The cell cycle and Myc intersect with mechanisms that regulate pluripotency and reprogramming. - Wikidata - awgldk - TriplyDB

The cell cycle and Myc intersect with mechanisms that regulate pluripotency and reprogramming.

The cell cycle and Myc intersect with mechanisms that regulate pluripotency and reprogramming.

http://www.wikidata.org/entity/Q34017933

about

Direct observation of cell cycle progression in living mouse embryonic stem cells on an extracellular matrix of E-cadherin Functional interactions among members of the MAX and MLX transcriptional network during oncogenesis Concise review: The Sox2-Oct4 connection: critical players in a much larger interdependent network integrated at multiple levels The cell cycle and pluripotency The cell cycle as a brake for β-cell regeneration from embryonic stem cells Inhibition of c-Myc overcomes cytotoxic drug resistance in acute myeloid leukemia cells by promoting differentiation V-myc immortalizes human neural stem cells in the absence of pluripotency-associated traits The architectural organization of human stem cell cycle regulatory machinery Esrrb is a pivotal target of the Gsk3/Tcf3 axis regulating embryonic stem cell self-renewal MYC on the path to cancer Direct inhibition of c-Myc-Max heterodimers by celastrol and celastrol-inspired triterpenoids.Dynamic single-cell imaging of direct reprogramming reveals an early specifying event.Live imaging of the Drosophila spermatogonial stem cell niche reveals novel mechanisms regulating germline stem cell output.Invasive Cell Fate Requires G1 Cell-Cycle Arrest and Histone Deacetylase-Mediated Changes in Gene Expression Utilizing FUCCI reporters to understand pluripotent stem cell biology.Stem cell characteristics in glioblastoma are maintained by the ecto-nucleotidase E-NPP1.Structure of Nascent Chromatin Is Essential for Hematopoietic Lineage Specification.TIMELESS contributes to the progression of breast cancer through activation of MYC.G1 checkpoint establishment in vivo during embryonic liver development.Induced pluripotent stem cells: what lies beyond the paradigm shift The Dual Roles of MYC in Genomic Instability and Cancer Chemoresistance Molecular ties between the cell cycle and differentiation in embryonic stem cells.Cell-type independent MYC target genes reveal a primordial signature involved in biomass accumulation.Computational analysis of expression of human embryonic stem cell-associated signatures in tumors.Antiviral responses in mouse embryonic stem cells: differential development of cellular mechanisms in type I interferon production and response.Destroy to create: E3 ubiquitin ligases in neurogenesis.Myc represses primitive endoderm differentiation in pluripotent stem cells.Transcriptional amplification in tumor cells with elevated c-Myc.Genome-wide profiling of pluripotent cells reveals a unique molecular signature of human embryonic germ cells.Regulation of male germ cell cycle arrest and differentiation by DND1 is modulated by genetic background.The B-MYB transcriptional network guides cell cycle progression and fate decisions to sustain self-renewal and the identity of pluripotent stem cells.Noncoding RNA landmarks of pluripotency and reprogramming.LIN9, a subunit of the DREAM complex, regulates mitotic gene expression and proliferation of embryonic stem cells.Self-renewal induced efficiently, safely, and effective therapeutically with one regulatable gene in a human somatic progenitor cell.Transcription factor TFAP2C regulates major programs required for murine fetal germ cell maintenance and haploinsufficiency predisposes to teratomas in male mice Overexpression of cdk4 and cyclinD1 triggers greater expansion of neural stem cells in the adult mouse brain Myc transcription factors: key regulators behind establishment and maintenance of pluripotency.TRAF2 recruitment via T61 in CD30 drives NFκB activation and enhances hESC survival and proliferation.Single cell analysis reveals the stochastic phase of reprogramming to pluripotency is an ordered probabilistic process.Myc orchestrates a regulatory network required for the establishment and maintenance of pluripotency.

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The cell cycle and Myc intersect with mechanisms that regulate pluripotency and reprogramming.

http://www.wikidata.org/entity/Q34017933

description

2009 nî lūn-bûn

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2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի օգոստոսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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The cell cycle and Myc interse ...... luripotency and reprogramming.

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The cell cycle and Myc interse ...... luripotency and reprogramming.

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The cell cycle and Myc interse ...... luripotency and reprogramming.

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The cell cycle and Myc interse ...... luripotency and reprogramming.

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The cell cycle and Myc interse ...... luripotency and reprogramming.

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The cell cycle and Myc interse ...... luripotency and reprogramming.

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The cell cycle and Myc interse ...... luripotency and reprogramming.

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The cell cycle and Myc interse ...... luripotency and reprogramming.

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The cell cycle and Myc interse ...... luripotency and reprogramming.

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J.STEM.2009.07.003

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The cell cycle and Myc interse ...... luripotency and reprogramming.

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Amar M Singh

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Stephen Dalton

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P2860

mTOR controls cell cycle progression through its cell growth effectors S6K1 and 4E-BP1/eukaryotic translation initiation factor 4E

Oct4/Sox2-regulated miR-302 targets cyclin D1 in human embryonic stem cells

A restriction point for control of normal animal cell proliferation

Metastable pluripotent states in NOD-mouse-derived ESCs

Genomic targets of the human c-Myc protein

Induction of pluripotent stem cells from mouse embryonic and adult fibroblast cultures by defined factors

N-myc is essential during neurogenesis for the rapid expansion of progenitor cell populations and the inhibition of neuronal differentiation

An extended transcriptional network for pluripotency of embryonic stem cells

Promotion of reprogramming to ground state pluripotency by signal inhibition

Nmyc plays an essential role during lung development as a dosage-sensitive regulator of progenitor cell proliferation and differentiation

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