Characterization of cocaine-induced block of cardiac sodium channels.
about
Role of voltage-gated sodium, potassium and calcium channels in the development of cocaine-associated cardiac arrhythmiasA Thermally Stable Form of Bacterial Cocaine Esterase: A Potential Therapeutic Agent for Treatment of Cocaine AbuseStructural analysis of thermostabilizing mutations of cocaine esteraseCocaine serves as a peripheral interoceptive conditioned stimulus for central glutamate and dopamine releaseState-dependent compound inhibition of Nav1.2 sodium channels using the FLIPR Vm dye: on-target and off-target effects of diverse pharmacological agents.Differences in steady-state inactivation between Na channel isoforms affect local anesthetic binding affinity.Characteristics of cocaine block of purified cardiac sarcoplasmic reticulum calcium release channels.Vulnerability in an excitable medium: analytical and numerical studies of initiating unidirectional propagation.State-dependent cocaine block of sodium channel isoforms, chimeras, and channels coexpressed with the beta1 subunit.Cocaine-induced channelopathies: emerging evidence on the multiple mechanisms of sudden death.Ventral tegmental area neurons are either excited or inhibited by cocaine's actions in the peripheral nervous system.Pioglitazone attenuates acute cocaine toxicity in rat isolated heart: potential protection by metabolic modulation.The effect of amiodarone pretreatment on survival of mice with cocaine toxicityProbing kinetic drug binding mechanism in voltage-gated sodium ion channel: open state versus inactive state blockers.Mechanisms of acute cocaine toxicity.Fundamental reaction mechanism and free energy profile for (-)-cocaine hydrolysis catalyzed by cocaine esteraseElectrocardiographic findings associated with cocaine use in humans: a systematic review.Addictive drugs, arrhythmias, and cardiac inward rectifiers.Cocaine-related sudden cardiac death: a hypothesis correlating basic science and clinical observations.Crack cocaine-induced cardiac conduction abnormalities are reversed by sodium bicarbonate infusion.Prevention and reversal by cocaine esterase of cocaine-induced cardiovascular effects in rats.Cocaine binds to a common site on open and inactivated human heart (Na(v)1.5) sodium channels.Erratum: Drug-primed reinstatement of cocaine seeking in mice: increased excitability of mediumsized spiny neurons in the nucleus accumbens.Electrophysiologic and hemodynamic effects of sodium bicarbonate in a canine model of severe cocaine intoxication.Tolerance develops to the sympathomimetic but not the local anesthetic effects of cocaine.Ca-dependent action of cocaine on K current in freshly dissociated dorsal root ganglia from rats.
P2860
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P2860
Characterization of cocaine-induced block of cardiac sodium channels.
description
1990 nî lūn-bûn
@nan
1990 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի մարտին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Characterization of cocaine-induced block of cardiac sodium channels.
@ast
Characterization of cocaine-induced block of cardiac sodium channels.
@en
Characterization of cocaine-induced block of cardiac sodium channels.
@nl
type
label
Characterization of cocaine-induced block of cardiac sodium channels.
@ast
Characterization of cocaine-induced block of cardiac sodium channels.
@en
Characterization of cocaine-induced block of cardiac sodium channels.
@nl
prefLabel
Characterization of cocaine-induced block of cardiac sodium channels.
@ast
Characterization of cocaine-induced block of cardiac sodium channels.
@en
Characterization of cocaine-induced block of cardiac sodium channels.
@nl
P2860
P1433
P1476
Characterization of cocaine-induced block of cardiac sodium channels.
@en
P2093
C W Clarkson
P2860
P304
P356
10.1016/S0006-3495(90)82574-1
P407
P577
1990-03-01T00:00:00Z