A defect in mismatch repair in Saccharomyces cerevisiae stimulates ectopic recombination between homeologous genes by an excision repair dependent process.
about
Sister chromatids are preferred over homologs as substrates for recombinational repair in Saccharomyces cerevisiaeRequirement for three novel protein complexes in the absence of the Sgs1 DNA helicase in Saccharomyces cerevisiaeInteraction between mismatch repair and genetic recombination in Saccharomyces cerevisiae.Rapamycin sensitivity in Saccharomyces cerevisiae is mediated by a peptidyl-prolyl cis-trans isomerase related to human FK506-binding proteinMultiple transcriptional activation complexes tether the yeast activator Met4 to DNA.Genome rearrangement in top3 mutants of Saccharomyces cerevisiae requires a functional RAD1 excision repair gene.The Saccharomyces cerevisiae RAD9 checkpoint reduces the DNA damage-associated stimulation of directed translocations.Mismatch repair proteins regulate heteroduplex formation during mitotic recombination in yeast.Pathways and Mechanisms that Prevent Genome Instability in Saccharomyces cerevisiaeGene conversions and crossing over during homologous and homeologous ectopic recombination in Saccharomyces cerevisiae.Multiple pathways for homologous recombination in Saccharomyces cerevisiae.Mismatch correction acts as a barrier to homeologous recombination in Saccharomyces cerevisiaeHomologous and homeologous intermolecular gene conversion are not differentially affected by mutations in the DNA damage or the mismatch repair genes RAD1, RAD50, RAD51, RAD52, RAD54, PMS1 and MSH2Novel function of Rad27 (FEN-1) in restricting short-sequence recombination.Highly mismatched molecules resembling recombination intermediates efficiently transform mismatch repair proficient Escherichia coli.Gene conversion tracts from double-strand break repair in mammalian cells.DNA-dependent protein kinase suppresses double-strand break-induced and spontaneous homologous recombination.The Alu-rich genomic architecture of SPAST predisposes to diverse and functionally distinct disease-associated CNV alleles.Structure-function analysis of yeast mRNA cap methyltransferase and high-copy suppression of conditional mutants by AdoMet synthase and the ubiquitin conjugating enzyme Cdc34p.(CA/TG) microsatellite sequences escape the inhibition of recombination by mismatch repair in Saccharomyces cerevisiae.Ku-dependent and Ku-independent end-joining pathways lead to chromosomal rearrangements during double-strand break repair in Saccharomyces cerevisiae.The effects of mismatch repair and RAD1 genes on interchromosomal crossover recombination in Saccharomyces cerevisiae.SCF(Met30)-mediated control of the transcriptional activator Met4 is required for the G(1)-S transitionIdentification of a chicken RAD52 homologue suggests conservation of the RAD52 recombination pathway throughout the evolution of higher eukaryotes.Alleles of the homologous recombination gene, RAD59, identify multiple responses to disrupted DNA replication in Saccharomyces cerevisiaeMultiple recombination pathways for sister chromatid exchange in Saccharomyces cerevisiae: role of RAD1 and the RAD52 epistasis group genes.Cloning-free PCR-based allele replacement methods.Double-strand break repair by interchromosomal recombination: suppression of chromosomal translocations.Nucleotide excision repair gene function in short-sequence recombination.The role of recombination and RAD52 in mutation of chromosomal DNA transformed into yeastA mutant allele of the transcription factor IIH helicase gene, RAD3, promotes loss of heterozygosity in response to a DNA replication defect in Saccharomyces cerevisiae.Microhomology directs diverse DNA break repair pathways and chromosomal translocations.The essential helicase gene RAD3 suppresses short-sequence recombination in Saccharomyces cerevisiae.Mitotic crossovers between diverged sequences are regulated by mismatch repair proteins in Saccaromyces cerevisiaeEffects of terminal nonhomology and homeology on double-strand-break-induced gene conversion tract directionality.Influence of DNA sequence identity on efficiency of targeted gene replacement.Fine-resolution analysis of products of intrachromosomal homeologous recombination in mammalian cells.Dual requirement in yeast DNA mismatch repair for MLH1 and PMS1, two homologs of the bacterial mutL gene.Homologous, homeologous, and illegitimate repair of double-strand breaks during transformation of a wild-type strain and a rad52 mutant strain of Saccharomyces cerevisiae.Homologous recombination of monkey alpha-satellite repeats in an in vitro simian virus 40 replication system: possible association of recombination with DNA replication.
P2860
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P2860
A defect in mismatch repair in Saccharomyces cerevisiae stimulates ectopic recombination between homeologous genes by an excision repair dependent process.
description
1990 nî lūn-bûn
@nan
1990 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1990年の論文
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1990年学术文章
@wuu
1990年学术文章
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1990年学术文章
@zh-hans
1990年学术文章
@zh-my
1990年学术文章
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1990年學術文章
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name
A defect in mismatch repair in ...... sion repair dependent process.
@ast
A defect in mismatch repair in ...... sion repair dependent process.
@en
type
label
A defect in mismatch repair in ...... sion repair dependent process.
@ast
A defect in mismatch repair in ...... sion repair dependent process.
@en
prefLabel
A defect in mismatch repair in ...... sion repair dependent process.
@ast
A defect in mismatch repair in ...... sion repair dependent process.
@en
P2860
P1433
P1476
A defect in mismatch repair in ...... sion repair dependent process.
@en
P2093
A M Bailis
R Rothstein
P2860
P304
P407
P577
1990-11-01T00:00:00Z