The c-fos proto-oncogene is a target for transactivation by the p53 tumor suppressor.
about
The transcription factor p53: not a repressor, solely an activatorMCG10, a novel p53 target gene that encodes a KH domain RNA-binding protein, is capable of inducing apoptosis and cell cycle arrest in G(2)-MUV-Induced stabilization of c-fos and other short-lived mRNAsMLL2 is required in oocytes for bulk histone 3 lysine 4 trimethylation and transcriptional silencingPhenotype anchoring in zebrafish reveals a potential role for matrix metalloproteinases (MMPs) in tamoxifen's effects on skin epithelium.Identification of genes up-regulated by retinoic-acid-induced differentiation of the human neuronal precursor cell line NTERA-2 cl.D1.Molecular interaction map of the mammalian cell cycle control and DNA repair systems.p53 regulation by post-translational modification and nuclear retention in response to diverse stresses.Differential transactivation by the p53 transcription factor is highly dependent on p53 level and promoter target sequence.Negative cross-talk between p53 and the glucocorticoid receptor and its role in neuroblastoma cells.The tumor suppressor p53 inhibits Net, an effector of Ras/extracellular signal-regulated kinase signalingCyclin-dependent kinase 2 (CDK2) is a key mediator for EGF-induced cell transformation mediated through the ELK4/c-Fos signaling pathway.Signalling pathways for transactivation by dexmedetomidine of epidermal growth factor receptors in astrocytes and its paracrine effect on neurons.Cell type-specific gene expression and editing responses to chronic fluoxetine treatment in the in vivo mouse brain and their relevance for stress-induced anhedonia.Specific interaction of p53 with target binding sites is determined by DNA conformation and is regulated by the C-terminal domain.Platelet-activating factor induces proliferation in differentiated keratinocytes.A novel AP-1 element in the CD95 ligand promoter is required for induction of apoptosis in hepatocellular carcinoma cells upon treatment with anticancer drugs.A novel p53 mutational hotspot in skin tumors from UV-irradiated Xpc mutant mice alters transactivation functions.p53 down-regulates human bradykinin B1 receptor gene expression.Reversal of 5-flouroucial resistance by adenovirus-mediated transfer of wild-type p53 gene in multidrug-resistant human colon carcinoma LoVo/5-FU cells.Inhibition of the putative tumor suppressor gene TIMP-3 by tumor-derived p53 mutants and wild type p53.Fluoxetine-mediated 5-HT2B receptor stimulation in astrocytes causes EGF receptor transactivation and ERK phosphorylation.Biphasic Regulation of Caveolin-1 Gene Expression by Fluoxetine in Astrocytes: Opposite Effects of PI3K/AKT and MAPK/ERK Signaling Pathways on c-fos.
P2860
Q24273295-CF2B4E28-70EB-4EE8-9E15-200D9B24E6F1Q24552865-C2E19A9D-5316-4DED-9553-43E883AD2AD3Q24554221-3F77E4E6-5923-4A87-956A-BB1A6DECEAC4Q27323960-C0800BE4-6048-4285-BFB2-A20C322881D5Q30727473-0B326FD2-52F0-4913-BEC1-07A9EF879CA4Q31832242-7877C657-5335-4AAF-BA60-055BE215026AQ33704559-FDB841B3-2B18-44B7-8978-EFBBA3ADFC85Q33807807-FCC8299E-0624-4AF4-8EC8-E4FE7A345D50Q34443272-C3E82B08-8000-4DCF-9BE2-D184BFC7B7DEQ34677800-AB90E86C-233F-41F7-91FE-F0A666A840C0Q35544832-9CA0D7CD-D335-49F6-8417-69D05F834447Q36338868-865B5EFB-1CF3-4FCE-8C58-157620EAAABEQ36735872-9E8A3222-DBDA-4DFB-B2B8-FBA89C0106B0Q38019843-07765006-8846-42CA-8089-DC5D9D5CE6A4Q38363728-7C2EFE49-B13A-4E29-931C-DF1D3526DCF8Q39105282-F8D5BABC-7D66-46B9-AB3B-31F55AAFAFE2Q39456231-98978126-7D2C-40EB-B3E6-52172211ED8AQ40711394-1F9E3B71-8568-491A-AE03-D448FBE762FDQ40799379-540BAEAE-722A-4476-A0E8-C68673DED6F5Q43248608-1ED096FF-8DE7-48F6-9426-C8F0158058F9Q43831424-E45BB857-11A6-4E0C-969B-7A9D8AFC9565Q46402539-BF0AFA2E-1729-41AC-80EF-2DD5ABAA322CQ47152588-4D373FD9-0B7B-4686-9939-89A26F6183EB
P2860
The c-fos proto-oncogene is a target for transactivation by the p53 tumor suppressor.
description
1999 nî lūn-bûn
@nan
1999 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
The c-fos proto-oncogene is a target for transactivation by the p53 tumor suppressor.
@ast
The c-fos proto-oncogene is a target for transactivation by the p53 tumor suppressor.
@en
type
label
The c-fos proto-oncogene is a target for transactivation by the p53 tumor suppressor.
@ast
The c-fos proto-oncogene is a target for transactivation by the p53 tumor suppressor.
@en
prefLabel
The c-fos proto-oncogene is a target for transactivation by the p53 tumor suppressor.
@ast
The c-fos proto-oncogene is a target for transactivation by the p53 tumor suppressor.
@en
P2093
P2860
P356
P1476
The c-fos proto-oncogene is a target for transactivation by the p53 tumor suppressor
@en
P2093
P2860
P304
P356
10.1128/MCB.19.4.2594
P407
P577
1999-04-01T00:00:00Z