Fluoxetine-mediated 5-HT2B receptor stimulation in astrocytes causes EGF receptor transactivation and ERK phosphorylation.
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Role of Glycogenolysis in Memory and Learning: Regulation by Noradrenaline, Serotonin and ATPSignal Transduction in Astrocytes during Chronic or Acute Treatment with Drugs (SSRIs, Antibipolar Drugs, GABA-ergic Drugs, and Benzodiazepines) Ameliorating Mood DisordersFluoxetine affects GluK2 editing, glutamate-evoked Ca(2+) influx and extracellular signal-regulated kinase phosphorylation in mouse astrocytes.Chronic SSRI stimulation of astrocytic 5-HT2B receptors change multiple gene expressions/editings and metabolism of glutamate, glucose and glycogen: a potential paradigm shift.Serotonin receptors and heart valve disease--it was meant 2B.Fluoxetine and all other SSRIs are 5-HT2B Agonists - Importance for their Therapeutic Effects.Docosahexaenoic acid suppresses neuroinflammatory responses and induces heme oxygenase-1 expression in BV-2 microglia: implications of antidepressant effects for ω-3 fatty acids.5-HT(2B) receptors are required for serotonin-selective antidepressant actionsReactive oxygen species are required for 5-HT-induced transactivation of neuronal platelet-derived growth factor and TrkB receptors, but not for ERK1/2 activation.Fluoxetine induces alkalinization of astroglial cytosol through stimulation of sodium-hydrogen exchanger 1: dissection of intracellular signaling pathwaysDecrease of gene expression of astrocytic 5-HT2B receptors parallels development of depressive phenotype in a mouse model of Parkinson's disease.Cell type-specific gene expression and editing responses to chronic fluoxetine treatment in the in vivo mouse brain and their relevance for stress-induced anhedonia.5-HT2 ligands in the treatment of anxiety and depression.Current concepts in diagnosis and perioperative management of carcinoid heart disease.Astrocytic glycogenolysis: mechanisms and functions.Serotonin mediation of early memory formation via 5-HT2B receptor-induced glycogenolysis in the day-old chick.LPA1 Mediates Antidepressant-Induced ERK1/2 Signaling and Protection from Oxidative Stress in Glial Cells.Bi-phasic regulation of glycogen content in astrocytes via Cav-1/PTEN/PI3K/AKT/GSK-3β pathway by fluoxetine.Fluoxetine ameliorates behavioral and neuropathological deficits in a transgenic model mouse of α-synucleinopathy.Trazodone regulates neurotrophic/growth factors, mitogen-activated protein kinases and lactate release in human primary astrocytes.Sequential Astrocytic 5-HT2B Receptor Stimulation, [Ca(2+)]i Regulation, Glycogenolysis, Glutamate Synthesis, and K(+) Homeostasis are Similar but Not Identical in Learning and Mood Regulation.Response: Commentary: Chronic SSRI Stimulation of Astrocytic 5-HT2B Receptors Change Multiple Gene Expressions/Editings and Metabolism of Glutamate, Glucose and Glycogen: A Potential Paradigm Shift.5-HT2B receptors are expressed on astrocytes from brain and in culture and are a chronic target for all five conventional 'serotonin-specific reuptake inhibitors'.Serotonin and histamine mediate gastroprotective effect of fluoxetine against experimentally-induced ulcers in rats.Biphasic Regulation of Caveolin-1 Gene Expression by Fluoxetine in Astrocytes: Opposite Effects of PI3K/AKT and MAPK/ERK Signaling Pathways on c-fos.The ameliorative effect of fluoxetine on neuroinflammation induced by sleep deprivation.Fibroblast growth factor 2 mRNA expression evoked by amitriptyline involves extracellular signal-regulated kinase-dependent early growth response 1 production in rat primary cultured astrocytes.Cell type-specific in vivo expression of genes encoding signalling molecules in the brain in response to chronic mild stress and chronic treatment with fluoxetine.Ammonium increases Ca(2+) signalling and upregulates expression of Cav1.2 gene in astrocytes in primary cultures and in the in vivo brain.Ammonium Increases TRPC1 Expression Via Cav-1/PTEN/AKT/GSK3β Pathway.Expression of nucleoside transporter in freshly isolated neurons and astrocytes from mouse brain.Regulation of astrocyte pathology by fluoxetine prevents the deterioration of Alzheimer phenotypes in an APP/PS1 mouse model.Echocardiography in functional midgut neuroendocrine tumors: When and how often.Epidermal growth factor (EGF)-receptor signalling is needed for murine beta cell mass expansion in response to high-fat diet and pregnancy but not after pancreatic duct ligation.The Effect of Antidepressants on Mesenchymal Stem Cell Differentiation.
P2860
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P2860
Fluoxetine-mediated 5-HT2B receptor stimulation in astrocytes causes EGF receptor transactivation and ERK phosphorylation.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Fluoxetine-mediated 5-HT2B rec ...... ation and ERK phosphorylation.
@en
Fluoxetine-mediated 5-HT2B rec ...... ation and ERK phosphorylation.
@nl
type
label
Fluoxetine-mediated 5-HT2B rec ...... ation and ERK phosphorylation.
@en
Fluoxetine-mediated 5-HT2B rec ...... ation and ERK phosphorylation.
@nl
prefLabel
Fluoxetine-mediated 5-HT2B rec ...... ation and ERK phosphorylation.
@en
Fluoxetine-mediated 5-HT2B rec ...... ation and ERK phosphorylation.
@nl
P2093
P2860
P1433
P1476
Fluoxetine-mediated 5-HT2B rec ...... ation and ERK phosphorylation.
@en
P2093
Hongyan Zhang
Liang Peng
Liping Cai
Shiquen Zhang
P2860
P2888
P304
P356
10.1007/S00213-008-1306-5
P50
P577
2008-08-29T00:00:00Z