomyopathy.

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type

label

Super-suppression of mitochond ...... ary mitophagic cardiomyopathy.

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Super-suppression of mitochond ...... ary mitophagic cardiomyopathy.

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prefLabel

Super-suppression of mitochond ...... ary mitophagic cardiomyopathy.

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Super-suppression of mitochond ...... ary mitophagic cardiomyopathy.

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CIRCRESAHA.115.304384

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Circulation Research

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Super-suppression of mitochond ...... ary mitophagic cardiomyopathy.

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P2093

Elizabeth Murphy

http://www.w3.org/2001/XMLSchema#string

Gerald W Dorn

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Guohua Gong

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Moshi Song

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Peter S Rabinovitch

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Yun Chen

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P2860

PINK1 is selectively stabilized on impaired mitochondria to activate Parkin

Redox regulation of protein tyrosine phosphatase 1B involves a sulphenyl-amide intermediate

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

PINK1-phosphorylated mitofusin 2 is a Parkin receptor for culling damaged mitochondria

A role for ubiquitin in selective autophagy

Extension of murine life span by overexpression of catalase targeted to mitochondria

PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy

Mitofusin-2 maintains mitochondrial structure and contributes to stress-induced permeability transition in cardiac myocytes.

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

Dual autonomous mitochondrial cell death pathways are activated by Nix/BNip3L and induce cardiomyopathy.

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3

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115

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4106429

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