Control of hair follicle cell fate by underlying mesenchyme through a CSL-Wnt5a-FoxN1 regulatory axis. - Wikidata - awgldk - TriplyDB

Control of hair follicle cell fate by underlying mesenchyme through a CSL-Wnt5a-FoxN1 regulatory axis.

Control of hair follicle cell fate by underlying mesenchyme through a CSL-Wnt5a-FoxN1 regulatory axis.

http://www.wikidata.org/entity/Q33998879

about

WNT-5A: signaling and functions in health and disease Feeder Layer Cell Actions and Applications Mesenchymal-epithelial interactions during hair follicle morphogenesis and cycling Multifocal epithelial tumors and field cancerization: stroma as a primary determinant Foxn1 Transcription Factor Regulates Wound Healing of Skin through Promoting Epithelial-Mesenchymal Transition Mouse hair cycle expression dynamics modeled as coupled mesenchymal and epithelial oscillators Notch signaling regulates late-stage epidermal differentiation and maintains postnatal hair cycle homeostasis.Wnt5a can both activate and repress Wnt/β-catenin signaling during mouse embryonic development.Epidermal Wnt controls hair follicle induction by orchestrating dynamic signaling crosstalk between the epidermis and dermis.Wls is expressed in the epidermis and regulates embryonic hair follicle induction in mice Pharmacologic inhibition of JAK-STAT signaling promotes hair growth Highly upregulated Lhx2 in the Foxn1-/- nude mouse phenotype reflects a dysregulated and expanded epidermal stem cell niche.Distinct requirements for cranial ectoderm and mesenchyme-derived wnts in specification and differentiation of osteoblast and dermal progenitors.UV-Induced Wnt7a in the Human Skin Microenvironment Specifies the Fate of Neural Crest-Like Cells via Suppression of Notch.Dermal β-catenin activity in response to epidermal Wnt ligands is required for fibroblast proliferation and hair follicle initiation.Shh maintains dermal papilla identity and hair morphogenesis via a Noggin-Shh regulatory loop.Neotenic phenomenon in gene expression in the skin of Foxn1- deficient (nude) mice - a projection for regenerative skin wound healing.Separate and distinctive roles for Wnt5a in tongue, lingual tissue and taste papilla development.Wnt5a controls Notch1 signaling through CaMKII-mediated degradation of the SMRT corepressor protein Combined CSL and p53 downregulation promotes cancer-associated fibroblast activation Wnt signaling in skin development, homeostasis, and disease.Multifocal epithelial tumors and field cancerization from loss of mesenchymal CSL signaling.Adenovirus-mediated Wnt5a expression inhibits the telogen-to-anagen transition of hair follicles in mice Aire-expressing thymic medullary epithelial cells originate from β5t-expressing progenitor cells.Negative control of CSL gene transcription by stress/DNA damage response and p53.Iterative Role of Notch Signaling in Spinal Motor Neuron Diversification Signaling involved in hair follicle morphogenesis and development.SOX9: a stem cell transcriptional regulator of secreted niche signaling factors.Hairy tale of signaling in hair follicle development and cycling.Gata6 promotes hair follicle progenitor cell renewal by genome maintenance during proliferation.FOXN1 Transcription Factor in Epithelial Growth and Wound Healing.Defining the identity of mouse embryonic dermal fibroblasts.Somatic Mutations in NEK9 Cause Nevus Comedonicus.CD133-positive dermal papilla-derived Wnt ligands regulate postnatal hair growth.Wnt Signalling Pathways in Skin Development and Epidermal Stem Cells.Skin transcriptome reveals the intrinsic molecular mechanisms underlying hair follicle cycling in Cashmere goats under natural and shortened photoperiod conditions.KLF4 transcriptionally activates non-canonical WNT5A to control epithelial stratification.Gsdma3 regulates hair follicle differentiation via Wnt5a-mediated non-canonical Wnt signaling pathway.Fibroblast heterogeneity: implications for human disease.Overexpression of Wnt5a in mouse epidermis causes no psoriasis phenotype but an impairment of hair follicle anagen development.

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Control of hair follicle cell fate by underlying mesenchyme through a CSL-Wnt5a-FoxN1 regulatory axis.

http://www.wikidata.org/entity/Q33998879

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Control of hair follicle cell ...... L-Wnt5a-FoxN1 regulatory axis.

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Control of hair follicle cell ...... L-Wnt5a-FoxN1 regulatory axis.

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type

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Control of hair follicle cell ...... L-Wnt5a-FoxN1 regulatory axis.

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Control of hair follicle cell ...... L-Wnt5a-FoxN1 regulatory axis.

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Control of hair follicle cell ...... L-Wnt5a-FoxN1 regulatory axis.

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Control of hair follicle cell ...... L-Wnt5a-FoxN1 regulatory axis.

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Genes & Development

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Control of hair follicle cell ...... L-Wnt5a-FoxN1 regulatory axis.

@en

P2093

Bach-Cuc Nguyen

http://www.w3.org/2001/XMLSchema#string

Bing Hu

http://www.w3.org/2001/XMLSchema#string

Cathrin Brisken

http://www.w3.org/2001/XMLSchema#string

Einar Castillo

http://www.w3.org/2001/XMLSchema#string

Fenglei He

http://www.w3.org/2001/XMLSchema#string

G Paolo Dotto

http://www.w3.org/2001/XMLSchema#string

Karine Lefort

http://www.w3.org/2001/XMLSchema#string

Peter Angel

http://www.w3.org/2001/XMLSchema#string

Renuga Devi Rajaram

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Wenying Qiu

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P2860

The Wingless homolog WNT5A and its receptor Frizzled-5 regulate inflammatory responses of human mononuclear cells induced by microbial stimulation

The orphan tyrosine kinase receptor, ROR2, mediates Wnt5A signaling in metastatic melanoma

Notch1 is a p53 target gene involved in human keratinocyte tumor suppression through negative regulation of ROCK1/2 and MRCKalpha kinases

Molecular dissection of mesenchymal-epithelial interactions in the hair follicle

Generalized lacZ expression with the ROSA26 Cre reporter strain

A Wnt5a pathway underlies outgrowth of multiple structures in the vertebrate embryo

Canonical notch signaling functions as a commitment switch in the epidermal lineage

Purified Wnt5a protein activates or inhibits beta-catenin-TCF signaling depending on receptor context

Spatio-temporally regulated expression of receptor tyrosine kinases, mRor1, mRor2, during mouse development: implications in development and function of the nervous system

Role of the nude gene in epithelial terminal differentiation

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1519-1532

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10.1101/GAD.1886910

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14

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24

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2010-07-01T00:00:00Z

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45199892

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20634318

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2904942

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