Transcriptional regulation of the human papillomavirus-16 E6-E7 promoter by a keratinocyte-dependent enhancer, and by viral E2 trans-activator and repressor gene products: implications for cervical carcinogenesis.
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Cloning and functional analysis of spliced isoforms of human nuclear factor I-X: interference with transcriptional activation by NFI/CTF in a cell-type specific mannerAMF-1/Gps2 binds p300 and enhances its interaction with papillomavirus E2 proteinsThe human 230-kD bullous pemphigoid antigen gene (BPAG1). Exon-intron organization and identification of regulatory tissue specific elements in the promoter regionThe opposite and antagonistic effects of the closely related POU family transcription factors Brn-3a and Brn-3b on the activity of a target promoter are dependent on differences in the POU domainDifferent papillomaviruses have different repertoires of transcription factor binding sites: convergence and divergence in the upstream regulatory region.Two novel promoters in the upstream regulatory region of human papillomavirus type 31b are negatively regulated by epithelial differentiationThe chromatin structure of the long control region of human papillomavirus type 16 represses viral oncoprotein expression.Human papillomavirus type 16 cooperates with activated ras and fos oncogenes in the hormone-dependent transformation of primary mouse cells.The HPV Cellular Transactivator Brn-3a Can Be Used to Predict Cervical Adenocarcinoma and Squamous Carcinoma Precancer Lesions in the Developed and Developing Worlds.Induction of the human papillomavirus type 31 late promoter requires differentiation but not DNA amplificationVariant upstream regulatory region sequences differentially regulate human papillomavirus type 16 DNA replication throughout the viral life cycle.Novel binding sites for regulatory factors in the human papillomavirus type 18 enhancer and promoter identified by in vivo footprintingTransactivation-competent bovine papillomavirus E2 protein is specifically required for efficient repression of human papillomavirus oncogene expression and for acute growth inhibition of cervical carcinoma cell linesTransactivation by the E2 protein of oncogenic human papillomavirus type 31 is not essential for early and late viral functions.Role of the E1--E4 protein in the differentiation-dependent life cycle of human papillomavirus type 31.Nuclear matrix attachment regions of human papillomavirus type 16 repress or activate the E6 promoter, depending on the physical state of the viral DNA.Mechanisms of human papillomavirus E2-mediated repression of viral oncogene expression and cervical cancer cell growth inhibition.Tumourigenesis driven by the human papillomavirus type 16 Asian-American e6 variant in a three-dimensional keratinocyte modelAutostimulation of the Epstein-Barr virus BRLF1 promoter is mediated through consensus Sp1 and Sp3 binding sites.Cervical cancer stem cells manifest radioresistance: Association with upregulated AP-1 activity.The bovine papillomavirus constitutive enhancer is essential for viral transformation, DNA replication, and the maintenance of latencyTranscriptional enhancer factor (TEF)-1 and its cell-specific co-activator activate human papillomavirus-16 E6 and E7 oncogene transcription in keratinocytes and cervical carcinoma cells.Retinoic acid-mediated repression of human papillomavirus 18 transcription and different ligand regulation of the retinoic acid receptor beta gene in non-tumorigenic and tumorigenic HeLa hybrid cellsHuman papillomaviruses and the interferon responseGenetic analysis of cis regulatory elements within the 5' region of the human papillomavirus type 31 upstream regulatory region during different stages of the viral life cycleHuman TATA binding protein inhibits human papillomavirus type 11 DNA replication by antagonizing E1-E2 protein complex formation on the viral origin of replication.Pathogenesis of human papillomaviruses in differentiating epithelia.A transactivator function of cottontail rabbit papillomavirus e2 is essential for tumor induction in rabbits.Human papillomavirus type 16 (HPV-16) genomes integrated in head and neck cancers and in HPV-16-immortalized human keratinocyte clones express chimeric virus-cell mRNAs similar to those found in cervical cancers.Human papillomavirus type 31 replication modes during the early phases of the viral life cycle depend on transcriptional and posttranscriptional regulation of E1 and E2 expression.The octamer binding site in the HPV16 regulatory region produces opposite effects on gene expression in cervical and non-cervical cellsDuring negative regulation of the human papillomavirus-16 E6 promoter, the viral E2 protein can displace Sp1 from a proximal promoter element.Transactivation of the human papilloma virus 16 octamer motif by the octamer binding protein Oct-2 requires both the N and C terminal activation domains.Brd4 is required for e2-mediated transcriptional activation but not genome partitioning of all papillomaviruses.Replication and partitioning of papillomavirus genomes.Clusters of nuclear factor I binding sites identify enhancers of several papillomaviruses but alone are not sufficient for enhancer function.The constitutively expressed octamer binding protein OTF-1 and a novel octamer binding protein expressed specifically in cervical cells bind to an octamer-related sequence in the human papillomavirus 16 enhancer.Delineation of human papillomavirus type 18 enhancer binding proteins: the intracellular distribution of a novel octamer binding protein p92 is cell cycle regulated.Transcription of the transforming genes of the oncogenic human papillomavirus-16 is stimulated by tumor promotors through AP1 binding sites.Neuroepithelial carcinomas in mice transgenic with human papillomavirus type 16 E6/E7 ORFs
P2860
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P2860
Transcriptional regulation of the human papillomavirus-16 E6-E7 promoter by a keratinocyte-dependent enhancer, and by viral E2 trans-activator and repressor gene products: implications for cervical carcinogenesis.
description
1987 nî lūn-bûn
@nan
1987 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1987 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
name
Transcriptional regulation of ...... s for cervical carcinogenesis.
@ast
Transcriptional regulation of ...... s for cervical carcinogenesis.
@en
type
label
Transcriptional regulation of ...... s for cervical carcinogenesis.
@ast
Transcriptional regulation of ...... s for cervical carcinogenesis.
@en
prefLabel
Transcriptional regulation of ...... s for cervical carcinogenesis.
@ast
Transcriptional regulation of ...... s for cervical carcinogenesis.
@en
P2093
P2860
P1433
P1476
Transcriptional regulation of ...... s for cervical carcinogenesis.
@en
P2093
Gissmann L
Schmid PG 3rd
Tabatabai F
P2860
P304
P407
P577
1987-12-01T00:00:00Z