Metabolic cooperation between different oncogenes during cell transformation: interaction between activated ras and HPV-16 E7. - Wikidata - awgldk - TriplyDB

Metabolic cooperation between different oncogenes during cell transformation: interaction between activated ras and HPV-16 E7.

Metabolic cooperation between different oncogenes during cell transformation: interaction between activated ras and HPV-16 E7.

http://www.wikidata.org/entity/Q34099933

about

Warburg effect revisited: an epigenetic link between glycolysis and gastric carcinogenesis Faecal tumour M2 pyruvate kinase: a new, sensitive screening tool for colorectal cancer Serine and glycine metabolism in cancer Stealing the Keys to the Kitchen: Viral Manipulation of the Host Cell Metabolic Network Evaluation of the Pyruvate Kinase isoenzyme tumor (Tu M2-PK) as a tumor marker for cervical carcinoma Mass spectrometric characterization of protein structure details refines the proteome signature for invasive ductal breast carcinoma.The metabolic marker tumour pyruvate kinase type M2 (tumour M2-PK) shows increased expression along the metaplasia-dysplasia-adenocarcinoma sequence in Barrett's oesophagus Coexistence of K-ras mutations and HPV infection in colon cancer.Complete absence of M2-pyruvate kinase expression in benign pancreatic ductal epithelium and pancreaticobiliary and duodenal neoplasia Regulation of the pentose phosphate pathway by an androgen receptor-mTOR-mediated mechanism and its role in prostate cancer cell growth.Colorectal cancer screening by non-invasive metabolic biomarker fecal tumor M2-PK.Block copolymer micelles for controlled delivery of glycolytic enzyme inhibitors.Mitogenic and oncogenic stimulation of K433 acetylation promotes PKM2 protein kinase activity and nuclear localization.Metabolism and proliferation share common regulatory pathways in cancer cells.Structure-based drug design: from nucleic acid to membrane protein targets.Non-stationary 13C metabolic flux analysis of Chinese hamster ovary cells in batch culture using extracellular labeling highlights metabolic reversibility and compartmentation.Modulation of apoptosis by human papillomavirus (HPV) oncoproteins.Pyruvate kinase M2 promotes de novo serine synthesis to sustain mTORC1 activity and cell proliferation.Oxidative Dimerization of PHD2 is Responsible for its Inactivation and Contributes to Metabolic Reprogramming via HIF-1α Activation Enhancing mitochondrial respiration suppresses tumor promoter TPA-induced PKM2 expression and cell transformation in skin epidermal JB6 cells.Estrogen-related receptor alpha is critical for the growth of estrogen receptor-negative breast cancer.Reexpression of pyruvate kinase M2 in type 1 myofibers correlates with altered glucose metabolism in myotonic dystrophy.Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.HPV, KRAS mutations, alcohol consumption and tobacco smoking effects on esophageal squamous-cell carcinoma carcinogenesis.A survey of proteins encoded by non-synonymous single nucleotide polymorphisms reveals a significant fraction with altered stability and activity.Metabolic reprogramming: a hallmark of viral oncogenesis.Metabolic alterations accompanying oncogene-induced senescence.ATM couples replication stress and metabolic reprogramming during cellular senescence.Pyruvate kinase type M2 is upregulated in colorectal cancer and promotes proliferation and migration of colon cancer cells.Impact of exogenous lactate on survival and radioresponse of carcinoma cells in vitro.Isotype-specific inhibitors of the glycolytic key regulator pyruvate kinase subtype M2 moderately decelerate tumor cell proliferation.Metabolic signatures of Besnoitia besnoiti-infected endothelial host cells and blockage of key metabolic pathways indicate high glycolytic and glutaminolytic needs of the parasite.PKM2 inhibitor shikonin suppresses TPA-induced mitochondrial malfunction and proliferation of skin epidermal JB6 cells.Ras transformation requires metabolic control by 6-phosphofructo-2-kinase.Metabolomic changes during cellular transformation monitored by metabolite-metabolite correlation analysis and correlated with gene expression.Phosphoglycerate mutase-derived polypeptide inhibits glycolytic flux and induces cell growth arrest in tumor cell lines.Alteration of cellular metabolism in cancer cells and its therapeutic prospects.The role of glucose metabolism and glucose-associated signalling in cancer.Fructose-1,6-bisphosphate couples glycolytic flux to activation of Ras.Metabolic analysis of senescent human fibroblasts reveals a role for AMP in cellular senescence.

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P2860

Metabolic cooperation between different oncogenes during cell transformation: interaction between activated ras and HPV-16 E7.

http://www.wikidata.org/entity/Q34099933

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2001 nî lūn-bûn

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Eigenbrodt E

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Jansen-Dürr P

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Mazurek S

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Zwerschke W

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P2860

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

Maximum activities of key enzymes of glycolysis, glutaminolysis, pentose phosphate pathway and tricarboxylic acid cycle in normal, neoplastic and suppressed cells

Effects of the human papilloma virus HPV-16 E7 oncoprotein on glycolysis and glutaminolysis: role of pyruvate kinase type M2 and the glycolytic-enzyme complex

Modulation of type M2 pyruvate kinase activity by the human papillomavirus type 16 E7 oncoprotein

Cloning and functional characterization of the promoter region of the gene encoding human adenylate kinase isozyme 3

Role of HIF-1alpha in hypoxia-mediated apoptosis, cell proliferation and tumour angiogenesis

Distribution and developmental changes of adenylate kinase isozymes in the rat brain: localization of adenylate kinase 1 in the olfactory bulb

A unique glucose-dependent apoptotic pathway induced by c-Myc

Oncogenic alterations of metabolism.

Release of adenylate kinase 2 from the mitochondrial intermembrane space during apoptosis.

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10.1038/SJ.ONC.1204792

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