Synaptic activity becomes excitotoxic in neurons exposed to elevated levels of platelet-activating factor.
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HIV-1 Tat Activates Neuronal Ryanodine Receptors with Rapid Induction of the Unfolded Protein Response and Mitochondrial HyperpolarizationHIV and antiretroviral therapy in the brain: neuronal injury and repairHearing Loss After Activation of Hearing Preservation Cochlear Implants Might Be Related to Afferent Cochlear Innervation Injury.Human immunodeficiency virus-1 Tat activates calpain proteases via the ryanodine receptor to enhance surface dopamine transporter levels and increase transporter-specific uptake and Vmax.HIV-1 neuroimmunity in the era of antiretroviral therapy.Time-Dependent, HIV-Tat-Induced Perturbation of Human Neurons In Vitro: Towards a Model for the Molecular Pathology of HIV-Associated Neurocognitive DisordersInteractive comorbidity between opioid drug abuse and HIV-1 Tat: chronic exposure augments spine loss and sublethal dendritic pathology in striatal neuronsThe new small-molecule mixed-lineage kinase 3 inhibitor URMC-099 is neuroprotective and anti-inflammatory in models of human immunodeficiency virus-associated neurocognitive disorders.Voltage-gated potassium channels in human immunodeficiency virus type-1 (HIV-1)-associated neurocognitive disorders.Parallel high throughput neuronal toxicity assays demonstrate uncoupling between loss of mitochondrial membrane potential and neuronal damage in a model of HIV-induced neurodegenerationCharacterization of monocyte maturation/differentiation that facilitates their transmigration across the blood-brain barrier and infection by HIV: implications for NeuroAIDS.Role of the transcription factor E2F1 in CXCR4-mediated neurotoxicity and HIV neuropathology.Potentiation of Excitotoxicity in HIV-1 Associated Dementia and the Significance of Glutaminase.Morphine and gp120 toxic interactions in striatal neurons are dependent on HIV-1 strain.Opiate drug use and the pathophysiology of neuroAIDS.Platelet Activating Factor Enhances Synaptic Vesicle Exocytosis Via PKC, Elevated Intracellular Calcium, and Modulation of Synapsin 1 Dynamics and Phosphorylation.Platelet-Activating Factor Receptors Mediate Excitatory Postsynaptic Hippocampal Injury in Experimental Autoimmune EncephalomyelitisSynaptic dysfunction in the hippocampus accompanies learning and memory deficits in human immunodeficiency virus type-1 Tat transgenic mice.Antiretroviral neurotoxicityMetabolomic analysis of the cerebrospinal fluid reveals changes in phospholipase expression in the CNS of SIV-infected macaques.Anaphylactic shock depends on endothelial Gq/G11.Multiphysics neuron model for cellular volume dynamics.Neuronal injury in simian immunodeficiency virus and other animal models of neuroAIDS.Morphine causes rapid increases in glial activation and neuronal injury in the striatum of inducible HIV-1 Tat transgenic miceThe Role of Platelet-Activating Factor in Chronic Inflammation, Immune Activation, and Comorbidities Associated with HIV Infection.Lithium therapy for human immunodeficiency virus type 1-associated neurocognitive impairment.Cocaine Exposure Results in Formation of Dendritic Varicosity in Rat Primary Hippocampal Neurons.Molecular pathology of neuro-AIDS (CNS-HIV).Loss of PAFR prevents neuroinflammation and brain dysfunction after traumatic brain injury.CNS inflammation and macrophage/microglial biology associated with HIV-1 infection.Rebuilding synaptic architecture in HIV-1 associated neurocognitive disease: a therapeutic strategy based on modulation of mixed lineage kinaseNeurotrophin strategies for neuroprotection: are they sufficient?An overview of the biological and psychosocial context surrounding neurocognition in HIV.Platelet-activating factor attenuation of long-term potentiation in rat hippocampal slices via protein tyrosine kinase signaling.Macrophages and Phospholipases at the Intersection between Inflammation and the Pathogenesis of HIV-1 Infection.Heterogeneity in the sn-1 carbon chain of platelet-activating factor glycerophospholipids determines pro- or anti-apoptotic signaling in primary neurons.Phospholipase A2 inhibitors protect against prion and Abeta mediated synapse degeneration.α-Synuclein-induced synapse damage in cultured neurons is mediated by cholesterol-sensitive activation of cytoplasmic phospholipase A2.Pathogenesis of Human Immunodeficiency Virus Type-1 (HIV-1)-Associated Dementia: Role of Voltage-Gated Potassium Channels.Noise induced reversible changes of cochlear ribbon synapses contribute to temporary hearing loss in mice.
P2860
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P2860
Synaptic activity becomes excitotoxic in neurons exposed to elevated levels of platelet-activating factor.
description
2005 nî lūn-bûn
@nan
2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Synaptic activity becomes exci ...... of platelet-activating factor.
@ast
Synaptic activity becomes exci ...... of platelet-activating factor.
@en
Synaptic activity becomes exci ...... of platelet-activating factor.
@nl
type
label
Synaptic activity becomes exci ...... of platelet-activating factor.
@ast
Synaptic activity becomes exci ...... of platelet-activating factor.
@en
Synaptic activity becomes exci ...... of platelet-activating factor.
@nl
prefLabel
Synaptic activity becomes exci ...... of platelet-activating factor.
@ast
Synaptic activity becomes exci ...... of platelet-activating factor.
@en
Synaptic activity becomes exci ...... of platelet-activating factor.
@nl
P2860
P356
P1476
Synaptic activity becomes exci ...... of platelet-activating factor.
@en
P2093
Matthew J Bellizzi
Shao-Ming Lu
P2860
P304
P356
10.1172/JCI25444
P407
P577
2005-11-01T00:00:00Z