Combined zebrafish-yeast chemical-genetic screens reveal gene-copper-nutrition interactions that modulate melanocyte pigmentation. - Wikidata - awgldk - TriplyDB

Combined zebrafish-yeast chemical-genetic screens reveal gene-copper-nutrition interactions that modulate melanocyte pigmentation.

Combined zebrafish-yeast chemical-genetic screens reveal gene-copper-nutrition interactions that modulate melanocyte pigmentation.

http://www.wikidata.org/entity/Q34123441

about

COMMD1 is linked to the WASH complex and regulates endosomal trafficking of the copper transporter ATP7A Systematic chemical-genetic and chemical-chemical interaction datasets for prediction of compound synergism Functional toxicology: tools to advance the future of toxicity testing.A functional screen for copper homeostasis genes identifies a pharmacologically tractable cellular system.Small molecule screening identifies targetable zebrafish pigmentation pathways.Continual low-level MEK inhibition ameliorates cardio-facio-cutaneous phenotypes in zebrafish 3D imaging of transition metals in the zebrafish embryo by X-ray fluorescence microtomography.Characterization of two novel small molecules targeting melanocyte development in zebrafish embryogenesis.A yeast chemical genetic screen identifies inhibitors of human telomerase.A novel role for copper in Ras/mitogen-activated protein kinase signaling.Chemical screening in zebrafish for novel biological and therapeutic discovery.FGF1 Mediates Overnutrition-Induced Compensatory β-Cell Differentiation Trafficking of the Menkes copper transporter ATP7A is regulated by clathrin-, AP-2-, AP-1-, and Rab22-dependent steps ALDH2 mediates 5-nitrofuran activity in multiple species.Juxtaposition of chemical and mutation-induced developmental defects in zebrafish reveal a copper-chelating activity for kalihinol F.Zebrafish in the sea of mineral (iron, zinc, and copper) metabolism.Hypomelanoses in children.Zebrafish as tools for drug discovery.Copper toxicology, oxidative stress and inflammation using zebrafish as experimental model.Insights from zebrafish on human pigment cell disease and treatment.MEDNIK syndrome: a novel defect of copper metabolism treatable by zinc acetate therapy.Highly efficient and energy-conserved flocculation of copper in wastewater by pulse-alternating current.Effects of different sources and levels of copper on growth performance, nutrient digestibility, and elemental balance in young female mink (Mustela vison).How Surrogate and Chemical Genetics in Model Organisms Can Suggest Therapies for Human Genetic Diseases.

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P2860

Combined zebrafish-yeast chemical-genetic screens reveal gene-copper-nutrition interactions that modulate melanocyte pigmentation.

http://www.wikidata.org/entity/Q34123441

description

2010 nî lūn-bûn

@nan

2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Combined zebrafish-yeast chemi ...... ulate melanocyte pigmentation.

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Combined zebrafish-yeast chemi ...... ulate melanocyte pigmentation.

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Combined zebrafish-yeast chemi ...... ulate melanocyte pigmentation.

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type

label

Combined zebrafish-yeast chemi ...... ulate melanocyte pigmentation.

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Combined zebrafish-yeast chemi ...... ulate melanocyte pigmentation.

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Combined zebrafish-yeast chemi ...... ulate melanocyte pigmentation.

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prefLabel

Combined zebrafish-yeast chemi ...... ulate melanocyte pigmentation.

@ast

Combined zebrafish-yeast chemi ...... ulate melanocyte pigmentation.

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Combined zebrafish-yeast chemi ...... ulate melanocyte pigmentation.

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Disease Models & Mechanisms

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Combined zebrafish-yeast chemi ...... ulate melanocyte pigmentation.

@en

P2093

Corina Anastasaki

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E Elizabeth Patton

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Erik Madsen

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Hironori Ishizaki

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Jonathan Gitlin

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Michael Shaw

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Mike Tyers

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Sonam Dolma

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Zhiqiang Zeng

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P2860

Disruption of AP1S1, causing a novel neurocutaneous syndrome, perturbs development of the skin and spinal cord

Copper Deficiency Myelopathy (Human Swayback)

Finding the missing heritability of complex diseases

The Menkes copper transporter is required for the activation of tyrosinase

Melanosomes--dark organelles enlighten endosomal membrane transport

Predicting new molecular targets for known drugs

The INT6 cancer gene and MEK signaling pathways converge during zebrafish development

Functional profiling of the Saccharomyces cerevisiae genome

Functional characterization of the S. cerevisiae genome by gene deletion and parallel analysis

The yeast GID complex, a novel ubiquitin ligase (E3) involved in the regulation of carbohydrate metabolism

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639-651

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10.1242/DMM.005769

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9-10

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3

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