Hypertrophic chondrocytes can become osteoblasts and osteocytes in endochondral bone formation. - Wikidata - awgldk - TriplyDB

Hypertrophic chondrocytes can become osteoblasts and osteocytes in endochondral bone formation.

Hypertrophic chondrocytes can become osteoblasts and osteocytes in endochondral bone formation.

http://www.wikidata.org/entity/Q34083023

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Wnt signaling in cartilage development and diseases: lessons from animal studies HIF-1α and growth plate development: what we really know Fibroblast growth factor signaling in skeletal development and disease Skeletal stem cells A pathway to bone: signaling molecules and transcription factors involved in chondrocyte development and maturation The multifaceted role of the vasculature in endochondral fracture repair Cell Death in Chondrocytes, Osteoblasts, and Osteocytes Tyrosine kinase inhibitors and mesenchymal stromal cells: effects on self-renewal, commitment and functions Role of IGF-I signaling in muscle bone interactions Regulation of Long Bone Growth in Vertebrates; It Is Time to Catch Up Advances in Skeletal Dysplasia Genetics Bone development Hydrogel based cartilaginous tissue regeneration: recent insights and technologies.Deletion of Runx2 in Articular Chondrocytes Decelerates the Progression of DMM-Induced Osteoarthritis in Adult Mice.Quiescent Bone Lining Cells Are a Major Source of Osteoblasts During Adulthood.Specific ablation of mouse Fam20C in cells expressing type I collagen leads to skeletal defects and hypophosphatemia.The Good the Bad and the Ugly of Glycosaminoglycans in Tissue Engineering Applications Chondrocytes transdifferentiate into osteoblasts in endochondral bone during development, postnatal growth and fracture healing in mice.Mutant activated FGFR3 impairs endochondral bone growth by preventing SOX9 downregulation in differentiating chondrocytes Dual pathways to endochondral osteoblasts: a novel chondrocyte-derived osteoprogenitor cell identified in hypertrophic cartilage The developmental basis of mesenchymal stem/stromal cells (MSCs).SOXC Transcription Factors Induce Cartilage Growth Plate Formation in Mouse Embryos by Promoting Noncanonical WNT Signaling.Chondrocytes-Specific Expression of Osteoprotegerin Modulates Osteoclast Formation in Metaphyseal Bone Deficiency and Also Transgenic Overexpression of Timp-3 Both Lead to Compromised Bone Mass and Architecture In Vivo.Histone Deacetylases in Bone Development and Skeletal Disorders.Runx2-interacting genes identified by yeast two-hybrid screening of libraries generated from hypertrophic chondrocytes.Chondrocytes Directly Transform into Bone Cells in Mandibular Condyle Growth.The Mighty Chondrocyte: No Bones about It.Col2-Cre and tamoxifen-inducible Col2-CreER target different cell populations in the knee joint Inflammation-driven bone formation in a mouse model of ankylosing spondylitis: sequential not parallel processes.Interplay between CaSR and PTH1R signaling in skeletal development and osteoanabolism.Transcriptional profiling of cortical versus cancellous bone from mechanically-loaded murine tibiae reveals differential gene expression.Understanding craniosynostosis as a growth disorder Tissue engineering strategies for promoting vascularized bone regeneration.Ihha induces hybrid cartilage-bone cells during zebrafish jawbone regeneration.Mechanical loading inhibits hypertrophy in chondrogenically differentiating hMSCs within a biomimetic hydrogel.Basic science of osteoarthritis.Osteoblast Differentiation at a Glance.β-catenin activity in late hypertrophic chondrocytes locally orchestrates osteoblastogenesis and osteoclastogenesis.Clec11a/osteolectin is an osteogenic growth factor that promotes the maintenance of the adult skeleton.

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Hypertrophic chondrocytes can become osteoblasts and osteocytes in endochondral bone formation.

http://www.wikidata.org/entity/Q34083023

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2014 nî lūn-bûn

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2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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Hypertrophic chondrocytes can ...... n endochondral bone formation.

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Hypertrophic chondrocytes can ...... n endochondral bone formation.

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Hypertrophic chondrocytes can ...... n endochondral bone formation.

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Hypertrophic chondrocytes can ...... n endochondral bone formation.

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Hypertrophic chondrocytes can ...... n endochondral bone formation.

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Hypertrophic chondrocytes can ...... n endochondral bone formation.

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PNAS.1302703111

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Hypertrophic chondrocytes can ...... n endochondral bone formation.

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P2093

Danny Chan

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Hoi Ching Tang

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Kwok Yeung Tsang

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Liu Yang

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P2860

The novel zinc finger-containing transcription factor osterix is required for osteoblast differentiation and bone formation

Ligand-activated site-specific recombination in mice

Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development

Wnt/beta-catenin signaling in mesenchymal progenitors controls osteoblast and chondrocyte differentiation during vertebrate skeletogenesis

Critical roles for collagenase-3 (Mmp13) in development of growth plate cartilage and in endochondral ossification

The transcription factor Sox9 has essential roles in successive steps of the chondrocyte differentiation pathway and is required for expression of Sox5 and Sox6

Osteoblast precursors, but not mature osteoblasts, move into developing and fractured bones along with invading blood vessels.

Surviving endoplasmic reticulum stress is coupled to altered chondrocyte differentiation and function

The timing of ossification of the limb bones, and growth rates of various long bones of the fore and hind limbs of the prenatal and early postnatal laboratory mouse

Osteo-chondroprogenitor cells are derived from Sox9 expressing precursors.

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Kathryn S E Cheah

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