Novel prognostic subgroups in childhood 11q23/MLL-rearranged acute myeloid leukemia: results of an international retrospective study.
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Targeting histone methyltransferases and demethylases in clinical trials for cancer therapyEpigenetic regulators and their impact on therapy in acute myeloid leukemiaRare Cytogenetic Abnormalities and Alteration of microRNAs in Acute Myeloid Leukemia and Response to TherapySystematic Classification of Mixed-Lineage Leukemia Fusion Partners Predicts Additional Cancer PathwaysPediatric AML: From Biology to Clinical ManagementRecent progress toward epigenetic therapies: the example of mixed lineage leukemiaHow I treat pediatric acute myeloid leukemiaChildhood acute myeloid leukaemiaCurrent Management of Childhood Acute Myeloid Leukemia.Strategies for reducing the treatment-related physical burden of childhood acute myeloid leukaemia - a review.MLL rearrangements impact outcome in HOXA-deregulated T-lineage acute lymphoblastic leukemia: a Children's Oncology Group Study.Discovering and understanding oncogenic gene fusions through data intensive computational approachesChromatin modifications as therapeutic targets in MLL-rearranged leukemia.Mixed lineage leukemia-septin 5 fusion transcript in de novo adult acute myeloid leukemia with t(11;22)(q23;q11.2): A case reportRole of misfolded N-CoR mediated transcriptional deregulation of Flt3 in acute monocytic leukemia (AML)-M5 subtype.Prognostic significance of 11q23 aberrations in adult acute myeloid leukemia and the role of allogeneic stem cell transplantation.Histone H3 lysine 79 methyltransferase Dot1 is required for immortalization by MLL oncogenes.Development of five dual-color, double-fusion fluorescence in situ hybridization assays for the detection of common MLL translocation partnersThe MLL recombinome of acute leukemias in 2013.Prognostic factors in children with acute myeloid leukaemia and excellent response to remission induction therapy.Biology, risk stratification, and therapy of pediatric acute leukemias: an update.Pediatric acute myeloid leukemia with t(8;16)(p11;p13), a distinct clinical and biological entity: a collaborative study by the International-Berlin-Frankfurt-Munster AML-study group.ARID5B polymorphism confers an increased risk to acquire specific MLL rearrangements in early childhood leukemia.Prognostic significance of additional cytogenetic aberrations in 733 de novo pediatric 11q23/MLL-rearranged AML patients: results of an international studymll ortholog containing functional domains of human MLL is expressed throughout the zebrafish lifespan and in haematopoietic tissuesIntegrative analysis of type-I and type-II aberrations underscores the genetic heterogeneity of pediatric acute myeloid leukemiaGene expression profiles associated with pediatric relapsed AML.Vandetanib mediates anti-leukemia activity by multiple mechanisms and interacts synergistically with DNA damaging agents.The role of matched sibling donor allogeneic stem cell transplantation in pediatric high-risk acute myeloid leukemia: results from the AML-BFM 98 study.Subtype prediction in pediatric acute myeloid leukemia: classification using differential network rank conservation revisitedCollaborative Efforts Driving Progress in Pediatric Acute Myeloid Leukemia.Heterogeneous cytogenetic subgroups and outcomes in childhood acute megakaryoblastic leukemia: a retrospective international studyFuGePrior: A novel gene fusion prioritization algorithm based on accurate fusion structure analysis in cancer RNA-seq samples.Does hematopoietic stem cell transplantation benefit infants with acute leukemia?Cell of origin determines clinically relevant subtypes of MLL-rearranged AMLMolecular monitoring of minimal residual disease in two patients with MLL-rearranged acute myeloid leukemia and haploidentical transplantation after relapse.Advances in paediatric cancer treatmentLeukemic transformation by the MLL-AF6 fusion oncogene requires the H3K79 methyltransferase Dot1l.High EVI1 expression is associated with MLL rearrangements and predicts decreased survival in paediatric acute myeloid leukaemia: a report from the children's oncology group.MLL fusion protein-driven AML is selectively inhibited by targeted disruption of the MLL-PAFc interaction.
P2860
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P2860
Novel prognostic subgroups in childhood 11q23/MLL-rearranged acute myeloid leukemia: results of an international retrospective study.
description
2009 nî lūn-bûn
@nan
2009 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Novel prognostic subgroups in ...... rnational retrospective study.
@ast
Novel prognostic subgroups in ...... rnational retrospective study.
@en
Novel prognostic subgroups in ...... rnational retrospective study.
@nl
type
label
Novel prognostic subgroups in ...... rnational retrospective study.
@ast
Novel prognostic subgroups in ...... rnational retrospective study.
@en
Novel prognostic subgroups in ...... rnational retrospective study.
@nl
prefLabel
Novel prognostic subgroups in ...... rnational retrospective study.
@ast
Novel prognostic subgroups in ...... rnational retrospective study.
@en
Novel prognostic subgroups in ...... rnational retrospective study.
@nl
P2093
P2860
P50
P1433
P1476
Novel prognostic subgroups in ...... ernational retrospective study
@en
P2093
Akira Morimoto
Anna Leszl
Anne Auvrignon
Brenda Gibson
Brian V Balgobind
C Michel Zwaan
Christine Perot
David Webb
Dirk Reinhardt
Erik Forestier
P2860
P304
P356
10.1182/BLOOD-2009-04-215152
P407
P50
P577
2009-06-15T00:00:00Z