Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations. - Wikidata - awgldk - TriplyDB

Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations.

Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations.

http://www.wikidata.org/entity/Q34079245

about

The sodium-potassium pump controls the intrinsic firing of the cerebellar Purkinje neuron Inhibitory synaptic plasticity: spike timing-dependence and putative network function.Computational modeling of seizure dynamics using coupled neuronal networks: factors shaping epileptiform activity The blue brain project Bistable dynamics underlying excitability of ion homeostasis in neuron models Potassium dynamics in the epileptic cortex: new insights on an old topic.Maintenance and termination of neocortical oscillations by dynamic modulation of intrinsic and synaptic excitability Slow state transitions of sustained neural oscillations by activity-dependent modulation of intrinsic excitability Computational modeling reveals dendritic origins of GABA(A)-mediated excitation in CA1 pyramidal neurons.Network bistability mediates spontaneous transitions between normal and pathological brain states.Divide and conquer: functional segregation of synaptic inputs by astrocytic microdomains could alleviate paroxysmal activity following brain trauma.Enhanced multiple vibrational resonances by Na+ and K+ dynamics in a neuron model.Cellular and network mechanisms of electrographic seizures.Control of sleep-to-wake transitions via fast aminoacid and slow neuropeptide transmission Ion concentration dynamics as a mechanism for neuronal bursting.Efficacy of synaptic inhibition depends on multiple, dynamically interacting mechanisms implicated in chloride homeostasis.Dynamics from seconds to hours in Hodgkin-Huxley model with time-dependent ion concentrations and buffer reservoirs.Electrogenic properties of the Na⁺/K⁺ ATPase control transitions between normal and pathological brain states.Metabotropic glutamate receptors (mGluR5) activate transient receptor potential canonical channels to improve the regularity of the respiratory rhythm generated by the pre-Bötzinger complex in mice.Characterising seizures in anti-NMDA-receptor encephalitis with dynamic causal modelling Modeling of Age-Dependent Epileptogenesis by Differential Homeostatic Synaptic Scaling.In vivo models of cortical acquired epilepsy Diffusive coupling and network periodicity: a computational study.Dynamics of high-frequency synchronization during seizures.The unimodal distribution of sub-threshold, ongoing activity in cortical networks.Focal generation of paroxysmal fast runs during electrographic seizures.Interaction between synaptic inhibition and glial-potassium dynamics leads to diverse seizure transition modes in biophysical models of human focal seizures.Synaptic Conductances during Interictal Discharges in Pyramidal Neurons of Rat Entorhinal Cortex.A primate-specific, brain isoform of KCNH2 affects cortical physiology, cognition, neuronal repolarization and risk of schizophrenia Potassium diffusive coupling in neural networks.Computational models of neuron-astrocyte interaction in epilepsy.Ionic dynamics mediate spontaneous termination of seizures and postictal depression state.Gain control through divisive inhibition prevents abrupt transition to chaos in a neural mass model Tracking and control of neuronal Hodgkin-Huxley dynamics.The influence of potassium concentration on epileptic seizures in a coupled neuronal model in the hippocampus.Modulation of axonal excitability by high-frequency biphasic electrical current.The influence of sodium and potassium dynamics on excitability, seizures, and the stability of persistent states: I. Single neuron dynamics Influence of frequency and temperature on the mechanisms of nerve conduction block induced by high-frequency biphasic electrical current.A Hamilton-Jacobi-Bellman approach for termination of seizure-like bursting.The role of slow potassium current in nerve conduction block induced by high-frequency biphasic electrical current.

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P2860

Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations.

http://www.wikidata.org/entity/Q34079245

description

2004 nî lūn-bûn

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2004 թուականի Մարտին հրատարակուած գիտական յօդուած

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2004 թվականի մարտին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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name

Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations.

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Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations.

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Potassium model for slow

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type

label

Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations.

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Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations.

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Potassium model for slow

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prefLabel

Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations.

@ast

Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations.

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Potassium model for slow

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Journal of Neurophysiology

P1476

Potassium model for slow (2-3 Hz) in vivo neocortical paroxysmal oscillations.

@en

P2093

I Timofeev

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M Bazhenov

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M Steriade

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P2860

Potassium-induced enhancement of persistent inward current in hippocampal neurons in isolation and in tissue slices.

A model of a CA3 hippocampal pyramidal neuron incorporating voltage-clamp data on intrinsic conductances.

Homeostatic plasticity in neuronal networks: the more things change, the more they stay the same.

Impaired K(+) homeostasis and altered electrophysiological properties of post-traumatic hippocampal glia.

Cortical hyperpolarization-activated depolarizing current takes part in the generation of focal paroxysmal activities.

The role of chloride-dependent inhibition and the activity of fast-spiking neurons during cortical spike-wave electrographic seizures.

Lennox-Gastaut syndrome. Clinical description and diagnosis.

The neural code between neocortical pyramidal neurons depends on neurotransmitter release probability.

Synthesis of models for excitable membranes, synaptic transmission and neuromodulation using a common kinetic formalism.

Mechanisms of neocortical epileptogenesis in vitro.

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1116-1132

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scholarly article

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10.1152/JN.00529.2003

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2

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92

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Terrence J. Sejnowski

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2004-03-31T00:00:00Z

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8643968

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