Correlation between KIT expression and KIT mutation in melanoma: a study of 173 cases with emphasis on the acral-lentiginous/mucosal type - Wikidata - awgldk - TriplyDB

Correlation between KIT expression and KIT mutation in melanoma: a study of 173 cases with emphasis on the acral-lentiginous/mucosal type

Correlation between KIT expression and KIT mutation in melanoma: a study of 173 cases with emphasis on the acral-lentiginous/mucosal type

http://www.wikidata.org/entity/Q33995919

about

Analysis of the genome to personalize therapy for melanoma Efficacy and safety of nilotinib in patients with KIT-mutated metastatic or inoperable melanoma: final results from the global, single-arm, phase II TEAM trial Melanoma of the hand and foot: epidemiological, prognostic and genetic features. A systematic review.Mucosal melanomas: a case-based review of the literature.Phase II Trial of Nilotinib in Patients With Metastatic Malignant Melanoma Harboring KIT Gene Aberration: A Multicenter Trial of Korean Cancer Study Group (UN10-06).Noninvasive genomic detection of melanoma.Analysis of Somatic Mutations in Cancer: Molecular Mechanisms of Activation in the ErbB Family of Receptor Tyrosine Kinases.Multiplex mutation screening by mass spectrometry evaluation of 820 cases from a personalized cancer medicine registry NUAK2: an emerging acral melanoma oncogene Phase I clinical trial of the Src inhibitor dasatinib with dacarbazine in metastatic melanoma Oncogenic mutations in melanomas and benign melanocytic nevi of the female genital tract.Comparison of c-Kit expression between primary and metastatic melanoma of skin and mucosa.The usefulness of c-Kit in the immunohistochemical assessment of melanocytic lesions Additional Primary Malignancies in Patients with Gastrointestinal Stromal Tumor (GIST): A Clinicopathologic Study of 260 Patients with Molecular Analysis and Review of the Literature.The current state of targeted therapy in melanoma: this time it's personal Delving into somatic variation in sporadic melanoma.Expression of Sox10 and c-kit in sinonasal mucosal melanomas arising in the Chinese population Concordance of c-kit mutational status in matched primary and metastatic cutaneous canine mast cell tumors at baseline Diagnostic criteria, specific mutations, and genetic predisposition in gastrointestinal stromal tumors.Oncogene abnormalities in a series of primary melanomas of the sinonasal tract: NRAS mutations and cyclin D1 amplification are more frequent than KIT or BRAF mutations.Targeting KIT in melanoma: a paradigm of molecular medicine and targeted therapeutics.Use of immunohistochemistry in the diagnosis of melanocytic lesions: applications and pitfalls.The role of tissue microarray in the era of target-based agents.Molecular testing in malignant melanoma.Update on the targeted therapy of melanoma.The potential role of sunitinib targeting melanomas.Treating advanced melanoma: current insights and opportunities.KIT Mutations and CD117 Overexpression Are Markers of Better Progression-Free Survival in Vulvar Melanomas.A 2015 update on predictive molecular pathology and its role in targeted cancer therapy: a review focussing on clinical relevance.A potential role for targeted therapy in a subset of metastasizing adnexal carcinomas.Targeting the Cellular Signaling: BRAF Inhibition and Beyond for the Treatment of Metastatic Malignant Melanoma Vulvar and vaginal melanoma: A unique subclass of mucosal melanoma based on a comprehensive molecular analysis of 51 cases compared with 2253 cases of nongynecologic melanoma.Molecular alterations in malignant blue nevi and related blue lesions.Cell proliferation in cutaneous malignant melanoma: relationship with neoplastic progression.A case series of anal melanoma including the results of treatment with imatinib in selected patients.Analysis of KIT mutations and c-KIT expression in Chinese Uyghur and Han patients with melanoma.KIT mutations in ocular melanoma: frequency and anatomic distribution.Spectrum of mutations in gastrointestinal stromal tumor patients - a population-based study from Slovakia.Acral melanoma foot lesions. Part 1: epidemiology, aetiology, and molecular pathology.Tumor infiltrating lymphocytes in acral lentiginous melanoma: a study of a large cohort of cases from Latin America.

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Correlation between KIT expression and KIT mutation in melanoma: a study of 173 cases with emphasis on the acral-lentiginous/mucosal type

http://www.wikidata.org/entity/Q33995919

description

2009 nî lūn-bûn

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2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2009 թվականի օգոստոսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Correlation between KIT expres ...... acral-lentiginous/mucosal type

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Correlation between KIT expres ...... acral-lentiginous/mucosal type

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Correlation between KIT expres ...... acral-lentiginous/mucosal type

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Correlation between KIT expres ...... acral-lentiginous/mucosal type

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Correlation between KIT expres ...... acral-lentiginous/mucosal type

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Correlation between KIT expres ...... acral-lentiginous/mucosal type

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MODPATHOL.2009.116

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Modern Pathology

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Correlation between KIT expres ...... acral-lentiginous/mucosal type

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Carlos A Torres-Cabala

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Dan Yang

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Doina Ivan

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Michael Davies

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Scott Woodman

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P2860

Immunohistochemical determination of HER-2/neu overexpression in malignant melanoma reveals no prognostic value, while c-Kit (CD117) overexpression exhibits potential therapeutic implications

KIT mutations and dose selection for imatinib in patients with advanced gastrointestinal stromal tumours.

L576P KIT mutation in anal melanomas correlates with KIT protein expression and is sensitive to specific kinase inhibition.

Major response to imatinib mesylate in KIT-mutated melanoma.

Activating c-kit gene mutations in human germ cell tumors

Primary melanoma of the skin and cutaneous melanomatous metastases: comparative histologic features and immunophenotypes.

Anorectal malignant melanoma: morphologic and immunohistochemical features.

c-Kit-kinase induces a cascade of protein tyrosine phosphorylation in normal human melanocytes in response to mast cell growth factor and stimulates mitogen-activated protein kinase but is down-regulated in melanomas.

Loss of AP-2 results in downregulation of c-KIT and enhancement of melanoma tumorigenicity and metastasis.

A missense mutation in KIT kinase domain 1 correlates with imatinib resistance in gastrointestinal stromal tumors.

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